n(1)-methylnicotinamide has been researched along with Hepatitis in 2 studies
N(1)-methylnicotinamide: RN given refers to parent cpd
1-methylnicotinamide : A pyridinium ion comprising nicotinamide having a methyl group at the 1-position. It is a metabolite of nicotinamide which was initially considered to be biologically inactive but has emerged as an anti-thrombotic and anti-inflammatory agent.
Hepatitis: INFLAMMATION of the LIVER.
Excerpt | Relevance | Reference |
---|---|---|
" The aim of this work was to characterize the profile of release of endogenous MNA during the initiation and progression of murine hepatitis induced by Concanavalin A (ConA)." | 7.81 | Differential involvement of IL-6 in the early and late phase of 1-methylnicotinamide (MNA) release in Concanavalin A-induced hepatitis. ( Chlopicki, S; Czarnowska, E; Jakubowski, A; Jasztal, A; Kaminski, K; Sitek, B; Slominska, EM; Smolenski, RT; Sternak, M; Szafarz, M; Walczak, M; Wojcik, T, 2015) |
" In this study, we demonstrated that IL-10-producing macrophages contribute to immune tolerance in the inflamed liver under intestinal barrier disruption in a murine tandem model of dextran sulfate sodium (DSS) colitis and concanavalin A (Con A) hepatitis." | 3.88 | Intestinal barrier regulates immune responses in the liver via IL-10-producing macrophages. ( Amiya, T; Chu, PS; Ebinuma, H; Fukuda, S; Kanai, T; Katayama, T; Mikami, Y; Miyake, R; Nakamoto, N; Shiba, S; Suzuki, T; Taniki, N; Teratani, T; Tsukimi, T; Yamaguchi, A, 2018) |
" The aim of this work was to characterize the profile of release of endogenous MNA during the initiation and progression of murine hepatitis induced by Concanavalin A (ConA)." | 3.81 | Differential involvement of IL-6 in the early and late phase of 1-methylnicotinamide (MNA) release in Concanavalin A-induced hepatitis. ( Chlopicki, S; Czarnowska, E; Jakubowski, A; Jasztal, A; Kaminski, K; Sitek, B; Slominska, EM; Smolenski, RT; Sternak, M; Szafarz, M; Walczak, M; Wojcik, T, 2015) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Taniki, N | 1 |
Nakamoto, N | 1 |
Chu, PS | 1 |
Mikami, Y | 1 |
Amiya, T | 1 |
Teratani, T | 1 |
Suzuki, T | 1 |
Tsukimi, T | 1 |
Fukuda, S | 1 |
Yamaguchi, A | 1 |
Shiba, S | 1 |
Miyake, R | 1 |
Katayama, T | 1 |
Ebinuma, H | 1 |
Kanai, T | 1 |
Sternak, M | 1 |
Jakubowski, A | 1 |
Czarnowska, E | 1 |
Slominska, EM | 1 |
Smolenski, RT | 1 |
Szafarz, M | 1 |
Walczak, M | 1 |
Sitek, B | 1 |
Wojcik, T | 1 |
Jasztal, A | 1 |
Kaminski, K | 1 |
Chlopicki, S | 1 |
2 other studies available for n(1)-methylnicotinamide and Hepatitis
Article | Year |
---|---|
Intestinal barrier regulates immune responses in the liver via IL-10-producing macrophages.
Topics: Animals; Chemical and Drug Induced Liver Injury; Colitis; Concanavalin A; Dextran Sulfate; Disease M | 2018 |
Differential involvement of IL-6 in the early and late phase of 1-methylnicotinamide (MNA) release in Concanavalin A-induced hepatitis.
Topics: Acute-Phase Reaction; Aldehyde Oxidase; Animals; Chemical and Drug Induced Liver Injury; Concanavali | 2015 |