Page last updated: 2024-11-01

n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Peritonitis

n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with Peritonitis in 2 studies

N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide: structure given in first source
NS-398 : A C-nitro compound that is N-methylsulfonyl-4-nitroaniline bearing an additional cyclohexyloxy substituent at position 2.

Peritonitis: INFLAMMATION of the PERITONEUM lining the ABDOMINAL CAVITY as the result of infectious, autoimmune, or chemical processes. Primary peritonitis is due to infection of the PERITONEAL CAVITY via hematogenous or lymphatic spread and without intra-abdominal source. Secondary peritonitis arises from the ABDOMINAL CAVITY itself through RUPTURE or ABSCESS of intra-abdominal organs.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Reddy, RC1
Chen, GH1
Tateda, K1
Tsai, WC1
Phare, SM1
Mancuso, P1
Peters-Golden, M1
Standiford, TJ1
Kolaczkowska, E1
Shahzidi, S1
Seljelid, R1
van Rooijen, N1
Plytycz, B1

Other Studies

2 other studies available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Peritonitis

ArticleYear
Selective inhibition of COX-2 improves early survival in murine endotoxemia but not in bacterial peritonitis.
    American journal of physiology. Lung cellular and molecular physiology, 2001, Volume: 281, Issue:3

    Topics: Animals; Bacterial Infections; Body Temperature Regulation; Cecum; Cyclooxygenase 2; Cyclooxygenase

2001
Early vascular permeability in murine experimental peritonitis is co-mediated by resident peritoneal macrophages and mast cells: crucial involvement of macrophage-derived cysteinyl-leukotrienes.
    Inflammation, 2002, Volume: 26, Issue:2

    Topics: 6-Ketoprostaglandin F1 alpha; Animals; Capillary Leak Syndrome; Capillary Permeability; Cyclooxygena

2002