Page last updated: 2024-11-02

moxonidine and Fibrosis

moxonidine has been researched along with Fibrosis in 2 studies

moxonidine: structure given in first source

Fibrosis: Any pathological condition where fibrous connective tissue invades any organ, usually as a consequence of inflammation or other injury.

Research Excerpts

ExcerptRelevanceReference
"6 hamsters (6 and 10 months old, with moderate and advanced heart failure, respectively) received moxonidine at 2 concentrations: low (2."3.77Functional and molecular effects of imidazoline receptor activation in heart failure. ( Abdel Rahman, AA; Aceros, H; Mukaddam-Daher, S; Noiseux, N; Stabile, AM; Stockmeyer, K, 2011)
"Regression of left ventricular hypertrophy by moxonidine, a centrally acting sympatholytic imidazoline compound, results from a sustained reduction of DNA synthesis and transient stimulation of DNA fragmentation."3.77Moxonidine improves cardiac structure and performance in SHR through inhibition of cytokines, p38 MAPK and Akt. ( Aceros, H; Cobos-Puc, L; Farah, G; Mukaddam-Daher, S; Noiseux, N; Stabile, AM, 2011)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Stabile, AM2
Aceros, H2
Stockmeyer, K1
Abdel Rahman, AA1
Noiseux, N2
Mukaddam-Daher, S2
Farah, G1
Cobos-Puc, L1

Other Studies

2 other studies available for moxonidine and Fibrosis

ArticleYear
Functional and molecular effects of imidazoline receptor activation in heart failure.
    Life sciences, 2011, Mar-14, Volume: 88, Issue:11-12

    Topics: Animals; Apoptosis; Blotting, Western; Collagen; Cricetinae; Cytokines; Disease Models, Animal; Dose

2011
Moxonidine improves cardiac structure and performance in SHR through inhibition of cytokines, p38 MAPK and Akt.
    British journal of pharmacology, 2011, Volume: 164, Issue:3

    Topics: Animals; Antihypertensive Agents; Blood Pressure; Cell Death; Cell Survival; Collagen; Cytokines; Ec

2011