methylproamine and Inflammation

methylproamine has been researched along with Inflammation* in 1 studies

Other Studies

1 other study(ies) available for methylproamine and Inflammation

Article	Year
[A case of inflammatory cerebral amyloid angiopathy with white matter lesions appearing after brain biopsy]. Rinsho shinkeigaku = Clinical neurology, 2021, Mar-25, Volume: 61, Issue:3 A 76-year-old woman with a 7-year history of dementia presented to our hospital with generalized convulsive seizure for the first time. Contrast-enhanced brain magnetic resonance imaging revealed leptomeningeal enhancement mainly in the right occipital lobe and multiple lobar microbleeds in the bilateral cerebral and cerebellar subcortex. No white matter lesions were observed. A brain biopsy of the right parieto-occipital lobe revealed cerebral amyloid angiopathy (CAA). White matter lesions appeared in the right parieto-occipital lobe three days after the biopsy, and we considered inflammatory CAA. Three courses of methylprednisolone pulse followed by oral prednisolone therapy gradually reduced leptomeningeal and white matter lesions. An apolipoprotein E genotype investigation identified the ε2/ε3 genotype. In patients with inflammatory CAA, a risk of exacerbation should be considered after brain biopsy, in which the ε2 allele might play a role. Topics: Administration, Oral; Aged; Alleles; Apolipoproteins E; Benzimidazoles; Biopsy; Cerebral Amyloid Angiopathy; Disease Progression; Female; Genotype; Humans; Inflammation; Leukoencephalopathies; Occipital Lobe; Parietal Lobe; Prednisolone; Pulse Therapy, Drug; White Matter	2021

Article

Year

[A case of inflammatory cerebral amyloid angiopathy with white matter lesions appearing after brain biopsy].

Rinsho shinkeigaku = Clinical neurology, 2021, Mar-25, Volume: 61, Issue:3

A 76-year-old woman with a 7-year history of dementia presented to our hospital with generalized convulsive seizure for the first time. Contrast-enhanced brain magnetic resonance imaging revealed leptomeningeal enhancement mainly in the right occipital lobe and multiple lobar microbleeds in the bilateral cerebral and cerebellar subcortex. No white matter lesions were observed. A brain biopsy of the right parieto-occipital lobe revealed cerebral amyloid angiopathy (CAA). White matter lesions appeared in the right parieto-occipital lobe three days after the biopsy, and we considered inflammatory CAA. Three courses of methylprednisolone pulse followed by oral prednisolone therapy gradually reduced leptomeningeal and white matter lesions. An apolipoprotein E genotype investigation identified the ε2/ε3 genotype. In patients with inflammatory CAA, a risk of exacerbation should be considered after brain biopsy, in which the ε2 allele might play a role.

Topics: Administration, Oral; Aged; Alleles; Apolipoproteins E; Benzimidazoles; Biopsy; Cerebral Amyloid Angiopathy; Disease Progression; Female; Genotype; Humans; Inflammation; Leukoencephalopathies; Occipital Lobe; Parietal Lobe; Prednisolone; Pulse Therapy, Drug; White Matter

2021