Page last updated: 2024-08-23

mifepristone and Hypoxia-Ischemia, Brain

mifepristone has been researched along with Hypoxia-Ischemia, Brain in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's1 (25.00)24.3611
2020's1 (25.00)2.80

Authors

AuthorsStudies
Alves, T; Andrade, L; Cardoso, A; Dulce Madeira, M; Pereira, PA; Rocha, R; Sá, S1
Bhatt, AJ; Feng, Y1
Hagberg, H; Ikeda, T; Ikenoue, T; Mallard, C; Yang, L1
Fan, XJ; Jiang, ZL; Ke, KF; Li, X; Peng, LL; Wang, GH; Zhang, YF1

Other Studies

4 other study(ies) available for mifepristone and Hypoxia-Ischemia, Brain

ArticleYear
Behavioral and brain morphological analysis of non-inflammatory and inflammatory rat models of preterm brain injury.
    Neurobiology of learning and memory, 2021, Volume: 185

    Topics: Animals; Brain; Disease Models, Animal; Female; Hippocampus; Hypoxia-Ischemia, Brain; Infant, Premature, Diseases; Male; Mifepristone; Morris Water Maze Test; Open Field Test; Pregnancy; Premature Birth; Rats; Rats, Wistar; Reflex; Spatial Memory

2021
Corticosteroid responses following hypoxic preconditioning provide neuroprotection against subsequent hypoxic-ischemic brain injury in the newborn rats.
    International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 2015, Volume: 44

    Topics: Adrenal Cortex Hormones; Analysis of Variance; Animals; Animals, Newborn; Brain Edema; Caspase 3; Female; Hormone Antagonists; Hypoxia-Ischemia, Brain; Ischemic Preconditioning; Male; Mifepristone; Pregnancy; Rats; Rats, Sprague-Dawley; Time Factors

2015
Endotoxin-induced hypoxic-ischemic tolerance is mediated by up-regulation of corticosterone in neonatal rat.
    Pediatric research, 2006, Volume: 59, Issue:1

    Topics: Animals; Animals, Newborn; Corticosterone; Hormone Antagonists; Hypoxia-Ischemia, Brain; Lipopolysaccharides; Mifepristone; Rats; Rats, Wistar; Up-Regulation

2006
Ginsenoside Rg1 protects neurons from hypoxic-ischemic injury possibly by inhibiting Ca2+ influx through NMDA receptors and L-type voltage-dependent Ca2+ channels.
    European journal of pharmacology, 2008, May-31, Volume: 586, Issue:1-3

    Topics: Animals; Calcium; Calcium Channels, L-Type; Cell Survival; Dose-Response Relationship, Drug; Ginsenosides; Glucose; Hippocampus; Hormone Antagonists; Hypoxia-Ischemia, Brain; Immunohistochemistry; Infarction, Middle Cerebral Artery; Magnetic Resonance Imaging; Male; Mifepristone; Neurons; Neuroprotective Agents; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Tetrazolium Salts; Thiazoles

2008