mifepristone and Osteosarcoma
mifepristone has been researched along with Osteosarcoma in 5 studies
Research
Studies (5)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (80.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 1 (20.00) | 2.80 |
Authors
Authors | Studies |
---|---|
Check, D; Check, JH; Poretta, T; Wilson, C | 1 |
Kidani, T; Mashima, N; Masuno, H; Miyazaki, T; Sakayama, K; Yamamoto, H | 1 |
Archer, TK; Fryer, CJ; Garabedian, MJ; Kinyamu, HK; Rogatsky, I | 1 |
Liu, C; Watson, CE; Werth, VP; Williams, KJ; Zhang, W | 1 |
Rogatsky, I; Yamamoto, KR; Zarember, KA | 1 |
Other Studies
5 other study(ies) available for mifepristone and Osteosarcoma
Article | Year |
---|---|
Palliative Benefits of Oral Mifepristone for the Treatment of Metastatic Fibroblastic Osteosarcoma.
Topics: Administration, Oral; Bone Neoplasms; Cancer Pain; Humans; Male; Middle Aged; Mifepristone; Neoplasm Metastasis; Osteosarcoma; Palliative Care; Quality of Life; Tibia; Treatment Outcome | 2021 |
Effect of cortisol on cell proliferation and the expression of lipoprotein lipase and vascular endothelial growth factor in a human osteosarcoma cell line.
Topics: Blotting, Western; Bone Neoplasms; Cell Line, Tumor; Cell Proliferation; DNA Replication; Hormone Antagonists; Humans; Hydrocortisone; Immunohistochemistry; Lipoprotein Lipase; Mifepristone; Osteosarcoma; Thymidine; Vascular Endothelial Growth Factor A | 2008 |
Selective activation of the glucocorticoid receptor by steroid antagonists in human breast cancer and osteosarcoma cells.
Topics: Animals; Breast Neoplasms; Dexamethasone; Female; Gene Expression Regulation, Neoplastic; Glucocorticoids; Hormone Antagonists; Humans; Hydroxycorticosteroids; Mammary Tumor Virus, Mouse; Mice; Mifepristone; Osteosarcoma; Promoter Regions, Genetic; Receptors, Glucocorticoid; Transcription, Genetic; Tumor Cells, Cultured | 2000 |
Glucocorticoids induce a near-total suppression of hyaluronan synthase mRNA in dermal fibroblasts and in osteoblasts: a molecular mechanism contributing to organ atrophy.
Topics: Atrophy; Blotting, Northern; Cell Nucleus; Cells, Cultured; Cloning, Molecular; Dactinomycin; Dexamethasone; Dose-Response Relationship, Drug; Fibroblasts; Glucocorticoids; Glucuronosyltransferase; Glycosyltransferases; Hormone Antagonists; Humans; Hyaluronan Synthases; Membrane Proteins; Mifepristone; Nucleic Acid Synthesis Inhibitors; Osteoblasts; Osteosarcoma; RNA, Messenger; Skin; Time Factors; Transcription, Genetic; Transferases; Tumor Cells, Cultured; Uridine; Xenopus Proteins | 2000 |
Factor recruitment and TIF2/GRIP1 corepressor activity at a collagenase-3 response element that mediates regulation by phorbol esters and hormones.
Topics: Animals; Collagenases; Gene Expression Regulation, Neoplastic; Hormone Antagonists; Hormones; Humans; Matrix Metalloproteinase 13; Mice; Mifepristone; Nuclear Receptor Coactivator 2; Osteosarcoma; Phorbol Esters; Protein Binding; Rats; Receptors, Glucocorticoid; Receptors, Thyroid Hormone; Repressor Proteins; Response Elements; Transcription Factor AP-1; Transcription Factors; Tumor Cells, Cultured | 2001 |