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n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Endometriosis

n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with Endometriosis in 3 studies

N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide: structure given in first source
NS-398 : A C-nitro compound that is N-methylsulfonyl-4-nitroaniline bearing an additional cyclohexyloxy substituent at position 2.

Endometriosis: A condition in which functional endometrial tissue is present outside the UTERUS. It is often confined to the PELVIS involving the OVARY, the ligaments, cul-de-sac, and the uterovesical peritoneum.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Takenaka, Y1
Taniguchi, F1
Miyakoda, H1
Takai, E1
Terakawa, N1
Harada, T1
Chen, YJ1
Li, HY1
Chang, YL1
Yuan, CC1
Tai, LK1
Lu, KH1
Chang, CM1
Chiou, SH1
Ozawa, Y1
Murakami, T1
Tamura, M1
Terada, Y1
Yaegashi, N1
Okamura, K1

Other Studies

3 other studies available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Endometriosis

ArticleYear
Lipopolysaccharide promoted proliferation and invasion of endometriotic stromal cells via induction of cyclooxygenase-2 expression.
    Fertility and sterility, 2010, Volume: 93, Issue:1

    Topics: Case-Control Studies; Cell Movement; Cell Proliferation; Cells, Cultured; Cyclooxygenase 2; Cyclooxy

2010
Suppression of migratory/invasive ability and induction of apoptosis in adenomyosis-derived mesenchymal stem cells by cyclooxygenase-2 inhibitors.
    Fertility and sterility, 2010, Volume: 94, Issue:6

    Topics: Adult; Apoptosis; Cell Adhesion; Cell Movement; Cells, Cultured; Cyclooxygenase 2 Inhibitors; Down-R

2010
A selective cyclooxygenase-2 inhibitor suppresses the growth of endometriosis xenografts via antiangiogenic activity in severe combined immunodeficiency mice.
    Fertility and sterility, 2006, Volume: 86, Issue:4 Suppl

    Topics: Adult; Angiogenesis Inhibitors; Animals; Apoptosis; Blood Vessels; Cell Proliferation; Cyclooxygenas

2006