metformin has been researched along with Stomach Neoplasms in 53 studies
Metformin: A biguanide hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. Metformin improves glycemic control by improving insulin sensitivity and decreasing intestinal absorption of glucose. (From Martindale, The Extra Pharmacopoeia, 30th ed, p289)
metformin : A member of the class of guanidines that is biguanide the carrying two methyl substituents at position 1.
Stomach Neoplasms: Tumors or cancer of the STOMACH.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Metformin may improve the prognosis in gastric adenocarcinoma, but the existing literature is limited and contradictory." | 8.02 | Improved prognosis in gastric adenocarcinoma among metformin users in a population-based study. ( Lagergren, J; Santoni, G; Xie, SH; Zheng, J, 2021) |
| " Associations between metformin use and gastric non-cardia and cardia adenocarcinomas were examined within two cohorts; a diabetes cohort of participants using anti-diabetes medications, and a matched cohort of common-medication users, where metformin non-users were frequency matched (10:1) with metformin users for sex and age." | 7.91 | Metformin use and risk of gastric adenocarcinoma in a Swedish population-based cohort study. ( Lagergren, J; Santoni, G; Xie, SH; Zheng, J, 2019) |
| "In the first population based study of metformin use in gastric cancer adenocarcinoma patients with previous diabetes, our findings suggest that metformin use might improve overall mortality." | 7.88 | Impact of metformin on gastric adenocarcinoma survival: A Belgian population based study. ( Cardwell, CR; Couttenier, A; De Schutter, H; Lacroix, O; Robert, A; Vaes, E, 2018) |
| "Metformin may improve the prognosis in gastric adenocarcinoma, but the existing literature is limited and contradictory." | 4.02 | Improved prognosis in gastric adenocarcinoma among metformin users in a population-based study. ( Lagergren, J; Santoni, G; Xie, SH; Zheng, J, 2021) |
| " Associations between metformin use and gastric non-cardia and cardia adenocarcinomas were examined within two cohorts; a diabetes cohort of participants using anti-diabetes medications, and a matched cohort of common-medication users, where metformin non-users were frequency matched (10:1) with metformin users for sex and age." | 3.91 | Metformin use and risk of gastric adenocarcinoma in a Swedish population-based cohort study. ( Lagergren, J; Santoni, G; Xie, SH; Zheng, J, 2019) |
| "Although prior studies showed metformin could reduce gastric cancer (GC) risk in patients with diabetes mellitus, they failed to adjust for Helicobacter pylori infection and glycemic control." | 3.91 | Metformin Use and Gastric Cancer Risk in Diabetic Patients After Helicobacter pylori Eradication. ( Chan, EW; Chen, L; Cheung, KS; Leung, WK; Seto, WK; Wong, AYS; Wong, ICK, 2019) |
| "In the first population based study of metformin use in gastric cancer adenocarcinoma patients with previous diabetes, our findings suggest that metformin use might improve overall mortality." | 3.88 | Impact of metformin on gastric adenocarcinoma survival: A Belgian population based study. ( Cardwell, CR; Couttenier, A; De Schutter, H; Lacroix, O; Robert, A; Vaes, E, 2018) |
| "The protective effect of statin against gastric cancer was also significant in patients with diabetes mellitus." | 2.82 | Aspirin, metformin, and statin use on the risk of gastric cancer: A nationwide population-based cohort study in Korea with systematic review and meta-analysis. ( Bang, CS; Kim, HH; Kim, J; Kim, JY; Kim, TJ; Lee, KJ; Park, CH; Seo, SI; Shin, WG; You, SC, 2022) |
| "Metformin has been reported to be associated with improved cancer prognosis when used in combination with chemotherapy and/or radiotherapy." | 2.82 | Association of Tumor Pathological Response with the Use of Metformin During Neoadjuvant Chemoradiotherapy in Rectal and Esophageal/Gastroesophageal Cancer Patients: a Systematic Review and Meta-analysis. ( Kitagawa, Y; Matsui, S; Okabayashi, K; Sakamoto, K; Seishima, R; Shigeta, K, 2022) |
| "Metformin could reduce the risk of gastric cancer in T2DM patients, particularly in Asian populations." | 2.66 | The effect of metformin on gastric cancer in patients with type 2 diabetes: a systematic review and meta-analysis. ( Li, C; Shuai, Y; Zhou, X, 2020) |
| "Metformin is a biguanide molecule used since 1957 to treat type 2 diabetes patients." | 2.61 | The therapeutic potential of metformin in gastric cancer. ( Bessède, E; Courtois, S; Lehours, P, 2019) |
| "Diabetes may increase the risk of gastric cancer through shared risk factors including obesity, insulin resistance, hyperinsulinemia and smoking." | 2.50 | Diabetes and gastric cancer: the potential links. ( Tseng, CH; Tseng, FH, 2014) |
| "Metformin is an oral biguanide used to treat diabetes." | 1.72 | Metformin and MiR-365 synergistically promote the apoptosis of gastric cancer cells via MiR-365-PTEN-AMPK axis. ( Dai, ZT; Huang, F; Huang, Y; Li, H; Li, HH; Li, JP; Li, T; Liao, XH; Wang, J; Xiang, Y; Zhang, HM; Zhou, J, 2022) |
| "In trastuzumab-resistant HER2-positive gastric cancer, glycolysis fluctuates with a circadian oscillation regulated by the BMAL1-CLOCK-PER1-HK2 axis, which can be disrupted with a metformin-based chronotherapy to overcome trastuzumab resistance." | 1.72 | Disrupting Circadian Rhythm via the PER1-HK2 Axis Reverses Trastuzumab Resistance in Gastric Cancer. ( Hu, K; Hu, X; Huang, Q; Jiang, Y; Liang, B; Liao, W; Ma, Z; Rong, X; Shi, M; Wang, C; Wang, J; Wang, Y; Wu, Q; Wu, Z; Xu, X; Yao, G; Zhang, S, 2022) |
| "Gastric cancer is one of the most common cancers worldwide." | 1.72 | Combination of metformin and oxaliplatin inhibits gastric cancer cell proliferation and induces apoptosis. ( Wang, J; Zhou, R; Zhu, M, 2022) |
| "Gastric cancer is often comorbid with hypertension and diabetes mellitus and increases the mortality risk." | 1.72 | Prognostic value of long-term antidiabetic and antihypertensive therapy in postoperative gastric cancer patients: the FIESTA study. ( Chen, X; Fan, Z; Hu, D; Lin, J; Lin, X; Lin, Y; Peng, F; Wang, L; Yan, X; Yu, J; Zhang, S, 2022) |
| " In conclusion, long-term use of aspirin was independently associated with reduced incidence and mortality of gastric cancer in the general population, but metformin or statin use was only associated with a reduction of gastric cancer mortality in patients with diabetes and in the general population, respectively." | 1.62 | Association of Aspirin, Metformin, and Statin Use with Gastric Cancer Incidence and Mortality: A Nationwide Cohort Study. ( Cho, MH; Jeong, SM; Shin, DW; Yoo, TG, 2021) |
| "The AGS gastric cancer cell line were pretreated with three different sub-toxic concentration of metformin and then treated with various concentrations of 5-FU and docetaxel." | 1.62 | Efficacy of Metformin and Chemotherapeutic Agents on the Inhibition of Colony Formation and Shh/Gli1 Pathway: Metformin/Docetaxel Versus Metformin/5-Fluorouracil. ( Fatehi-Agdam, M; Jeddi, F; Najafzadeh, N; Panahizadeh, R; Vatankhah, MA, 2021) |
| "Furthermore, the survival rate of gastric cancer patients was positively correlated with p-AMPK and negative with TGF-β1." | 1.62 | AMPK inhibits Smad3-mediated autoinduction of TGF-β1 in gastric cancer cells. ( Huang, D; Jiang, S; Li, C; Luo, L; Luo, Z; Yan, X; Zou, J, 2021) |
| "Stomach cancer is the most common cancer in Korea." | 1.56 | Metformin use reduced the risk of stomach cancer in diabetic patients in Korea: an analysis of Korean NHIS-HEALS database. ( Bae, YJ; Choi, EA; Hyun, HJ; Kang, HT; Kim, J; Kim, Y, 2020) |
| "Metformin use was an independent prognostic factor of overall survival, cancer recurrence, and peritoneal recurrence." | 1.51 | The Effect of Metformin on Prognosis in Patients With Locally Advanced Gastric Cancer Associated With Type 2 Diabetes Mellitus. ( Jung, YJ; Kim, JH; Lee, HH; Park, CH; Seo, HS, 2019) |
| "Gastric cancer is one of the most common malignant tumor types worldwide, with a high morbidity and associated mortality." | 1.51 | Metformin suppresses gastric cancer progression through calmodulin‑like protein 3 secreted from tumor‑associated fibroblasts. ( An, F; Cao, J; Chen, G; Liu, S; Tang, Z; Wu, Q; Yu, C; Zhan, Q; Zhang, S; Zhu, J, 2019) |
| "Metformin effects were evaluated on the proliferation and tumourigenic properties of the gastric CSCs from patient-derived primary tumour xenografts (PDXs) and cancer cell lines (MKN45, AGS and MKN74) in vitro in conventional 2 dimensional (2D) and in 3 dimensional (3D) culture systems, in which only CSCs are able to form tumourspheres and in mouse xenograft models in vivo." | 1.46 | Metformin targets gastric cancer stem cells. ( Bessède, E; Courtois, S; Durán, RV; Giraud, J; Izotte, J; Lehours, P; Mégraud, F; Sifré, E; Varon, C, 2017) |
| "To explore the effect of metformin combined with chemotherapeutic agents on gastric cancer cell line AGS, 24 patients with gastric cancer were tracked for treatment." | 1.46 | Effect of metformin combined with chemotherapeutic agents on gastric cancer cell line AGS. ( Wu, X, 2017) |
| "Metformin is an antidiabetic drug used in treatment of type 2 diabetes." | 1.46 | Metformin inhibits gastric cancer cells metastatic traits through suppression of epithelial-mesenchymal transition in a glucose-independent manner. ( Shamsara, M; Valaee, S; Yaghoobi, MM, 2017) |
| "Type 2 diabetes can elevate risk of gastric cancer and metformin, an anti-diabetic agent, has an inhibitory effect against gastric cancer cell in vitro." | 1.46 | Metformin Inhibits N-Methyl-N-Nitrosourea Induced Gastric Tumorigenesis in db/db Mice. ( Jian, Y; Sun, Y; Zhuang, S, 2017) |
| "HGC‑27 and MKN‑45 human gastric cancer cells were treated with metformin at different concentrations and for different durations." | 1.46 | Metformin suppresses the expression of Sonic hedgehog in gastric cancer cells. ( Chen, L; Huang, X; Li, P; Lu, C; Song, Z; Wei, B, 2017) |
| "Metformin has recently received attention as a potential anticancer treatment." | 1.43 | Cumulative Metformin Use and Its Impact on Survival in Gastric Cancer Patients After Gastrectomy. ( An, JY; Cheong, JH; Chung, HC; Heo, SJ; Hyung, WJ; Jeong, YH; Jung, I; Jung, M; Kim, HI; Kim, HS; Lee, CK; Noh, SH; Rha, SY, 2016) |
| "Patients with type 2 diabetes diagnosed during 1999-2005 and newly treated with metformin (n=287971, "ever users of metformin") or other antidiabetic drugs (n=16217, "never users of metformin") were followed until December 31, 2011." | 1.43 | Metformin reduces gastric cancer risk in patients with type 2 diabetes mellitus. ( Tseng, CH, 2016) |
| "Three differently-differentiated gastric cancer cell lines, MKN-28, SGC-7901 and BGC-823, along with one noncancerous gastric cell line GES-1 were used." | 1.42 | AMPK/mTOR-mediated inhibition of survivin partly contributes to metformin-induced apoptosis in human gastric cancer cell. ( Gong, H; Guo, S; Han, G; Liu, K; Wang, Y, 2015) |
| "Gastric cancer is the second most common cause of cancer-related death worldwide." | 1.42 | Clinicopathological significance of N-cadherin and VEGF in advanced gastric cancer brain metastasis and the effects of metformin in preclinical models. ( Chin, HM; Choi, HJ; Jun, KH; Jung, JH; Kim, SH; Kim, YI; Lee, JE; Yang, SH, 2015) |
| "Metformin treatment leaded to increased expression of pACC, which, in turn, resulted in the reduction of cell proliferation and colony formation of gastric cancer cells (P < 0." | 1.40 | Increased expression of phospho-acetyl-CoA carboxylase protein is an independent prognostic factor for human gastric cancer without lymph node metastasis. ( Chen, Y; Cui, H; Fang, W; Wang, J; Yu, D; Yu, G, 2014) |
| "Metformin use has been associated with a decreased incidence and mortality of various cancers." | 1.40 | Long-term metformin use reduces gastric cancer risk in type 2 diabetics without insulin treatment: a nationwide cohort study. ( Cho, SJ; Choi, IJ; Kim, CG; Kim, SY; Kim, YI; Kim, YW; Kook, MC; Lee, EK; Lee, YJ; Park, JH; Ryu, KW, 2014) |
| "We used MKN1, MKN45, and MKN74 human gastric cancer cell lines to study the effects of metformin on human gastric cancer cells." | 1.38 | The antidiabetic drug metformin inhibits gastric cancer cell proliferation in vitro and in vivo. ( Aritomo, Y; Gong, J; Himoto, T; Iwama, H; Kato, K; Kitanaka, A; Kobara, H; Kobayashi, M; Masaki, T; Mimura, S; Miyoshi, H; Mori, H; Murao, K; Nomura, K; Okano, K; Suzuki, Y; Tani, J, 2012) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (1.89) | 29.6817 |
| 2010's | 31 (58.49) | 24.3611 |
| 2020's | 21 (39.62) | 2.80 |
| Authors | Studies |
|---|---|
| Seo, SI | 1 |
| Park, CH | 2 |
| Kim, TJ | 1 |
| Bang, CS | 1 |
| Kim, JY | 1 |
| Lee, KJ | 1 |
| Kim, J | 2 |
| Kim, HH | 1 |
| You, SC | 1 |
| Shin, WG | 1 |
| Huang, F | 1 |
| Xiang, Y | 2 |
| Li, T | 1 |
| Huang, Y | 2 |
| Wang, J | 9 |
| Zhang, HM | 1 |
| Li, HH | 1 |
| Dai, ZT | 1 |
| Li, JP | 1 |
| Li, H | 5 |
| Zhou, J | 1 |
| Liao, XH | 1 |
| Huang, Q | 2 |
| Hu, X | 2 |
| Zhang, S | 5 |
| Jiang, Y | 2 |
| Yao, G | 1 |
| Hu, K | 1 |
| Xu, X | 2 |
| Liang, B | 1 |
| Wu, Q | 3 |
| Ma, Z | 1 |
| Wang, Y | 5 |
| Wang, C | 4 |
| Wu, Z | 2 |
| Rong, X | 1 |
| Liao, W | 2 |
| Shi, M | 1 |
| Zhu, M | 1 |
| Zhou, R | 1 |
| Sakamoto, K | 1 |
| Okabayashi, K | 1 |
| Matsui, S | 1 |
| Seishima, R | 1 |
| Shigeta, K | 1 |
| Kitagawa, Y | 1 |
| Wang, L | 3 |
| Hu, D | 1 |
| Fan, Z | 1 |
| Yu, J | 1 |
| Lin, Y | 1 |
| Chen, X | 2 |
| Lin, X | 1 |
| Yan, X | 2 |
| Lin, J | 1 |
| Peng, F | 1 |
| Lan, WH | 3 |
| Lin, TY | 3 |
| Yeh, JA | 3 |
| Feng, CL | 3 |
| Hsu, JT | 3 |
| Lin, HJ | 3 |
| Kuo, CJ | 3 |
| Lai, CH | 3 |
| Kao, HW | 1 |
| Tsai, KW | 1 |
| Lin, WC | 1 |
| Zhang, Z | 2 |
| Zheng, J | 3 |
| Xie, SH | 2 |
| Santoni, G | 2 |
| Lagergren, J | 2 |
| Seo, HS | 1 |
| Jung, YJ | 2 |
| Kim, JH | 1 |
| Lee, HH | 1 |
| Bae, WJ | 1 |
| Ahn, JM | 1 |
| Byeon, HE | 1 |
| Kim, S | 1 |
| Lee, D | 1 |
| Shuai, Y | 1 |
| Li, C | 3 |
| Zhou, X | 3 |
| Hyun, HJ | 1 |
| Choi, EA | 1 |
| Kim, Y | 4 |
| Bae, YJ | 1 |
| Kang, HT | 1 |
| Nguépy Keubo, FR | 1 |
| Mboua, PC | 1 |
| Djifack Tadongfack, T | 1 |
| Fokouong Tchoffo, E | 1 |
| Tasson Tatang, C | 1 |
| Ide Zeuna, J | 1 |
| Noupoue, EM | 1 |
| Tsoplifack, CB | 1 |
| Folefack, GO | 1 |
| Kettani, M | 1 |
| Bandelier, P | 1 |
| Huo, J | 1 |
| Yu, D | 2 |
| Arulsamy, N | 1 |
| AlAbbad, S | 1 |
| Sardot, T | 1 |
| Lekashvili, O | 1 |
| Decato, D | 1 |
| Lelj, F | 1 |
| Alexander Ross, JB | 1 |
| Rosenberg, E | 1 |
| Nazir, H | 1 |
| Muthuswamy, N | 1 |
| Louis, C | 1 |
| Jose, S | 1 |
| Prakash, J | 1 |
| Buan, MEM | 1 |
| Flox, C | 1 |
| Chavan, S | 1 |
| Shi, X | 1 |
| Kauranen, P | 1 |
| Kallio, T | 1 |
| Maia, G | 1 |
| Tammeveski, K | 1 |
| Lymperopoulos, N | 1 |
| Carcadea, E | 1 |
| Veziroglu, E | 1 |
| Iranzo, A | 1 |
| M Kannan, A | 1 |
| Arunamata, A | 1 |
| Tacy, TA | 1 |
| Kache, S | 1 |
| Mainwaring, RD | 1 |
| Ma, M | 1 |
| Maeda, K | 1 |
| Punn, R | 1 |
| Noguchi, S | 1 |
| Hahn, S | 3 |
| Iwasa, Y | 3 |
| Ling, J | 2 |
| Voccio, JP | 2 |
| Song, J | 3 |
| Bascuñán, J | 2 |
| Chu, Y | 1 |
| Tomita, M | 1 |
| Cazorla, M | 1 |
| Herrera, E | 1 |
| Palomeque, E | 1 |
| Saud, N | 1 |
| Hoplock, LB | 1 |
| Lobchuk, MM | 1 |
| Lemoine, J | 1 |
| Li, X | 10 |
| Henson, MA | 1 |
| Unsihuay, D | 1 |
| Qiu, J | 1 |
| Swaroop, S | 1 |
| Nagornov, KO | 1 |
| Kozhinov, AN | 1 |
| Tsybin, YO | 1 |
| Kuang, S | 1 |
| Laskin, J | 1 |
| Zin, NNINM | 1 |
| Mohamad, MN | 1 |
| Roslan, K | 1 |
| Abdul Wafi, S | 1 |
| Abdul Moin, NI | 1 |
| Alias, A | 1 |
| Zakaria, Y | 1 |
| Abu-Bakar, N | 1 |
| Naveed, A | 1 |
| Jilani, K | 1 |
| Siddique, AB | 1 |
| Akbar, M | 1 |
| Riaz, M | 1 |
| Mushtaq, Z | 1 |
| Sikandar, M | 1 |
| Ilyas, S | 1 |
| Bibi, I | 1 |
| Asghar, A | 1 |
| Rasool, G | 1 |
| Irfan, M | 1 |
| Li, XY | 1 |
| Zhao, S | 1 |
| Fan, XH | 1 |
| Chen, KP | 1 |
| Hua, W | 1 |
| Liu, ZM | 1 |
| Xue, XD | 1 |
| Zhou, B | 1 |
| Xing, YL | 1 |
| Chen, MA | 1 |
| Sun, Y | 2 |
| Neradilek, MB | 1 |
| Wu, XT | 1 |
| Zhang, D | 2 |
| Huang, W | 1 |
| Cui, Y | 1 |
| Yang, QQ | 1 |
| Li, HW | 1 |
| Zhao, XQ | 1 |
| Hossein Rashidi, B | 1 |
| Tarafdari, A | 1 |
| Ghazimirsaeed, ST | 1 |
| Shahrokh Tehraninezhad, E | 1 |
| Keikha, F | 1 |
| Eslami, B | 1 |
| Ghazimirsaeed, SM | 1 |
| Jafarabadi, M | 1 |
| Silvani, Y | 1 |
| Lovita, AND | 1 |
| Maharani, A | 1 |
| Wiyasa, IWA | 1 |
| Sujuti, H | 1 |
| Ratnawati, R | 1 |
| Raras, TYM | 1 |
| Lemin, AS | 1 |
| Rahman, MM | 1 |
| Pangarah, CA | 1 |
| Kiyu, A | 1 |
| Zeng, C | 2 |
| Du, H | 1 |
| Lin, D | 1 |
| Jalan, D | 1 |
| Rubagumya, F | 1 |
| Hopman, WM | 1 |
| Vanderpuye, V | 1 |
| Lopes, G | 1 |
| Seruga, B | 1 |
| Booth, CM | 1 |
| Berry, S | 1 |
| Hammad, N | 1 |
| Sajo, EA | 1 |
| Okunade, KS | 1 |
| Olorunfemi, G | 1 |
| Rabiu, KA | 1 |
| Anorlu, RI | 1 |
| Xu, C | 2 |
| Zhou, L | 2 |
| Dong, X | 1 |
| Tang, S | 1 |
| Gao, XC | 1 |
| Wei, CH | 1 |
| Zhang, RG | 1 |
| Cai, Q | 1 |
| He, Y | 1 |
| Tong, F | 1 |
| Dong, JH | 1 |
| Wu, G | 1 |
| Dong, XR | 1 |
| Tang, X | 1 |
| Tao, F | 1 |
| Xiang, W | 1 |
| Zhao, Y | 2 |
| Jin, L | 1 |
| Tao, H | 1 |
| Lei, Y | 1 |
| Gan, H | 1 |
| Chen, Y | 5 |
| Chen, L | 5 |
| Shan, A | 1 |
| Zhao, H | 2 |
| Wu, M | 2 |
| Ma, Q | 1 |
| Zhang, E | 1 |
| Zhang, J | 3 |
| Li, Y | 6 |
| Xue, F | 1 |
| Deng, L | 1 |
| Liu, L | 2 |
| Yan, Z | 2 |
| Meng, J | 1 |
| Chen, G | 3 |
| Anastassiadou, M | 1 |
| Bernasconi, G | 1 |
| Brancato, A | 1 |
| Carrasco Cabrera, L | 1 |
| Greco, L | 1 |
| Jarrah, S | 1 |
| Kazocina, A | 1 |
| Leuschner, R | 1 |
| Magrans, JO | 1 |
| Miron, I | 1 |
| Nave, S | 1 |
| Pedersen, R | 1 |
| Reich, H | 1 |
| Rojas, A | 1 |
| Sacchi, A | 1 |
| Santos, M | 1 |
| Theobald, A | 1 |
| Vagenende, B | 1 |
| Verani, A | 1 |
| Du, L | 1 |
| Liu, X | 1 |
| Ren, Y | 1 |
| Li, J | 7 |
| Li, P | 3 |
| Jiao, Q | 1 |
| Meng, P | 1 |
| Wang, F | 2 |
| Wang, YS | 1 |
| Wang, W | 1 |
| Wang, S | 2 |
| Hou, J | 1 |
| Zhang, A | 1 |
| Lv, B | 1 |
| Gao, C | 1 |
| Pang, D | 1 |
| Lu, K | 1 |
| Ahmad, NH | 1 |
| Zhu, J | 3 |
| Zhang, L | 2 |
| Zhuang, T | 1 |
| Tu, J | 1 |
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| Wang, X | 5 |
| Lee, DF | 1 |
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| Wu, W | 1 |
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| Xiao, Z | 1 |
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| Wei, Y | 2 |
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| Na, KJ | 1 |
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| Lee, SB | 1 |
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| Park, S | 1 |
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| Jeon, YK | 1 |
| Chung, DH | 1 |
| Paeng, JC | 1 |
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| Kang, CH | 1 |
| Cheon, GJ | 1 |
| Kang, KW | 1 |
| Lee, DS | 1 |
| Kim, YT | 1 |
| Pajuelo-Lozano, N | 1 |
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| Perona, R | 1 |
| Sanchez-Perez, I | 1 |
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| Richter, C | 1 |
| Edelhäuser, BAH | 1 |
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| Wolkenhauer, O | 1 |
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| Spitschak, A | 1 |
| Pützer, BM | 1 |
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| Zhang, M | 4 |
| Zhang, H | 1 |
| Yang, T | 2 |
| Dong, Y | 1 |
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| Mei, Z | 1 |
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| Liang, C | 1 |
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11 reviews available for metformin and Stomach Neoplasms
| Article | Year |
|---|---|
Aspirin, metformin, and statin use on the risk of gastric cancer: A nationwide population-based cohort study in Korea with systematic review and meta-analysis.
Topics: Aspirin; Cohort Studies; Diabetes Mellitus; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; | 2022 |
Association of Tumor Pathological Response with the Use of Metformin During Neoadjuvant Chemoradiotherapy in Rectal and Esophageal/Gastroesophageal Cancer Patients: a Systematic Review and Meta-analysis.
Topics: Adenocarcinoma; Chemoradiotherapy; Esophageal Neoplasms; Humans; Metformin; Neoadjuvant Therapy; Rec | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Mechanism Underlying Metformin Action and Its Potential to Reduce Gastric Cancer Risk.
Topics: AMP-Activated Protein Kinases; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; St | 2022 |
Diabetes Mellitus and Gastric Cancer: Correlation and Potential Mechanisms.
Topics: Diabetes Mellitus; Helicobacter Infections; Humans; Hyperglycemia; Metformin; Risk Factors; Stomach | 2023 |
The effect of metformin on gastric cancer in patients with type 2 diabetes: a systematic review and meta-analysis.
Topics: Asian People; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agents; Metformin; Prognosis; Stomach | 2020 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Immortal time bias exaggerates the effect of metformin on the risk of gastric cancer: A meta-analysis.
Topics: Cohort Studies; Humans; Hypoglycemic Agents; Metformin; Randomized Controlled Trials as Topic; Risk | 2021 |
Chemopreventive Effect of Metformin on Gastric Cancer Development.
Topics: Anticarcinogenic Agents; Diabetes Mellitus; Helicobacter Infections; Helicobacter pylori; Humans; Me | 2022 |
The Relationship between Diabetes Mellitus and Gastric Cancer and the Potential Benefits of Metformin: An Extensive Review of the Literature.
Topics: Diabetes Mellitus, Type 1; Diabetes Mellitus, Type 2; Female; Gastrectomy; Humans; Hyperglycemia; Hy | 2021 |
The therapeutic potential of metformin in gastric cancer.
Topics: Humans; Hypoglycemic Agents; Metformin; Prognosis; Signal Transduction; Stomach Neoplasms | 2019 |
Diabetes and gastric cancer: the potential links.
Topics: Asian People; Comorbidity; Diabetes Complications; Diabetes Mellitus; Feeding Behavior; Female; Heli | 2014 |
1 trial available for metformin and Stomach Neoplasms
| Article | Year |
|---|---|
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
42 other studies available for metformin and Stomach Neoplasms
| Article | Year |
|---|---|
Metformin and MiR-365 synergistically promote the apoptosis of gastric cancer cells via MiR-365-PTEN-AMPK axis.
Topics: 3' Untranslated Regions; AMP-Activated Protein Kinases; Animals; Antineoplastic Agents; Apoptosis; C | 2022 |
Disrupting Circadian Rhythm via the PER1-HK2 Axis Reverses Trastuzumab Resistance in Gastric Cancer.
Topics: Circadian Rhythm; Hexokinase; Humans; Metformin; Period Circadian Proteins; Stomach Neoplasms; Trast | 2022 |
Combination of metformin and oxaliplatin inhibits gastric cancer cell proliferation and induces apoptosis.
Topics: Apoptosis; bcl-2-Associated X Protein; Caspase 3; Cell Line, Tumor; Cell Proliferation; Humans; Metf | 2022 |
Prognostic value of long-term antidiabetic and antihypertensive therapy in postoperative gastric cancer patients: the FIESTA study.
Topics: Antihypertensive Agents; Calcium Channel Blockers; Diabetes Mellitus; Gastrectomy; Humans; Hypertens | 2022 |
Synergistic Effect of Metformin and Lansoprazole Against Gastric Cancer through Growth Inhibition.
Topics: Animals; Diabetes Mellitus, Type 2; Humans; Lansoprazole; Metformin; Proton Pump Inhibitors; Stomach | 2023 |
Metformin use and risk of gastric adenocarcinoma in a Swedish population-based cohort study.
Topics: Adenocarcinoma; Adult; Aged; Anti-Inflammatory Agents, Non-Steroidal; Aspirin; Diabetes Mellitus; Fe | 2019 |
The Effect of Metformin on Prognosis in Patients With Locally Advanced Gastric Cancer Associated With Type 2 Diabetes Mellitus.
Topics: Adult; Aged; Cause of Death; Cohort Studies; Comorbidity; Databases, Factual; Diabetes Mellitus, Typ | 2019 |
PTPRD-inactivation-induced CXCL8 promotes angiogenesis and metastasis in gastric cancer and is inhibited by metformin.
Topics: Cell Line, Tumor; Down-Regulation; Gene Silencing; Humans; Hypoglycemic Agents; Interleukin-8; Metfo | 2019 |
Metformin use reduced the risk of stomach cancer in diabetic patients in Korea: an analysis of Korean NHIS-HEALS database.
Topics: Adult; Databases, Factual; Diabetes Complications; Diabetes Mellitus; Early Detection of Cancer; Fem | 2020 |
Association of Aspirin, Metformin, and Statin Use with Gastric Cancer Incidence and Mortality: A Nationwide Cohort Study.
Topics: Adult; Aged; Aspirin; Female; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Incidence; Mal | 2021 |
Efficacy of Metformin and Chemotherapeutic Agents on the Inhibition of Colony Formation and Shh/Gli1 Pathway: Metformin/Docetaxel Versus Metformin/5-Fluorouracil.
Topics: Antineoplastic Combined Chemotherapy Protocols; Cell Line, Tumor; Docetaxel; Drug Synergism; Fluorou | 2021 |
AMPK inhibits Smad3-mediated autoinduction of TGF-β1 in gastric cancer cells.
Topics: AMP-Activated Protein Kinases; Cells, Cultured; Female; Gene Expression Regulation, Neoplastic; Huma | 2021 |
Metformin attenuates synergic effect of diabetes mellitus and Helicobacter pylori infection on gastric cancer cells proliferation by suppressing PTEN expression.
Topics: Apoptosis; Cell Proliferation; Diabetes Mellitus; DNA Methylation; Gene Expression Regulation, Neopl | 2021 |
Improved prognosis in gastric adenocarcinoma among metformin users in a population-based study.
Topics: Adenocarcinoma; Aged; Aged, 80 and over; Cause of Death; Cohort Studies; Female; Humans; Male; Metfo | 2021 |
CCDC65 as a new potential tumor suppressor induced by metformin inhibits activation of AKT1 via ubiquitination of ENO1 in gastric cancer.
Topics: Animals; Biomarkers, Tumor; Cell Line, Tumor; Cell Movement; Cell Proliferation; China; DNA-Binding | 2021 |
Metformin inhibits gastric cancer cells metastatic traits through suppression of epithelial-mesenchymal transition in a glucose-independent manner.
Topics: beta Catenin; Cadherins; Cell Line, Tumor; Cell Movement; Epithelial-Mesenchymal Transition; Gene Ex | 2017 |
Metformin Inhibits N-Methyl-N-Nitrosourea Induced Gastric Tumorigenesis in db/db Mice.
Topics: Animals; Insulin; Insulin-Like Growth Factor I; Insulin-Like Growth Factor II; Male; Metformin; Meth | 2017 |
Metformin targets gastric cancer stem cells.
Topics: Animals; Antineoplastic Agents; Biomarkers, Tumor; Cell Cycle Checkpoints; Cell Differentiation; Cel | 2017 |
Effect of metformin combined with chemotherapeutic agents on gastric cancer cell line AGS.
Topics: Apoptosis; Cell Line, Tumor; Cell Migration Assays; Cell Proliferation; Cisplatin; Dose-Response Rel | 2017 |
Impact of metformin on gastric adenocarcinoma survival: A Belgian population based study.
Topics: Adenocarcinoma; Aged; Belgium; Cohort Studies; Diabetes Mellitus; Female; Humans; Hypoglycemic Agent | 2018 |
Long noncoding RNA H19 participates in metformin-mediated inhibition of gastric cancer cell invasion.
Topics: AMP-Activated Protein Kinases; Animals; Antineoplastic Agents; Cell Line, Tumor; Cell Movement; Cell | 2019 |
Metformin suppresses gastric cancer progression through calmodulin‑like protein 3 secreted from tumor‑associated fibroblasts.
Topics: Calmodulin; Cancer-Associated Fibroblasts; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cocu | 2019 |
Metformin Use and Gastric Cancer Risk in Diabetic Patients After Helicobacter pylori Eradication.
Topics: Aged; Anti-Bacterial Agents; Blood Glucose; Clarithromycin; Cohort Studies; Diabetes Mellitus; Drug | 2019 |
The Antitumor Effects of Metformin on Gastric Cancer
Topics: Adenylate Kinase; AMP-Activated Protein Kinase Kinases; Animals; Antineoplastic Agents; Cell Line, T | 2018 |
Anticancer effect of metformin: what about gastric cancer?
Topics: AMP-Activated Protein Kinases; Antineoplastic Agents; Cell Line, Tumor; Cell Proliferation; Diet; Fe | 2018 |
Metformin triggers the intrinsic apoptotic response in human AGS gastric adenocarcinoma cells by activating AMPK and suppressing mTOR/AKT signaling.
Topics: AMP-Activated Protein Kinases; Apoptosis; Cell Line, Tumor; Cell Proliferation; Cell Survival; Gene | 2019 |
Response to Khosrow-Khavar, Kurteva, and Douros.
Topics: Diabetes Mellitus; Helicobacter pylori; Humans; Metformin; Risk; Stomach Neoplasms | 2019 |
RE: Metformin Use and Gastric Cancer Risk in Diabetic Patients After Helicobacter pylori Eradication.
Topics: Diabetes Mellitus; Helicobacter Infections; Helicobacter pylori; Humans; Metformin; Risk Factors; St | 2019 |
Long-term metformin use reduces gastric cancer risk in type 2 diabetics without insulin treatment: a nationwide cohort study.
Topics: Adult; Aged; Aged, 80 and over; Cohort Studies; Databases, Factual; Diabetes Mellitus, Type 2; Femal | 2014 |
Commentary: metformin use is associated with reduced gastric cancer risk.
Topics: Diabetes Mellitus, Type 2; Female; Humans; Hypoglycemic Agents; Male; Metformin; Stomach Neoplasms | 2014 |
Commentary: metformin use is associated with reduced gastric cancer risk - authors' reply.
Topics: Diabetes Mellitus, Type 2; Female; Humans; Hypoglycemic Agents; Male; Metformin; Stomach Neoplasms | 2014 |
Does long-term metformin usage reduce gastric cancer risk?
Topics: Diabetes Mellitus, Type 2; Female; Humans; Hypoglycemic Agents; Male; Metformin; Stomach Neoplasms | 2014 |
Increased expression of phospho-acetyl-CoA carboxylase protein is an independent prognostic factor for human gastric cancer without lymph node metastasis.
Topics: Acetyl-CoA Carboxylase; Aged; Biomarkers, Tumor; Cell Proliferation; Female; Humans; Kaplan-Meier Es | 2014 |
AMPK/mTOR-mediated inhibition of survivin partly contributes to metformin-induced apoptosis in human gastric cancer cell.
Topics: AMP-Activated Protein Kinases; Animals; Antineoplastic Agents; Apoptosis; Cell Line, Tumor; Disease | 2015 |
Cumulative Metformin Use and Its Impact on Survival in Gastric Cancer Patients After Gastrectomy.
Topics: Adult; Aged; Aged, 80 and over; Cohort Studies; Diabetes Complications; Female; Gastrectomy; Humans; | 2016 |
Gastric cancer. Metformin improves survival and recurrence rate in patients with diabetes and gastric cancer.
Topics: Antineoplastic Agents; Chemotherapy, Adjuvant; Diabetes Mellitus, Type 2; Humans; Hypoglycemic Agent | 2015 |
Metformin potentiates rapamycin and cisplatin in gastric cancer in mice.
Topics: Adult; Aged; Animals; Antineoplastic Combined Chemotherapy Protocols; Blotting, Western; Cell Cycle; | 2015 |
Clinicopathological significance of N-cadherin and VEGF in advanced gastric cancer brain metastasis and the effects of metformin in preclinical models.
Topics: Adult; Aged; Aged, 80 and over; Animals; Antigens, CD; Brain Neoplasms; Cadherins; Case-Control Stud | 2015 |
Metformin reduces gastric cancer risk in patients with type 2 diabetes mellitus.
Topics: Aged; Databases, Factual; Diabetes Mellitus, Type 2; Female; Humans; Hypoglycemic Agents; Incidence; | 2016 |
Metformin suppresses the expression of Sonic hedgehog in gastric cancer cells.
Topics: AMP-Activated Protein Kinases; Antineoplastic Agents; Cell Line, Tumor; Down-Regulation; Gastric Muc | 2017 |
The antidiabetic drug metformin inhibits gastric cancer cell proliferation in vitro and in vivo.
Topics: Animals; Blotting, Western; Cell Cycle; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; C | 2012 |
A case of megaloblastic anemia due to vitamin B12 deficiency precipitated in a totally gastrectomized type II diabetic patient following the introduction of metformin therapy.
Topics: Aged; Anemia, Megaloblastic; Diabetes Mellitus, Type 2; Female; Gastrectomy; Humans; Hypoglycemic Ag | 2003 |