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n-(3-(aminomethyl)benzyl)acetamidine and Brain Injuries

n-(3-(aminomethyl)benzyl)acetamidine has been researched along with Brain Injuries in 3 studies

N-(3-(aminomethyl)benzyl)acetamidine: structure in first source
N-[3-(aminomethyl)benzyl]acetamidine : An aralkylamine that is Nbenzylacetamidine substituted at position 3 on the benzene ring by an aminomethyl group. An inhibitor of nitric oxide synthase.

Brain Injuries: Acute and chronic (see also BRAIN INJURIES, CHRONIC) injuries to the brain, including the cerebral hemispheres, CEREBELLUM, and BRAIN STEM. Clinical manifestations depend on the nature of injury. Diffuse trauma to the brain is frequently associated with DIFFUSE AXONAL INJURY or COMA, POST-TRAUMATIC. Localized injuries may be associated with NEUROBEHAVIORAL MANIFESTATIONS; HEMIPARESIS, or other focal neurologic deficits.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's2 (66.67)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Jafarian-Tehrani, M2
Louin, G2
Royo, NC1
Besson, VC1
Bohme, GA1
Plotkine, M2
Marchand-Verrecchia, C2
Palmier, B1
Chabrier, PE1
Demerlé-Pallardy, C1
Auguet, M1

Reviews

1 review available for n-(3-(aminomethyl)benzyl)acetamidine and Brain Injuries

ArticleYear
Nitric oxide synthases: targets for therapeutic strategies in neurological diseases.
    Cellular and molecular life sciences : CMLS, 1999, Volume: 55, Issue:8-9

    Topics: Alzheimer Disease; Amidines; Amyotrophic Lateral Sclerosis; Animals; Benzylamines; Brain Injuries; B

1999

Other Studies

2 other studies available for n-(3-(aminomethyl)benzyl)acetamidine and Brain Injuries

ArticleYear
1400W, a potent selective inducible NOS inhibitor, improves histopathological outcome following traumatic brain injury in rats.
    Nitric oxide : biology and chemistry, 2005, Volume: 12, Issue:2

    Topics: Amidines; Animals; Benzylamines; Brain Injuries; Calcium; Disease Models, Animal; Enzyme Inhibitors;

2005
Selective inhibition of inducible nitric oxide synthase reduces neurological deficit but not cerebral edema following traumatic brain injury.
    Neuropharmacology, 2006, Volume: 50, Issue:2

    Topics: Amidines; Animals; Benzylamines; Blood-Brain Barrier; Body Water; Brain Edema; Brain Injuries; Enzym

2006