mifepristone has been researched along with Blood Poisoning in 19 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 6 (31.58) | 18.2507 |
| 2000's | 10 (52.63) | 29.6817 |
| 2010's | 3 (15.79) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Alamdari, N; Aversa, Z; Hasselgren, PO; Smith, IJ | 1 |
| Alamdari, N; Aversa, Z; Hasselgren, PO; Petkova, V; Smith, IJ | 1 |
| Alamdari, N; Aversa, Z; Castillero, E; Hasselgren, PO; Jasuja, R; Larsson, L; Qaisar, R; Renaud, G; Smith, I; Toraldo, G | 1 |
| Hasselgren, PO; Robb, BW; Tomkinson, B; Wray, CJ | 1 |
| Chai, J; Shen, C; Sheng, Z | 1 |
| Brathwaite, CE; Chen, E; El-Maghrabi, MR; Maitra, SR; Rosa, D; Valane, PD | 1 |
| Mammen, JM; Sun, X; Tian, X | 1 |
| Chen, L; Da, J; Hedenstierna, G | 1 |
| Ayala, A; Chen, Y; Chung, CS; Doughty, L; Grutkoski, PS | 1 |
| Argilés, JM; del Santo, B; García-Martínez, C; Llovera, M; López-Soriano, FJ | 1 |
| Ayala, A; Ayala, CA; Chaudry, IH; DeMaso, CM; Herdon, CD; Lehman, DL | 1 |
| Fagan, J; Fischer, JE; Hasselgren, PO; Lieberman, M; Roegner, V; Tiao, G; Wang, JJ | 1 |
| Ayala, A; Ayala, CA; Chaudry, IH; Herdon, CD; Lehman, DL | 1 |
| Jacobs, DO; Mitsuo, T; Prechek, D; Rounds, J; Wilmore, DW | 1 |
| Ayala, A; Chaudry, IH; Chung, CS; Xu, YX | 1 |
| Fischer, D; Gang, G; Hasselgren, PO; Pritts, T; Sun, X | 1 |
| Fischer, JE; Gang, G; Hasselgren, PO; Penner, CG; Wray, C | 1 |
| Gang, G; Hasselgren, PO; Penner, G; Sun, X; Wray, C | 1 |
| Ashok, PW; Narvekar, NN; Smith, NC; Templeton, A; Wagaarachchi, PT | 1 |
19 other study(ies) available for mifepristone and Blood Poisoning
| Article | Year |
|---|---|
Sepsis and glucocorticoids upregulate p300 and downregulate HDAC6 expression and activity in skeletal muscle.
Topics: Animals; Dexamethasone; Disease Models, Animal; Down-Regulation; E1A-Associated p300 Protein; Gene Expression Regulation, Enzymologic; Glucocorticoids; Histone Deacetylase 6; Histone Deacetylase Inhibitors; Histone Deacetylases; Hormone Antagonists; Hydroxamic Acids; Male; Mifepristone; Muscle Proteins; Muscle, Skeletal; Muscular Atrophy; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; RNA, Messenger; Sepsis; Sirtuin 1; SKP Cullin F-Box Protein Ligases; Time Factors; Tripartite Motif Proteins; Ubiquitin-Protein Ligases; Up-Regulation | 2010 |
Sepsis downregulates myostatin mRNA levels without altering myostatin protein levels in skeletal muscle.
Topics: Activins; Animals; Dexamethasone; Down-Regulation; Fasting; Follistatin; Male; Mifepristone; Muscle Proteins; Muscle, Skeletal; Myostatin; Organ Size; Rats; Rats, Sprague-Dawley; RNA, Messenger; Sepsis; SKP Cullin F-Box Protein Ligases; Smad Proteins; Tripartite Motif Proteins; Ubiquitin-Protein Ligases | 2010 |
Loss of muscle strength during sepsis is in part regulated by glucocorticoids and is associated with reduced muscle fiber stiffness.
Topics: Actomyosin; Animals; Biomechanical Phenomena; Glucocorticoids; Male; Mifepristone; Muscle Fibers, Skeletal; Muscle Strength; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; Sepsis | 2012 |
Tripeptidyl-peptidase II expression and activity are increased in skeletal muscle during sepsis.
Topics: Aminopeptidases; Animals; Base Sequence; Blotting, Western; Dipeptidyl-Peptidases and Tripeptidyl-Peptidases; DNA Primers; Glucocorticoids; Hormone Antagonists; Hydrolysis; Male; Mifepristone; Muscle Proteins; Muscle, Skeletal; Polymerase Chain Reaction; Rats; Rats, Sprague-Dawley; RNA, Messenger; Sepsis; Serine Endopeptidases | 2002 |
[Changes in skeletal muscle protein metabolism in burned rats with sepsis and the role of glucocorticoid in skeletal muscle proteolysis].
Topics: Animals; Burns; Gene Expression Regulation; Glucocorticoids; Hydrocortisone; Male; Mifepristone; Muscle Proteins; Muscle, Skeletal; Rats; Rats, Wistar; Sepsis; Ubiquitin | 2002 |
Modulations of signal transduction pathways during sepsis and the effects of insulin and mifepristone.
Topics: Animals; Enzyme Activation; Insulin; Liver; Male; Mifepristone; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinases; Rats; Rats, Sprague-Dawley; Sepsis; Signal Transduction | 2003 |
Sepsis induces the transcription of the glucocorticoid receptor in skeletal muscle cells.
Topics: Animals; Cell Line; Corticosterone; Dexamethasone; Glucocorticoids; Hormone Antagonists; Hydrocortisone; Male; Mice; Mifepristone; Muscle Cells; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; Sepsis; Transcription, Genetic | 2003 |
Nitric oxide up-regulates the glucocorticoid receptor and blunts the inflammatory reaction in porcine endotoxin sepsis.
Topics: Administration, Inhalation; Animals; Anti-Inflammatory Agents; Blood Gas Analysis; Disease Models, Animal; Drug Evaluation, Preclinical; Drug Synergism; Drug Therapy, Combination; Hormone Antagonists; Hydrocortisone; Immunohistochemistry; Inflammation; Infusions, Intravenous; Lipopolysaccharides; Mifepristone; Nitric Oxide; Prospective Studies; Pulmonary Wedge Pressure; Random Allocation; Receptors, Glucocorticoid; Respiratory Distress Syndrome; Sepsis; Swine; Up-Regulation | 2007 |
SOCS-1 is a central mediator of steroid-increased thymocyte apoptosis and decreased survival following sepsis.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; Caspase 3; Dexamethasone; Male; Mice; Mice, Inbred C3H; Mifepristone; Mitochondria; Sepsis; Signal Transduction; Steroids; Suppressor of Cytokine Signaling 1 Protein; Suppressor of Cytokine Signaling Proteins; T-Lymphocytes; Thymus Gland | 2007 |
Lipopolysaccharide (LPS) increases the in vivo oxidation of branched-chain amino acids in the rat: a cytokine-mediated effect.
Topics: Amino Acids, Branched-Chain; Animals; Cytokines; Female; Interleukin-1; Lipopolysaccharides; Mifepristone; Muscle, Skeletal; Oxidation-Reduction; Rats; Rats, Wistar; Sepsis; Tumor Necrosis Factor-alpha | 1995 |
The induction of accelerated thymic programmed cell death during polymicrobial sepsis: control by corticosteroids but not tumor necrosis factor.
Topics: Adrenal Cortex Hormones; Animals; Apoptosis; Cells, Cultured; Male; Mice; Mice, Inbred C3H; Mifepristone; Sepsis; Thymus Gland; Tumor Necrosis Factor-alpha | 1995 |
Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids.
Topics: Animals; Base Sequence; Energy Metabolism; Gene Expression; Gene Expression Regulation; Glucocorticoids; Hormone Antagonists; Male; Mifepristone; Molecular Sequence Data; Muscle Proteins; Muscles; Oligonucleotide Probes; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; RNA, Messenger; Sepsis; Ubiquitins | 1996 |
Differential induction of apoptosis in lymphoid tissues during sepsis: variation in onset, frequency, and the nature of the mediators.
Topics: Animals; Apoptosis; Bone Marrow; Cell Cycle; Hematopoietic Stem Cells; Intestinal Perforation; Lymphoid Tissue; Male; Mice; Mice, Inbred C3H; Mifepristone; Receptors, Glucocorticoid; Receptors, Tumor Necrosis Factor; Receptors, Tumor Necrosis Factor, Type I; Sepsis; Spleen; Thymus Gland; Tumor Necrosis Factor Decoy Receptors; Tumor Necrosis Factor-alpha | 1996 |
Glucocorticoid receptor antagonism by mifepristone alters phosphocreatine breakdown during sepsis.
Topics: Adenosine Triphosphate; Animals; Corticosterone; Creatine; Creatine Kinase; Down-Regulation; Energy Metabolism; Hormone Antagonists; Hydrogen-Ion Concentration; Magnetic Resonance Spectroscopy; Male; Mifepristone; Muscle, Skeletal; Phosphates; Phosphocreatine; Phosphorus Isotopes; Placebos; Random Allocation; Rats; Rats, Wistar; Receptors, Glucocorticoid; Sepsis; Sodium-Potassium-Exchanging ATPase | 1996 |
Does Fas ligand or endotoxin contribute to thymic apoptosis during polymicrobial sepsis?
Topics: Animals; Apoptosis; bcl-X Protein; Endotoxins; Fas Ligand Protein; fas Receptor; Glucocorticoids; Hormone Antagonists; Ligation; Male; Membrane Glycoproteins; Mice; Mice, Inbred C3H; Mice, Inbred Strains; Mifepristone; Proto-Oncogene Proteins c-bcl-2; Sepsis; Species Specificity; Thymus Gland | 1999 |
The gene expression of ubiquitin ligase E3alpha is upregulated in skeletal muscle during sepsis in rats-potential role of glucocorticoids.
Topics: Animals; Base Sequence; DNA Primers; Gene Expression Regulation, Enzymologic; Glucocorticoids; Hormone Antagonists; Ligases; Male; Mifepristone; Muscle, Skeletal; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; RNA, Messenger; Sepsis; Ubiquitin-Protein Ligases; Up-Regulation | 2000 |
The transcription factors NF-kappab and AP-1 are differentially regulated in skeletal muscle during sepsis.
Topics: Animals; DNA; Gene Expression Regulation; Hormone Antagonists; I-kappa B Proteins; Kinetics; Male; Mifepristone; Muscle, Skeletal; NF-kappa B; Rats; Rats, Sprague-Dawley; Receptors, Glucocorticoid; Sepsis; Transcription Factor AP-1 | 2001 |
C/EBP DNA-binding activity is upregulated by a glucocorticoid-dependent mechanism in septic muscle.
Topics: Animals; Cachexia; CCAAT-Enhancer-Binding Protein-beta; CCAAT-Enhancer-Binding Protein-delta; CCAAT-Enhancer-Binding Proteins; Cell Nucleus; Cysteine Endopeptidases; DNA; Electrophoretic Mobility Shift Assay; Gene Expression; Glucocorticoids; Hormone Antagonists; Male; Mifepristone; Multienzyme Complexes; Muscle, Skeletal; Proteasome Endopeptidase Complex; Rats; Rats, Sprague-Dawley; Sepsis; Transcription Factors; Ubiquitin; Up-Regulation | 2002 |
Medical management of late intrauterine death using a combination of mifepristone and misoprostol.
Topics: Abortifacient Agents, Nonsteroidal; Abortifacient Agents, Steroidal; Abortion, Induced; Administration, Intravaginal; Adult; Drug Therapy, Combination; Female; Fetal Death; Gestational Age; Humans; Mifepristone; Misoprostol; Pregnancy; Scotland; Sepsis; Ultrasonography | 2002 |