molybdenum has been researched along with ALS - Amyotrophic Lateral Sclerosis in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Christopher A, B | 1 |
| Ishige, K; Ito, Y; Okawa, E; Ono, S; Suzuki, T; Tokuda, E; Watanabe, S | 1 |
| Marklund, SL; Okawa, E; Ono, S; Tokuda, E; Watanabe, S | 1 |
3 other study(ies) available for molybdenum and ALS - Amyotrophic Lateral Sclerosis
| Article | Year |
|---|---|
Inosine supplements only reach the CNS in molybdenum deficient humans and may cause astrocyte degeneration and bulbar-respiratory disease.
Topics: Amyotrophic Lateral Sclerosis; Astrocytes; Humans; Inosine; Molybdenum; Respiration Disorders | 2022 |
Ammonium tetrathiomolybdate delays onset, prolongs survival, and slows progression of disease in a mouse model for amyotrophic lateral sclerosis.
Topics: Amino Acid Sequence; Amyotrophic Lateral Sclerosis; Animals; Chelating Agents; Copper; Cysteine; Disease Models, Animal; Disease Progression; Enzyme Inhibitors; Female; Humans; Lipid Peroxidation; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Molybdenum; Spinal Cord; Superoxide Dismutase; Survival Rate; Treatment Outcome | 2008 |
Dysregulation of intracellular copper homeostasis is common to transgenic mice expressing human mutant superoxide dismutase-1s regardless of their copper-binding abilities.
Topics: Amyotrophic Lateral Sclerosis; Animals; Chelating Agents; Copper; Disease Models, Animal; Homeostasis; Humans; Mice; Mice, Transgenic; Molybdenum; Motor Neurons; Mutation; Spinal Cord; Superoxide Dismutase; Superoxide Dismutase-1 | 2013 |