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n(g),n(g')-dimethyl-l-arginine and Myocarditis

n(g),n(g')-dimethyl-l-arginine has been researched along with Myocarditis in 1 studies

N,N-dimethylarginine: asymmetric dimethylarginine; do not confuse with N,N'-dimethylarginine

Myocarditis: Inflammatory processes of the muscular walls of the heart (MYOCARDIUM) which result in injury to the cardiac muscle cells (MYOCYTES, CARDIAC). Manifestations range from subclinical to sudden death (DEATH, SUDDEN). Myocarditis in association with cardiac dysfunction is classified as inflammatory CARDIOMYOPATHY usually caused by INFECTION, autoimmune diseases, or responses to toxic substances. Myocarditis is also a common cause of DILATED CARDIOMYOPATHY and other cardiomyopathies.

Research Excerpts

ExcerptRelevanceReference
" The reduced bioavailability of endothelium-derived nitric oxide (NO) may play a role in endothelial vasodilator dysfunction and the structural changes that are characteristic of GCAD."5.33Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease. ( Cooke, JP; Gunawan, F; Jacobi, J; Robbins, RC; Sydow, K; Tanaka, M; Tsao, PS, 2005)
" The reduced bioavailability of endothelium-derived nitric oxide (NO) may play a role in endothelial vasodilator dysfunction and the structural changes that are characteristic of GCAD."1.33Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease. ( Cooke, JP; Gunawan, F; Jacobi, J; Robbins, RC; Sydow, K; Tanaka, M; Tsao, PS, 2005)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Tanaka, M1
Sydow, K1
Gunawan, F1
Jacobi, J1
Tsao, PS1
Robbins, RC1
Cooke, JP1

Other Studies

1 other study available for n(g),n(g')-dimethyl-l-arginine and Myocarditis

ArticleYear
Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease.
    Circulation, 2005, Sep-13, Volume: 112, Issue:11

    Topics: Amidohydrolases; Animals; Arginine; Coronary Artery Disease; Cytokines; Enzyme Inhibitors; Female; H

2005