Page last updated: 2024-10-17

n(1)-methylnicotinamide and Inflammation

n(1)-methylnicotinamide has been researched along with Inflammation in 2 studies

N(1)-methylnicotinamide: RN given refers to parent cpd
1-methylnicotinamide : A pyridinium ion comprising nicotinamide having a methyl group at the 1-position. It is a metabolite of nicotinamide which was initially considered to be biologically inactive but has emerged as an anti-thrombotic and anti-inflammatory agent.

Inflammation: A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function.

Research Excerpts

ExcerptRelevanceReference
" administration of D-galactosamine, and it rather showed a decrease together with necrosis after carbon tetrachloride administration."3.68N1-methylnicotinamide level in the blood after nicotinamide loading as further evidence for malignant tumor burden. ( Fujimura, S; Kato, N; Miyazaki, M; Moriyama, Y; Nakagawa, K; Okui, K, 1991)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Przygodzki, T1
Kazmierczak, P1
Sikora, J1
Watala, C1
Nakagawa, K1
Miyazaki, M1
Okui, K1
Kato, N1
Moriyama, Y1
Fujimura, S1

Other Studies

2 other studies available for n(1)-methylnicotinamide and Inflammation

ArticleYear
1-methylnicotinamide effects on the selected markers of endothelial function, inflammation and haemostasis in diabetic rats.
    European journal of pharmacology, 2010, Aug-25, Volume: 640, Issue:1-3

    Topics: Animals; Biomarkers; Diabetes Mellitus; Endothelium, Vascular; Hemostasis; Hypolipidemic Agents; Inf

2010
N1-methylnicotinamide level in the blood after nicotinamide loading as further evidence for malignant tumor burden.
    Japanese journal of cancer research : Gann, 1991, Volume: 82, Issue:11

    Topics: Animals; Carbon Tetrachloride Poisoning; Cells, Cultured; DNA Replication; Galactosamine; Inflammati

1991