n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with Hemorrhagic Shock in 5 studies
N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide: structure given in first source
NS-398 : A C-nitro compound that is N-methylsulfonyl-4-nitroaniline bearing an additional cyclohexyloxy substituent at position 2.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To test whether hemorrhagic shock (HS) increases the Cyclooxygenase-2 (COX-2) expression in the intestine and whether this enhanced COX-2 expression mediates the intestinal dysmotility after HS." | 1.42 | Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock. ( Hao, XQ; Tian, YT; Zhang, LM; Zhang, YP, 2015) |
| "Immune function after experimental hemorrhagic shock and subsequent sepsis may be gender-related, with enhanced immunity and better survival in females." | 1.32 | Inhibition of cyclooxygenase-2 by NS-398 following hemorrhage and subsequent sepsis: no beneficial effects in either gender. ( Brötzmann, K; Kahlke, V; Schafmayer, C; Schröder, J; Seidel, M; Stapela, S, 2004) |
| "Studies indicate that trauma-hemorrhage results in activation of Kupffer cells to release inflammatory mediators and it leads to immunosuppression and increased susceptibility to subsequent sepsis." | 1.31 | Cyclooxygenase-2-mediated regulation of Kupffer cell interleukin-6 production following trauma-hemorrhage and subsequent sepsis. ( Bland, KI; Chaudry, IH; Cioffi, WG; Diodato, MD; Knöferl, MW; Schwacha, MG, 2001) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (80.00) | 29.6817 |
| 2010's | 1 (20.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhang, YP | 1 |
| Hao, XQ | 1 |
| Zhang, LM | 1 |
| Tian, YT | 1 |
| Kahlke, V | 1 |
| Seidel, M | 1 |
| Stapela, S | 1 |
| Brötzmann, K | 1 |
| Schafmayer, C | 1 |
| Schröder, J | 1 |
| Md, S | 1 |
| Moochhala, SM | 1 |
| Siew Yang, KL | 1 |
| Lu, J | 1 |
| Anuar, F | 1 |
| Mok, P | 1 |
| Ng, KC | 1 |
| Tsukada, K | 1 |
| Hasegawa, T | 1 |
| Tsutsumi, S | 1 |
| Kuwano, H | 1 |
| Knöferl, MW | 1 |
| Diodato, MD | 1 |
| Schwacha, MG | 1 |
| Cioffi, WG | 1 |
| Bland, KI | 1 |
| Chaudry, IH | 1 |
5 other studies available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Hemorrhagic Shock
| Article | Year |
|---|---|
Enhanced cyclooxygenase-2 activity leads to intestinal dysmotility following hemorrhagic shock.
Topics: Acetylcholine; Animals; Cyclooxygenase 2; Dinoprostone; Duodenum; Gastrointestinal Motility; Male; M | 2015 |
Inhibition of cyclooxygenase-2 by NS-398 following hemorrhage and subsequent sepsis: no beneficial effects in either gender.
Topics: Animals; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Cyclooxygenase Inhibitors; Cytokines; Dinopr | 2004 |
The role of selective nitric oxide synthase inhibitor on nitric oxide and PGE2 levels in refractory hemorrhagic-shocked rats.
Topics: Animals; Aspartate Aminotransferases; Blood Pressure; Brain; Creatinine; Cyclooxygenase 2; Cyclooxyg | 2005 |
Roles of cyclooxygenase-2 in tissue injury during hemorrhagic shock.
Topics: Actins; Animals; Base Sequence; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Cyclooxygenase Inhibi | 2000 |
Cyclooxygenase-2-mediated regulation of Kupffer cell interleukin-6 production following trauma-hemorrhage and subsequent sepsis.
Topics: Animals; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Cyclooxygenase Inhibitors; Dinoprostone; In | 2001 |