metformin has been researched along with Absence Status in 2 studies
Metformin: A biguanide hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. Metformin improves glycemic control by improving insulin sensitivity and decreasing intestinal absorption of glucose. (From Martindale, The Extra Pharmacopoeia, 30th ed, p289)
metformin : A member of the class of guanidines that is biguanide the carrying two methyl substituents at position 1.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Metformin has exhibited anti-inflammatory and neuroprotective properties in numerous studies." | 5.62 | Metformin ameliorates the status epilepticus- induced hippocampal pathology through possible mTOR modulation. ( Anand, S; Bhatia, A; Bojja, SL; Joshi, R; Medhi, B; Minz, RW, 2021) |
| "Metformin has exhibited anti-inflammatory and neuroprotective properties in numerous studies." | 1.62 | Metformin ameliorates the status epilepticus- induced hippocampal pathology through possible mTOR modulation. ( Anand, S; Bhatia, A; Bojja, SL; Joshi, R; Medhi, B; Minz, RW, 2021) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bojja, SL | 1 |
| Medhi, B | 1 |
| Anand, S | 1 |
| Bhatia, A | 1 |
| Joshi, R | 1 |
| Minz, RW | 1 |
| Chen, J | 1 |
| Zheng, G | 1 |
| Guo, H | 1 |
| Shi, ZN | 1 |
| Jiang, J | 1 |
| Wang, XY | 1 |
| Yang, X | 1 |
| Liu, XY | 1 |
2 other studies available for metformin and Absence Status
| Article | Year |
|---|---|
Metformin ameliorates the status epilepticus- induced hippocampal pathology through possible mTOR modulation.
Topics: Animals; Anti-Inflammatory Agents; Cytokines; Disease Models, Animal; Dose-Response Relationship, Dr | 2021 |
The effect of metformin treatment on endoplasmic reticulum (ER) stress induced by status epilepticus (SE) via the PERK-eIF2α-CHOP pathway.
Topics: Animals; Apoptosis; Convulsants; eIF-2 Kinase; Endoplasmic Reticulum Stress; Eukaryotic Initiation F | 2018 |