mifepristone has been researched along with Cachexia in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (66.67) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Agell, N; Argilés, JM; Carbó, N; Costelli, P; García-Martínez, C; Llovera, M; López-Soriano, FJ | 1 |
Daly, JM; Evoy, D; Fujita, J; Mackrell, P; McCarter, MD; Naama, HA; Rivadeneira, DE | 1 |
Gang, G; Hasselgren, PO; Penner, G; Sun, X; Wray, C | 1 |
3 other study(ies) available for mifepristone and Cachexia
Article | Year |
---|---|
Muscle hypercatabolism during cancer cachexia is not reversed by the glucocorticoid receptor antagonist RU38486.
Topics: Animals; Cachexia; Corticosterone; Glucocorticoids; Hormone Antagonists; Liver Neoplasms, Experimental; Male; Mifepristone; Muscle, Skeletal; Neoplasm Proteins; Rats; Rats, Wistar; Receptors, Glucocorticoid; RNA, Messenger; Ubiquitins | 1996 |
Glucocorticoid blockade does not abrogate tumor-induced cachexia.
Topics: Adenocarcinoma; Animals; Body Composition; Body Weight; Cachexia; Colonic Neoplasms; Female; Glucocorticoids; Hormone Antagonists; Interleukin-6; Mice; Mice, Inbred BALB C; Mice, Inbred DBA; Mifepristone; Neoplasm Transplantation; Regression Analysis; Tumor Cells, Cultured | 1999 |
C/EBP DNA-binding activity is upregulated by a glucocorticoid-dependent mechanism in septic muscle.
Topics: Animals; Cachexia; CCAAT-Enhancer-Binding Protein-beta; CCAAT-Enhancer-Binding Protein-delta; CCAAT-Enhancer-Binding Proteins; Cell Nucleus; Cysteine Endopeptidases; DNA; Electrophoretic Mobility Shift Assay; Gene Expression; Glucocorticoids; Hormone Antagonists; Male; Mifepristone; Multienzyme Complexes; Muscle, Skeletal; Proteasome Endopeptidase Complex; Rats; Rats, Sprague-Dawley; Sepsis; Transcription Factors; Ubiquitin; Up-Regulation | 2002 |