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methylazoxymethanol acetate and Cognition Disorders

methylazoxymethanol acetate has been researched along with Cognition Disorders in 12 studies

Research

Studies (12)

TimeframeStudies, this research(%)All Research%
pre-19902 (16.67)18.7374
1990's2 (16.67)18.2507
2000's3 (25.00)29.6817
2010's5 (41.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Elefant, F; Gao, WJ; Gulchina, Y; Snyder, MA; Xu, SJ1
Feng, GF; Hu, M; Li, CJ; Liu, JX; Liu, Y; Xu, JH; Yuan, B; Zhu, K1
Jellett, AP; Jenks, K; Lucas, M; Scott, RC1
Giermaziak, W; Kus, K; Murawiecka, P; Nowakowska, E; Ratajczak, P; Słodzińska, I1
Ferguson, BR; Gao, WJ; Gulchina, Y; Hu, XQ; Li, F; Li, ML; Li, YC; Monaco, SA; Xing, B1
Didriksen, M; Flagstad, P; Glenthøj, BY1
Geyer, MA; Ghajarnia, M; Grace, AA; Jentsch, JD; Moore, H1
Featherstone, RE; Fletcher, PJ; Kapur, S; Nobrega, JN; Rizos, Z1
Balduini, W; Cattabeni, F; Cimino, M; De Graan, PN; Di Luca, M; Gispen, WH; Merazzi, F1
Caputi, A; Cattabeni, FN; Cimino, M; Di Luca, M; Pastorino, L; Rurale, S1
Abbracchio, MP; Balduini, W; Cattabeni, F; Cecchini, T; Cimino, M; Gazzanelli, GC; Lombardelli, G; Peruzzi, G1
Archer, T; Jonsson, G; Minor, BG; Mohammed, AK; Söderberg, U; Sundström, E1

Other Studies

12 other study(ies) available for methylazoxymethanol acetate and Cognition Disorders

ArticleYear
Epigenetic mechanisms underlying NMDA receptor hypofunction in the prefrontal cortex of juvenile animals in the MAM model for schizophrenia.
    Journal of neurochemistry, 2017, Volume: 143, Issue:3

    Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Animals, Newborn; Cognition Disorders; Disease Models, Animal; Epigenesis, Genetic; Excitatory Amino Acid Agonists; Excitatory Postsynaptic Potentials; Female; Gene Expression Regulation, Developmental; In Vitro Techniques; Male; Methylazoxymethanol Acetate; N-Methylaspartate; Neurons; Patch-Clamp Techniques; Prefrontal Cortex; Pregnancy; Rats; Receptors, N-Methyl-D-Aspartate; Schizophrenia

2017
Ablation of aberrant neurogenesis fails to attenuate cognitive deficit of chronically epileptic mice.
    Epilepsy research, 2018, Volume: 142

    Topics: Animals; Bromodeoxyuridine; Cell Proliferation; Cognition Disorders; Disease Models, Animal; Doublecortin Domain Proteins; Green Fluorescent Proteins; Hippocampus; Homeodomain Proteins; Ki-67 Antigen; Male; Maze Learning; Methylazoxymethanol Acetate; Mice; Microtubule-Associated Proteins; Muscarinic Agonists; Neurogenesis; Neuropeptides; Pilocarpine; Proto-Oncogene Proteins c-fos; Status Epilepticus; Transduction, Genetic; Tumor Suppressor Proteins

2018
Standard dose valproic acid does not cause additional cognitive impact in a rodent model of intractable epilepsy.
    Epilepsy research, 2015, Volume: 110

    Topics: Animals; Animals, Newborn; Anticonvulsants; Cognition; Cognition Disorders; Disease Models, Animal; Epilepsy; Female; Flurothyl; Hippocampus; Malformations of Cortical Development; Maze Learning; Methylazoxymethanol Acetate; Pregnancy; Prenatal Exposure Delayed Effects; Random Allocation; Rats, Sprague-Dawley; Seizures; Survival Analysis; Valproic Acid

2015
Biochemical and cognitive impairments observed in animal models of schizophrenia induced by prenatal stress paradigm or methylazoxymethanol acetate administration.
    Acta neurobiologiae experimentalis, 2015, Volume: 75, Issue:3

    Topics: Analysis of Variance; Animals; Brain-Derived Neurotrophic Factor; Cognition Disorders; Corticosterone; Disease Models, Animal; Dose-Response Relationship, Drug; Escape Reaction; Female; Gestational Age; Male; Maze Learning; Methylazoxymethanol Acetate; Motor Activity; Neurotoxins; Pregnancy; Prenatal Exposure Delayed Effects; Rats; Rats, Wistar; Schizophrenia

2015
Juvenile treatment with a novel mGluR2 agonist/mGluR3 antagonist compound, LY395756, reverses learning deficits and cognitive flexibility impairments in adults in a neurodevelopmental model of schizophrenia.
    Neurobiology of learning and memory, 2017, Volume: 140

    Topics: Amino Acids, Dicarboxylic; Animals; Brain; Bridged Bicyclo Compounds; Cognition; Cognition Disorders; Disease Models, Animal; Learning; Methylazoxymethanol Acetate; Rats; Rats, Sprague-Dawley; Receptors, Metabotropic Glutamate; Receptors, N-Methyl-D-Aspartate; Schizophrenia

2017
Cognitive deficits caused by late gestational disruption of neurogenesis in rats: a preclinical model of schizophrenia.
    Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 2005, Volume: 30, Issue:2

    Topics: Animals; Association Learning; Cognition Disorders; Conditioning, Operant; Disease Models, Animal; Exploratory Behavior; Female; Maze Learning; Methylazoxymethanol Acetate; Nervous System; Pregnancy; Prenatal Exposure Delayed Effects; Psychomotor Performance; Rats; Rats, Wistar; Recognition, Psychology; Reversal Learning; Schizophrenia; Swimming; Teratogens

2005
A neurobehavioral systems analysis of adult rats exposed to methylazoxymethanol acetate on E17: implications for the neuropathology of schizophrenia.
    Biological psychiatry, 2006, Aug-01, Volume: 60, Issue:3

    Topics: Amphetamine; Analysis of Variance; Animals; Behavior, Animal; Brain; Central Nervous System Stimulants; Cognition Disorders; Disease Models, Animal; Dyskinesia, Drug-Induced; Female; Gait Disorders, Neurologic; Methylazoxymethanol Acetate; Neurons; Neurotoxins; Pregnancy; Prenatal Exposure Delayed Effects; Rats; Rats, Inbred F344; Schizophrenia

2006
Gestational methylazoxymethanol acetate treatment impairs select cognitive functions: parallels to schizophrenia.
    Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 2007, Volume: 32, Issue:2

    Topics: Animals; Atrophy; Brain; Brain Damage, Chronic; Cognition Disorders; Discrimination Learning; Disease Models, Animal; Female; Learning Disabilities; Male; Methylazoxymethanol Acetate; Neuropsychological Tests; Pregnancy; Prenatal Exposure Delayed Effects; Psychomotor Performance; Rats; Reinforcement, Psychology; Schizophrenia; Teratogens

2007
Selective alteration in B-50/GAP-43 phosphorylation in brain areas of animals characterized by cognitive impairment.
    Brain research, 1993, Apr-02, Volume: 607, Issue:1-2

    Topics: Animals; Avoidance Learning; Behavior, Animal; Brain Chemistry; Cerebral Cortex; Cognition Disorders; Female; GAP-43 Protein; Hippocampus; In Situ Hybridization; Membrane Glycoproteins; Methylazoxymethanol Acetate; Nerve Tissue Proteins; Neuronal Plasticity; Phosphorylation; Pregnancy; Rats; Rats, Sprague-Dawley

1993
Differential translocation of protein kinase C isozymes in rats characterized by a chronic lack of LTP induction and cognitive impairment.
    FEBS letters, 1996, Sep-09, Volume: 393, Issue:1

    Topics: Animals; Cognition Disorders; Enzyme Induction; Female; Isoenzymes; Long-Term Potentiation; Methylazoxymethanol Acetate; Pregnancy; Protein Kinase C; Rats; Rats, Sprague-Dawley; Synapses

1996
Microencephalic rats as a model for cognitive disorders.
    Clinical neuropharmacology, 1986, Volume: 9 Suppl 3

    Topics: Animals; Azo Compounds; Cognition Disorders; Disease Models, Animal; Female; Gestational Age; Learning Disabilities; Methylazoxymethanol Acetate; Microcephaly; Motor Activity; Pregnancy; Rats; Reflex

1986
Selective attention and place navigation in rats treated prenatally with methylazoxymethanol.
    Brain research, 1986, Volume: 395, Issue:2

    Topics: Abnormalities, Drug-Induced; Animals; Attention; Azo Compounds; Brain; Cognition Disorders; Female; Inhibition, Psychological; Learning Disabilities; Methylazoxymethanol Acetate; Pregnancy; Prenatal Exposure Delayed Effects; Rats; Rats, Inbred Strains; Reaction Time; Space Perception

1986