midazolam has been researched along with Aphasia in 4 studies
Midazolam: A short-acting hypnotic-sedative drug with anxiolytic and amnestic properties. It is used in dentistry, cardiac surgery, endoscopic procedures, as preanesthetic medication, and as an adjunct to local anesthesia. The short duration and cardiorespiratory stability makes it useful in poor-risk, elderly, and cardiac patients. It is water-soluble at pH less than 4 and lipid-soluble at physiological pH.
midazolam : An imidazobenzodiazepine that is 4H-imidazo[1,5-a][1,4]benzodiazepine which is substituted by a methyl, 2-fluorophenyl and chloro groups at positions 1, 6 and 8, respectively.
Aphasia: A cognitive disorder marked by an impaired ability to comprehend or express language in its written or spoken form. This condition is caused by diseases which affect the language areas of the dominant hemisphere. Clinical features are used to classify the various subtypes of this condition. General categories include receptive, expressive, and mixed forms of aphasia.
Excerpt | Relevance | Reference |
---|---|---|
" Each patient underwent baseline testing for motor function, aphasia, and left hemispatial neglect, after which intravenous midazolam was delivered until mild drowsiness was detected." | 9.10 | Reemergence of stroke deficits with midazolam challenge. ( Berman, MF; Bustillo, MA; Fitzsimmons, BF; Lazar, RM; Marshall, RS; Mohr, JP; Robinson, JV; Shah, J; Young, WL, 2002) |
" Each patient underwent baseline testing for motor function and aphasia, after which intravenous midazolam was delivered until mild drowsiness was detected." | 5.10 | Midazolam challenge reinduces neurological deficits after transient ischemic attack. ( Berman, MF; Fitzsimmons, BF; Lazar, RM; Marshall, RS; Mohr, JP, 2003) |
" Each patient underwent baseline testing for motor function, aphasia, and left hemispatial neglect, after which intravenous midazolam was delivered until mild drowsiness was detected." | 5.10 | Reemergence of stroke deficits with midazolam challenge. ( Berman, MF; Bustillo, MA; Fitzsimmons, BF; Lazar, RM; Marshall, RS; Mohr, JP; Robinson, JV; Shah, J; Young, WL, 2002) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Shijo, T | 1 |
Kanno, S | 1 |
Shibuya, S | 1 |
Oikawa, T | 1 |
Ohnuma, A | 1 |
Mochizuki, H | 1 |
Lazar, RM | 3 |
Berman, MF | 3 |
Festa, JR | 1 |
Geller, AE | 1 |
Matejovsky, TG | 1 |
Marshall, RS | 3 |
Fitzsimmons, BF | 2 |
Mohr, JP | 2 |
Bustillo, MA | 1 |
Young, WL | 1 |
Shah, J | 1 |
Robinson, JV | 1 |
2 trials available for midazolam and Aphasia
Article | Year |
---|---|
Midazolam challenge reinduces neurological deficits after transient ischemic attack.
Topics: Adult; Aged; Aphasia; Dysarthria; Female; GABA Agonists; GABA-A Receptor Agonists; Humans; Ischemic | 2003 |
Reemergence of stroke deficits with midazolam challenge.
Topics: Adult; Aged; Aphasia; Female; GABA Modulators; Humans; Hypnotics and Sedatives; Male; Midazolam; Mid | 2002 |
2 other studies available for midazolam and Aphasia
Article | Year |
---|---|
[Pure word deafness due to a localization-related seizure: a case study].
Topics: Adult; Aphasia; Cerebrum; Diffusion Magnetic Resonance Imaging; Electroencephalography; Epilepsies, | 2014 |
GABAergic but not anti-cholinergic agents re-induce clinical deficits after stroke.
Topics: Aged; Analysis of Variance; Aphasia; Cholinergic Antagonists; Cross-Over Studies; Double-Blind Metho | 2010 |