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n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Corneal Neovascularization

n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with Corneal Neovascularization in 1 studies

N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide: structure given in first source
NS-398 : A C-nitro compound that is N-methylsulfonyl-4-nitroaniline bearing an additional cyclohexyloxy substituent at position 2.

Corneal Neovascularization: New blood vessels originating from the corneal blood vessels and extending from the limbus into the adjacent CORNEAL STROMA. Neovascularization in the superficial and/or deep corneal stroma is a sequel to numerous inflammatory diseases of the ocular anterior segment, such as TRACHOMA, viral interstitial KERATITIS, microbial KERATOCONJUNCTIVITIS, and the immune response elicited by CORNEAL TRANSPLANTATION.

Research Excerpts

ExcerptRelevanceReference
"Both NS-398 and indomethacin significantly inhibited corneal neovascularization with the % inhibition of 36."3.70The effect of selective cyclooxygenase-2 inhibitor on corneal angiogenesis in the rat. ( Kawai, M; Kawai, Y; Mashima, Y; Yamada, M, 1999)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (100.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Yamada, M1
Kawai, M1
Kawai, Y1
Mashima, Y1

Other Studies

1 other study available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and Corneal Neovascularization

ArticleYear
The effect of selective cyclooxygenase-2 inhibitor on corneal angiogenesis in the rat.
    Current eye research, 1999, Volume: 19, Issue:4

    Topics: Administration, Topical; Animals; Cornea; Corneal Neovascularization; Cyclooxygenase 2; Cyclooxygena

1999