mevalonolactone has been researched along with Hyper IgD Syndrome in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bida, M; Kiabilua, O; Pillay, TS; Satekge, TM; Terblanche, AJ | 1 |
| Celsi, F; Crovella, S; Gratton, R; Tricarico, PM | 1 |
| Amaral, AU; Cecatto, C; da Silva, JC; Godoy, KDS; Gonçalves, AM; Ribeiro, RT; Vargas, CR; Wajner, A; Wajner, M | 1 |
3 other study(ies) available for mevalonolactone and Hyper IgD Syndrome
| Article | Year |
|---|---|
Mevalonate kinase deficiency masked by cytomegalovirus infection and obscure liver disease.
Topics: alpha 1-Antitrypsin Deficiency; Cytomegalovirus Infections; Diagnosis, Differential; Humans; Hyperbilirubinemia; Infant; Liver Diseases; Male; Mevalonate Kinase Deficiency; Mevalonic Acid | 2019 |
Prolonged treatment with mevalonolactone induces oxidative stress response with reactive oxygen species production, mitochondrial depolarization and inflammation in human glioblastoma U-87 MG cells.
Topics: Glioblastoma; Humans; Inflammation; Mevalonate Kinase Deficiency; Mevalonic Acid; Oxidative Stress; Phenotype; Phosphotransferases (Alcohol Group Acceptor); Reactive Oxygen Species | 2018 |
Mevalonolactone disrupts mitochondrial functions and induces permeability transition pore opening in rat brain mitochondria: Implications for the pathogenesis of mevalonic aciduria.
Topics: Animals; Brain; Liver; Membrane Potential, Mitochondrial; Mevalonate Kinase Deficiency; Mevalonic Acid; Mitochondria; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Rats; Rats, Wistar | 2017 |