n-(3-(aminomethyl)benzyl)acetamidine has been researched along with Nerve Degeneration in 3 studies
N-(3-(aminomethyl)benzyl)acetamidine: structure in first source
N-[3-(aminomethyl)benzyl]acetamidine : An aralkylamine that is Nbenzylacetamidine substituted at position 3 on the benzene ring by an aminomethyl group. An inhibitor of nitric oxide synthase.
Nerve Degeneration: Loss of functional activity and trophic degeneration of nerve axons and their terminal arborizations following the destruction of their cells of origin or interruption of their continuity with these cells. The pathology is characteristic of neurodegenerative diseases. Often the process of nerve degeneration is studied in research on neuroanatomical localization and correlation of the neurophysiology of neural pathways.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Putra, M | 1 |
| Sharma, S | 1 |
| Gage, M | 1 |
| Gasser, G | 1 |
| Hinojo-Perez, A | 1 |
| Olson, A | 1 |
| Gregory-Flores, A | 1 |
| Puttachary, S | 1 |
| Wang, C | 1 |
| Anantharam, V | 1 |
| Thippeswamy, T | 1 |
| Gao, HM | 1 |
| Zhang, F | 1 |
| Zhou, H | 1 |
| Kam, W | 1 |
| Wilson, B | 1 |
| Hong, JS | 1 |
| Chabrier, PE | 1 |
| Demerlé-Pallardy, C | 1 |
| Auguet, M | 1 |
1 review available for n-(3-(aminomethyl)benzyl)acetamidine and Nerve Degeneration
| Article | Year |
|---|---|
Nitric oxide synthases: targets for therapeutic strategies in neurological diseases.
Topics: Alzheimer Disease; Amidines; Amyotrophic Lateral Sclerosis; Animals; Benzylamines; Brain Injuries; B | 1999 |
2 other studies available for n-(3-(aminomethyl)benzyl)acetamidine and Nerve Degeneration
| Article | Year |
|---|---|
Inducible nitric oxide synthase inhibitor, 1400W, mitigates DFP-induced long-term neurotoxicity in the rat model.
Topics: Amidines; Animals; Benzylamines; Brain; Disease Models, Animal; Enzyme Inhibitors; Isoflurophate; Ma | 2020 |
Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.
Topics: alpha-Synuclein; Amidines; Animals; Animals, Genetically Modified; Benzylamines; Disease Models, Ani | 2011 |