metformin has been researched along with Infarction, Middle Cerebral Artery in 17 studies
Metformin: A biguanide hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. Metformin improves glycemic control by improving insulin sensitivity and decreasing intestinal absorption of glucose. (From Martindale, The Extra Pharmacopoeia, 30th ed, p289)
metformin : A member of the class of guanidines that is biguanide the carrying two methyl substituents at position 1.
Infarction, Middle Cerebral Artery: NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.
| Excerpt | Relevance | Reference |
|---|---|---|
| "This study is to analyze the neuroprotective effects of long-term metformin (Met) preconditioning on rats with ischemic brain injuries and the related mechanisms." | 8.02 | Neuroprotective Effects of Long-Term Metformin Preconditioning on Rats with Ischemic Brain Injuries. ( Guo, H; Liu, Y; Liu, Z; Pei, T; Ruan, C; Wang, A; Wang, L; Wang, S; Yang, D; Zhang, Z, 2021) |
| "Long-term metformin treatment reduces the risk of stroke." | 7.83 | Pre-stroke Metformin Treatment is Neuroprotective Involving AMPK Reduction. ( Chen, Y; Chen, Z; Deng, T; Hou, WW; Hu, WW; Shen, Z; Wu, XL; Yuan, Y; Zhang, LS; Zhang, XN; Zheng, YR, 2016) |
| "Accumulating evidence suggests that chronic metformin preconditioning offers potent neuroprotective effects against ischemic stroke." | 7.81 | Chronic Metformin Preconditioning Provides Neuroprotection via Suppression of NF-κB-Mediated Inflammatory Pathway in Rats with Permanent Cerebral Ischemia. ( Cao, L; Ding, ZZ; Jiang, T; Tan, L; Tan, MS; Wang, HF; Yu, JT; Zhang, QQ; Zhu, XC, 2015) |
| " Paradoxically, the clinical use of an AMPK activator metformin reduces the incidence of stroke." | 7.80 | Improvement of functional recovery by chronic metformin treatment is associated with enhanced alternative activation of microglia/macrophages and increased angiogenesis and neurogenesis following experimental stroke. ( Cheng, J; Jia, J; Jin, Q; Liu, Y; Qin, Z; Wang, X; Wei, S; Wu, J; Zhen, X; Zhou, X, 2014) |
| "Acute metformin exacerbated stroke damage, enhanced AMPK activation, and led to metabolic dysfunction." | 7.76 | Effects of metformin in experimental stroke. ( Benashski, SE; Li, J; McCullough, LD; Venna, VR, 2010) |
| "Hyperglycemia is a known exacerbating factor in ischemic stroke." | 6.47 | [Effectiveness of metformin in prevention of development of hyperglycemia and neuronal damage caused by ischemic stress]. ( Fujita-Hamabe, W; Harada, S; Tokuyama, S, 2011) |
| "Metformin (MET) has been the subject of many classic studies in possessing antiapoptotic, anti-inflammatory, antioxidation activities and antiviral." | 5.72 | Metformin protects against pericyte apoptosis and promotes neurogenesis through suppressing JNK p38 MAPK signalling activation in ischemia/reperfusion injury. ( Cui, K; Fu, X; Liu, C; Lu, Z; Man, J; Wang, J; Zhang, D; Zhang, Z, 2022) |
| "Metformin (Met) is a commonly used drug in the treatment of type 2 diabetes." | 5.62 | Neuroprotective effects of metformin on cerebral ischemia-reperfusion injury by regulating PI3K/Akt pathway. ( Gao, J; Guo, H; Li, X; Liu, Z; Lv, H; Ruan, C; Wang, Y; Yan, J, 2021) |
| "Metformin is a well-known activator of AMP-activated protein kinase (AMPK)." | 5.40 | Chronic metformin treatment improves post-stroke angiogenesis and recovery after experimental stroke. ( Hammond, MD; Li, J; Mancini, NS; McCullough, LD; Venna, VR, 2014) |
| "This study is to analyze the neuroprotective effects of long-term metformin (Met) preconditioning on rats with ischemic brain injuries and the related mechanisms." | 4.02 | Neuroprotective Effects of Long-Term Metformin Preconditioning on Rats with Ischemic Brain Injuries. ( Guo, H; Liu, Y; Liu, Z; Pei, T; Ruan, C; Wang, A; Wang, L; Wang, S; Yang, D; Zhang, Z, 2021) |
| "Long-term metformin treatment reduces the risk of stroke." | 3.83 | Pre-stroke Metformin Treatment is Neuroprotective Involving AMPK Reduction. ( Chen, Y; Chen, Z; Deng, T; Hou, WW; Hu, WW; Shen, Z; Wu, XL; Yuan, Y; Zhang, LS; Zhang, XN; Zheng, YR, 2016) |
| "Accumulating evidence suggests that chronic metformin preconditioning offers potent neuroprotective effects against ischemic stroke." | 3.81 | Chronic Metformin Preconditioning Provides Neuroprotection via Suppression of NF-κB-Mediated Inflammatory Pathway in Rats with Permanent Cerebral Ischemia. ( Cao, L; Ding, ZZ; Jiang, T; Tan, L; Tan, MS; Wang, HF; Yu, JT; Zhang, QQ; Zhu, XC, 2015) |
| " Paradoxically, the clinical use of an AMPK activator metformin reduces the incidence of stroke." | 3.80 | Improvement of functional recovery by chronic metformin treatment is associated with enhanced alternative activation of microglia/macrophages and increased angiogenesis and neurogenesis following experimental stroke. ( Cheng, J; Jia, J; Jin, Q; Liu, Y; Qin, Z; Wang, X; Wei, S; Wu, J; Zhen, X; Zhou, X, 2014) |
| "Acute metformin exacerbated stroke damage, enhanced AMPK activation, and led to metabolic dysfunction." | 3.76 | Effects of metformin in experimental stroke. ( Benashski, SE; Li, J; McCullough, LD; Venna, VR, 2010) |
| "Hyperglycemia is a known exacerbating factor in ischemic stroke." | 2.47 | [Effectiveness of metformin in prevention of development of hyperglycemia and neuronal damage caused by ischemic stress]. ( Fujita-Hamabe, W; Harada, S; Tokuyama, S, 2011) |
| "Then, stroke was induced by transient middle cerebral artery occlusion (tMCAO)." | 1.72 | Effects of co-administration of metformin and evogliptin on cerebral infarct volume in the diabetic rat. ( Hong, JM; Joe, EH; Lee, JS; Lee, SJ; Yoon, BS, 2022) |
| "Metformin (MET) has been the subject of many classic studies in possessing antiapoptotic, anti-inflammatory, antioxidation activities and antiviral." | 1.72 | Metformin protects against pericyte apoptosis and promotes neurogenesis through suppressing JNK p38 MAPK signalling activation in ischemia/reperfusion injury. ( Cui, K; Fu, X; Liu, C; Lu, Z; Man, J; Wang, J; Zhang, D; Zhang, Z, 2022) |
| "Metformin (Met) is a commonly used drug in the treatment of type 2 diabetes." | 1.62 | Neuroprotective effects of metformin on cerebral ischemia-reperfusion injury by regulating PI3K/Akt pathway. ( Gao, J; Guo, H; Li, X; Liu, Z; Lv, H; Ruan, C; Wang, Y; Yan, J, 2021) |
| "Metformin is a well-known activator of AMP-activated protein kinase (AMPK)." | 1.40 | Chronic metformin treatment improves post-stroke angiogenesis and recovery after experimental stroke. ( Hammond, MD; Li, J; Mancini, NS; McCullough, LD; Venna, VR, 2014) |
| "Metformin (200 mg/kg) was administrated for up to 14 days." | 1.40 | Metformin attenuates blood-brain barrier disruption in mice following middle cerebral artery occlusion. ( Chen, X; Gu, X; Li, Y; Liu, Y; Tang, G; Wang, Y; Yang, GY; Zhang, Z, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 10 (58.82) | 24.3611 |
| 2020's | 7 (41.18) | 2.80 |
| Authors | Studies |
|---|---|
| Ruan, C | 2 |
| Guo, H | 2 |
| Gao, J | 1 |
| Wang, Y | 6 |
| Liu, Z | 4 |
| Yan, J | 1 |
| Li, X | 11 |
| Lv, H | 1 |
| Lee, SJ | 1 |
| Yoon, BS | 1 |
| Hong, JM | 1 |
| Joe, EH | 1 |
| Lee, JS | 1 |
| Liu, C | 4 |
| Zhang, D | 3 |
| Lu, Z | 1 |
| Man, J | 1 |
| Zhang, Z | 3 |
| Fu, X | 1 |
| Cui, K | 1 |
| Wang, J | 5 |
| Zhang, L | 3 |
| Zhang, J | 4 |
| Zhu, X | 1 |
| Jiao, W | 1 |
| Yang, Y | 2 |
| Wu, Y | 2 |
| Yang, L | 1 |
| Navia, V | 1 |
| Riveros, R | 1 |
| Brunser, A | 1 |
| Nguépy Keubo, FR | 1 |
| Mboua, PC | 1 |
| Djifack Tadongfack, T | 1 |
| Fokouong Tchoffo, E | 1 |
| Tasson Tatang, C | 1 |
| Ide Zeuna, J | 1 |
| Noupoue, EM | 1 |
| Tsoplifack, CB | 1 |
| Folefack, GO | 1 |
| Kettani, M | 1 |
| Bandelier, P | 1 |
| Huo, J | 1 |
| Li, H | 4 |
| Yu, D | 1 |
| Arulsamy, N | 1 |
| AlAbbad, S | 1 |
| Sardot, T | 1 |
| Lekashvili, O | 1 |
| Decato, D | 1 |
| Lelj, F | 1 |
| Alexander Ross, JB | 1 |
| Rosenberg, E | 1 |
| Nazir, H | 1 |
| Muthuswamy, N | 1 |
| Louis, C | 1 |
| Jose, S | 1 |
| Prakash, J | 1 |
| Buan, MEM | 1 |
| Flox, C | 1 |
| Chavan, S | 1 |
| Shi, X | 1 |
| Kauranen, P | 1 |
| Kallio, T | 1 |
| Maia, G | 1 |
| Tammeveski, K | 1 |
| Lymperopoulos, N | 1 |
| Carcadea, E | 1 |
| Veziroglu, E | 1 |
| Iranzo, A | 1 |
| M Kannan, A | 1 |
| Arunamata, A | 1 |
| Tacy, TA | 1 |
| Kache, S | 1 |
| Mainwaring, RD | 1 |
| Ma, M | 1 |
| Maeda, K | 1 |
| Punn, R | 1 |
| Noguchi, S | 1 |
| Hahn, S | 3 |
| Iwasa, Y | 3 |
| Ling, J | 2 |
| Voccio, JP | 2 |
| Kim, Y | 3 |
| Song, J | 3 |
| Bascuñán, J | 2 |
| Chu, Y | 1 |
| Tomita, M | 1 |
| Cazorla, M | 1 |
| Herrera, E | 1 |
| Palomeque, E | 1 |
| Saud, N | 1 |
| Hoplock, LB | 1 |
| Lobchuk, MM | 1 |
| Lemoine, J | 1 |
| Henson, MA | 1 |
| Unsihuay, D | 1 |
| Qiu, J | 1 |
| Swaroop, S | 1 |
| Nagornov, KO | 1 |
| Kozhinov, AN | 1 |
| Tsybin, YO | 1 |
| Kuang, S | 1 |
| Laskin, J | 1 |
| Zin, NNINM | 1 |
| Mohamad, MN | 1 |
| Roslan, K | 1 |
| Abdul Wafi, S | 1 |
| Abdul Moin, NI | 1 |
| Alias, A | 1 |
| Zakaria, Y | 1 |
| Abu-Bakar, N | 1 |
| Naveed, A | 1 |
| Jilani, K | 1 |
| Siddique, AB | 1 |
| Akbar, M | 1 |
| Riaz, M | 1 |
| Mushtaq, Z | 1 |
| Sikandar, M | 1 |
| Ilyas, S | 1 |
| Bibi, I | 1 |
| Asghar, A | 1 |
| Rasool, G | 1 |
| Irfan, M | 1 |
| Li, XY | 1 |
| Zhao, S | 1 |
| Fan, XH | 1 |
| Chen, KP | 1 |
| Hua, W | 1 |
| Liu, ZM | 1 |
| Xue, XD | 1 |
| Zhou, B | 1 |
| Zhang, S | 2 |
| Xing, YL | 1 |
| Chen, MA | 1 |
| Sun, Y | 1 |
| Neradilek, MB | 1 |
| Wu, XT | 1 |
| Huang, W | 1 |
| Cui, Y | 1 |
| Yang, QQ | 1 |
| Li, HW | 1 |
| Zhao, XQ | 1 |
| Hossein Rashidi, B | 1 |
| Tarafdari, A | 1 |
| Ghazimirsaeed, ST | 1 |
| Shahrokh Tehraninezhad, E | 1 |
| Keikha, F | 1 |
| Eslami, B | 1 |
| Ghazimirsaeed, SM | 1 |
| Jafarabadi, M | 1 |
| Silvani, Y | 1 |
| Lovita, AND | 1 |
| Maharani, A | 1 |
| Wiyasa, IWA | 1 |
| Sujuti, H | 1 |
| Ratnawati, R | 1 |
| Raras, TYM | 1 |
| Lemin, AS | 1 |
| Rahman, MM | 1 |
| Pangarah, CA | 1 |
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| Zeng, C | 2 |
| Du, H | 1 |
| Lin, D | 1 |
| Jalan, D | 1 |
| Rubagumya, F | 1 |
| Hopman, WM | 1 |
| Vanderpuye, V | 1 |
| Lopes, G | 1 |
| Seruga, B | 1 |
| Booth, CM | 1 |
| Berry, S | 1 |
| Hammad, N | 1 |
| Sajo, EA | 1 |
| Okunade, KS | 1 |
| Olorunfemi, G | 1 |
| Rabiu, KA | 1 |
| Anorlu, RI | 1 |
| Xu, C | 2 |
| Xiang, Y | 1 |
| Xu, X | 1 |
| Zhou, L | 2 |
| Dong, X | 1 |
| Tang, S | 1 |
| Gao, XC | 1 |
| Wei, CH | 1 |
| Zhang, RG | 1 |
| Cai, Q | 1 |
| He, Y | 1 |
| Tong, F | 1 |
| Dong, JH | 1 |
| Wu, G | 1 |
| Dong, XR | 1 |
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| Zhao, Y | 2 |
| Jin, L | 1 |
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| Huang, Y | 1 |
| Chen, Y | 4 |
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| Shan, A | 1 |
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| Ma, Q | 1 |
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| Xue, F | 1 |
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| Leuschner, R | 1 |
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| Miron, I | 1 |
| Nave, S | 1 |
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| Reich, H | 1 |
| Rojas, A | 1 |
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| Vagenende, B | 1 |
| Verani, A | 1 |
| Du, L | 1 |
| Liu, X | 1 |
| Ren, Y | 1 |
| Li, J | 9 |
| Li, P | 1 |
| Jiao, Q | 1 |
| Meng, P | 1 |
| Wang, F | 2 |
| Wang, YS | 1 |
| Wang, C | 3 |
| Zhou, X | 3 |
| Wang, W | 1 |
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| Hou, J | 1 |
| Zhang, A | 1 |
| Lv, B | 1 |
| Gao, C | 1 |
| Pang, D | 1 |
| Lu, K | 1 |
| Ahmad, NH | 1 |
| Wang, L | 2 |
| Zhu, J | 2 |
| Zhuang, T | 1 |
| Tu, J | 1 |
| Zhao, Z | 1 |
| Qu, Y | 1 |
| Yao, H | 1 |
| Wang, X | 6 |
| Lee, DF | 1 |
| Shen, J | 3 |
| Wen, L | 1 |
| Huang, G | 2 |
| Xie, X | 1 |
| Zhao, Q | 1 |
| Hu, W | 1 |
| Zhang, Y | 4 |
| Wu, X | 1 |
| Lu, J | 2 |
| Li, M | 1 |
| Li, W | 2 |
| Wu, W | 1 |
| Du, F | 1 |
| Ji, H | 1 |
| Yang, X | 2 |
| Xu, Z | 1 |
| Wan, L | 1 |
| Wen, Q | 1 |
| Cho, CH | 1 |
| Zou, C | 1 |
| Xiao, Z | 1 |
| Liao, J | 1 |
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| Huang, H | 1 |
| Wei, Y | 2 |
| Gao, Y | 2 |
| Na, KJ | 1 |
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| Kim, YH | 1 |
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| Jung, YJ | 1 |
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| Lee, HJ | 1 |
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| Chung, DH | 1 |
| Paeng, JC | 1 |
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| Cheon, GJ | 1 |
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| Kim, YT | 1 |
| Pajuelo-Lozano, N | 1 |
| Alcalá, S | 1 |
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| Liu, Y | 7 |
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| Zhu, ZQ | 1 |
| Tang, HX | 1 |
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| Celestrin, CP | 1 |
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| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Effect of Dipeptidyl-4 Inhibitors in Reducing Stroke Severity, From the Health Insurance Review and Assessment Service Database[NCT05817097] | 22,119 participants (Anticipated) | Observational | 2023-08-31 | Not yet recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
2 reviews available for metformin and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
[Effectiveness of metformin in prevention of development of hyperglycemia and neuronal damage caused by ischemic stress].
Topics: Administration, Oral; AMP-Activated Protein Kinases; Animals; Brain; Brain Ischemia; Disease Models, | 2011 |
1 trial available for metformin and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
15 other studies available for metformin and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Neuroprotective effects of metformin on cerebral ischemia-reperfusion injury by regulating PI3K/Akt pathway.
Topics: Animals; Apoptosis; Brain Ischemia; Diabetes Mellitus, Type 2; Infarction, Middle Cerebral Artery; M | 2021 |
Effects of co-administration of metformin and evogliptin on cerebral infarct volume in the diabetic rat.
Topics: Animals; Brain Chemistry; Cerebral Infarction; Cerebrovascular Circulation; Diabetes Mellitus, Exper | 2022 |
Metformin protects against pericyte apoptosis and promotes neurogenesis through suppressing JNK p38 MAPK signalling activation in ischemia/reperfusion injury.
Topics: Animals; Apoptosis; Brain Ischemia; Caspase 3; Infarction, Middle Cerebral Artery; MAP Kinase Signal | 2022 |
Metformin enhances neural precursor cells migration and functional recovery after ischemic stroke in mice.
Topics: Animals; Bromodeoxyuridine; Cell Differentiation; Infarction, Middle Cerebral Artery; Ischemic Strok | 2023 |
[Intravenous thrombolysis after reversion of acenocoumarol anticoagulation. Report of one case].
Topics: Acenocoumarol; Administration, Intravenous; Aged, 80 and over; Amlodipine; Humans; Infarction, Middl | 2019 |
Metformin prevents stroke damage in non-diabetic female mice with chronic kidney disease.
Topics: Adenylate Kinase; Animals; Apoptosis; Body Weight; Brain Infarction; Enzyme Activation; Female; Gene | 2021 |
Neuroprotective Effects of Long-Term Metformin Preconditioning on Rats with Ischemic Brain Injuries.
Topics: Animals; Apoptosis; Brain Injuries; Brain Ischemia; Humans; Infarction, Middle Cerebral Artery; Metf | 2021 |
Offsetting the impact of smoking and e-cigarette vaping on the cerebrovascular system and stroke injury: Is Metformin a viable countermeasure?
Topics: Animals; Antioxidants; Blood-Brain Barrier; Cells, Cultured; Hypoglycemic Agents; Infarction, Middle | 2017 |
Improvement of functional recovery by chronic metformin treatment is associated with enhanced alternative activation of microglia/macrophages and increased angiogenesis and neurogenesis following experimental stroke.
Topics: AMP-Activated Protein Kinases; Angiogenesis Inducing Agents; Animals; Brain; Brain Ischemia; Infarct | 2014 |
Chronic metformin treatment improves post-stroke angiogenesis and recovery after experimental stroke.
Topics: AMP-Activated Protein Kinases; Animals; Apomorphine; Brain; Disease Models, Animal; Dopamine Agonist | 2014 |
Chronic Metformin Preconditioning Provides Neuroprotection via Suppression of NF-κB-Mediated Inflammatory Pathway in Rats with Permanent Cerebral Ischemia.
Topics: Acute Disease; Animals; Astrocytes; Brain; Brain Ischemia; Calcium-Binding Proteins; Cytokines; Glia | 2015 |
Metformin attenuates blood-brain barrier disruption in mice following middle cerebral artery occlusion.
Topics: AMP-Activated Protein Kinases; Animals; Blood-Brain Barrier; Brain Infarction; Cells, Cultured; Cyto | 2014 |
Pre-stroke Metformin Treatment is Neuroprotective Involving AMPK Reduction.
Topics: AMP-Activated Protein Kinases; Animals; Apoptosis; Disease Models, Animal; Infarction, Middle Cerebr | 2016 |
RETRACTED: The importance of regulation of blood glucose levels through activation of peripheral 5'-AMP-activated protein kinase on ischemic neuronal damage.
Topics: AMP-Activated Protein Kinases; Animals; Blood Glucose; Brain Ischemia; Enzyme Activation; Infarction | 2010 |
Effects of metformin in experimental stroke.
Topics: AMP-Activated Protein Kinases; Animals; Dose-Response Relationship, Drug; Hypoglycemic Agents; Incid | 2010 |