n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with Glial Cell Tumors in 5 studies
N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide: structure given in first source
NS-398 : A C-nitro compound that is N-methylsulfonyl-4-nitroaniline bearing an additional cyclohexyloxy substituent at position 2.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Notably, co-treatment of C6 glioma cells with 400 µmol/l NaHS and AOAA (an inhibitor of CBS) largely suppressed the above NaHS-induced effects." | 5.42 | Exogenous hydrogen sulfide promotes C6 glioma cell growth through activation of the p38 MAPK/ERK1/2-COX-2 pathways. ( Chen, J; Feng, J; Guo, R; He, J; Liu, C; Lu, Y; Zhang, W; Zhang, Y; Zhen, Y, 2015) |
| "The selective COX-2 inhibitors NS-398, Celecoxib and Meloxicam and three human glioma cell lines (D384, U251 and U87) were used." | 3.74 | Radiosensitization of human glioma cells by cyclooxygenase-2 (COX-2) inhibition: independent on COX-2 expression and dependent on the COX-2 inhibitor and sequence of administration. ( Berg, Jv; Kuipers, GK; Lafleur, MV; Slotman, BJ; Sminia, P; Stoter, TR; Wedekind, LE, 2007) |
| " In this study, we investigated the effect of chemically unrelated NSAIDs in the proliferation of glioma cell lines and the possible mechanisms involved in indomethacin-mediated inhibition of proliferation in glioma cells lines." | 3.73 | Nonsteroidal anti-inflammatory drugs inhibit the growth of C6 and U138-MG glioma cell lines. ( Battastini, AM; Bernardi, A; Delgado-Cañedo, A; Jacques-Silva, MC; Lenz, G, 2006) |
| "Notably, co-treatment of C6 glioma cells with 400 µmol/l NaHS and AOAA (an inhibitor of CBS) largely suppressed the above NaHS-induced effects." | 1.42 | Exogenous hydrogen sulfide promotes C6 glioma cell growth through activation of the p38 MAPK/ERK1/2-COX-2 pathways. ( Chen, J; Feng, J; Guo, R; He, J; Liu, C; Lu, Y; Zhang, W; Zhang, Y; Zhen, Y, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (80.00) | 29.6817 |
| 2010's | 1 (20.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhen, Y | 1 |
| Zhang, W | 1 |
| Liu, C | 1 |
| He, J | 1 |
| Lu, Y | 1 |
| Guo, R | 1 |
| Feng, J | 1 |
| Zhang, Y | 1 |
| Chen, J | 1 |
| Matsuo, M | 1 |
| Yoshida, N | 1 |
| Zaitsu, M | 1 |
| Ishii, K | 1 |
| Hamasaki, Y | 1 |
| Bernardi, A | 1 |
| Jacques-Silva, MC | 1 |
| Delgado-Cañedo, A | 1 |
| Lenz, G | 1 |
| Battastini, AM | 1 |
| Kuipers, GK | 1 |
| Slotman, BJ | 1 |
| Wedekind, LE | 1 |
| Stoter, TR | 1 |
| Berg, Jv | 1 |
| Sminia, P | 1 |
| Lafleur, MV | 1 |
| Nakano, Y | 1 |
| Kuroda, E | 1 |
| Kito, T | 1 |
| Uematsu, S | 1 |
| Akira, S | 1 |
| Yokota, A | 1 |
| Nishizawa, S | 1 |
| Yamashita, U | 1 |
5 other studies available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Glial Cell Tumors
| Article | Year |
|---|---|
Exogenous hydrogen sulfide promotes C6 glioma cell growth through activation of the p38 MAPK/ERK1/2-COX-2 pathways.
Topics: Animals; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cyclo | 2015 |
Inhibition of human glioma cell growth by a PHS-2 inhibitor, NS398, and a prostaglandin E receptor subtype EP1-selective antagonist, SC51089.
Topics: Animals; Brain Neoplasms; Cell Division; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Cyclooxygena | 2004 |
Nonsteroidal anti-inflammatory drugs inhibit the growth of C6 and U138-MG glioma cell lines.
Topics: Acetaminophen; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cell Line, Tumor; Cell Proliferatio | 2006 |
Radiosensitization of human glioma cells by cyclooxygenase-2 (COX-2) inhibition: independent on COX-2 expression and dependent on the COX-2 inhibitor and sequence of administration.
Topics: Celecoxib; Cell Line, Tumor; Cell Proliferation; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Dose | 2007 |
Induction of prostaglandin E2 synthesis and microsomal prostaglandin E synthase-1 expression in murine microglia by glioma-derived soluble factors. Laboratory investigation.
Topics: Animals; Blotting, Western; Cells, Cultured; Coculture Techniques; Culture Media, Conditioned; Cyclo | 2008 |