melatonin has been researched along with Hemorrhage, Subarachnoid in 24 studies
Excerpt | Relevance | Reference |
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"To determine whether melatonin (MLT) mitigates white matter (WM) injury by attenuating NOD-like receptor family pyrin domain-containing 3 (NLRP3)-associated oligodendrocyte apoptosis after subarachnoid hemorrhage (SAH)." | 7.96 | Melatonin Attenuates White Matter Injury via Reducing Oligodendrocyte Apoptosis After Subarachnoid Hemorrhage in Mice. ( Dong, Y; Feng, H; Hao, G; Li, G; Liang, G; Liu, D; Pan, P; Zou, Z, 2020) |
"The objective of this study was to identify the protective effect of melatonin (MT) against early brain injury (EBI) following subarachnoid hemorrhage (SAH) and explore the underlying molecular mechanism." | 7.88 | Long non-coding RNA and microRNA-675/let-7a mediates the protective effect of melatonin against early brain injury after subarachnoid hemorrhage via targeting TP53 and neural growth factor. ( He, Y; Li, S; Sun, B; Tang, W; Wen, L; Yang, S, 2018) |
"BACKGROUND A mouse model of subarachnoid hemorrhage (SAH) investigated the effects of melatonin treatment on the generation of reactive oxygen species (ROS) and the activation of the SIRT3 gene in early brain injury (EBI)." | 7.88 | Melatonin Treatment Regulates SIRT3 Expression in Early Brain Injury (EBI) Due to Reactive Oxygen Species (ROS) in a Mouse Model of Subarachnoid Hemorrhage (SAH). ( Chen, X; Hang, C; Li, S; Sun, B; Yang, S, 2018) |
"BACKGROUND The aim of this study was to investigate whether melatonin is involved in brain injury following subarachnoid hemorrhage (SAH)." | 7.88 | Melatonin Upregulates Nuclear Factor Erythroid-2 Related Factor 2 (Nrf2) and Mediates Mitophagy to Protect Against Early Brain Injury After Subarachnoid Hemorrhage. ( Hang, C; Li, S; Sun, B; Yang, S, 2018) |
"Melatonin (Mel) has been reported to alleviate early brain injury (EBI) following subarachnoid hemorrhage (SAH)." | 7.85 | Melatonin Attenuates Early Brain Injury via the Melatonin Receptor/Sirt1/NF-κB Signaling Pathway Following Subarachnoid Hemorrhage in Mice. ( Li, X; Lin, Y; Liu, H; Qu, Y; Wang, B; Yue, L; Zhang, J; Zhao, L, 2017) |
" The objective of this prospective study was to determine, in a cohort of patients with treated ruptured intracranial aneurysm (IA), the proportion of asthenia at 2months, in a cohort of patients treated with melatonin and in a control cohort." | 7.83 | Effects of melatonin in the treatment of asthenia in aneurysmal subarachnoid hemorrhage. ( Curey, S; Derrey, S; Ferracci, FX; Gilard, V; Langlois, O; Proust, F, 2016) |
"Melatonin is a strong antioxidant that has beneficial effects against early brain injury (EBI) following a subarachnoid hemorrhage (SAH) in rats; protection includes reduced mortality and brain water content." | 7.80 | Melatonin-enhanced autophagy protects against neural apoptosis via a mitochondrial pathway in early brain injury following a subarachnoid hemorrhage. ( Chen, G; Chen, J; Gu, C; Hu, Q; Li, J; Wang, L; Wu, C; Yan, F; Yan, W, 2014) |
"Melatonin is a strong anti-oxidant that has beneficial effects against early brain injury (EBI) following a subarachnoid hemorrhage (SAH) in rats; protection includes the reduction of both mortality and neurological deficits." | 7.80 | Melatonin attenuates inflammatory response-induced brain edema in early brain injury following a subarachnoid hemorrhage: a possible role for the regulation of pro-inflammatory cytokines. ( Chen, G; Chen, J; Gu, C; Li, J; Mo, H; Qian, C; Wang, L; Yan, F; Yan, W, 2014) |
"Previous studies proved that melatonin protected against secondary brain damage by modulating oxidative stress after experimental subarachnoid hemorrhage (SAH), but it has not been evaluated yet about its effects on inflammatory pathway and secondary cognitive dysfunction in SAH model." | 7.79 | Melatonin alleviates secondary brain damage and neurobehavioral dysfunction after experimental subarachnoid hemorrhage: possible involvement of TLR4-mediated inflammatory pathway. ( Chen, G; Ji, C; Wang, Z; Wu, L; You, W, 2013) |
"Melatonin has beneficial effects against early brain injury (EBI) by modulating cerebral oxidative stress after experimental subarachnoid hemorrhage (SAH); however, few investigations relate to the precise underlying molecular mechanisms." | 7.78 | Melatonin activates the Nrf2-ARE pathway when it protects against early brain injury in a subarachnoid hemorrhage model. ( Chen, G; He, WC; Huo, L; Liu, HX; Ma, C; Meng, CJ; Shen, XM; Shu, Z; Sun, XB; Wang, Z; Zhang, J; Zhu, GQ, 2012) |
"The current study was undertaken to determine whether melatonin therapy reverses vasospasm and prevents apoptosis by inhibiting lipid peroxidation in an experimental subarachnoid hemorrhage (SAH) model." | 7.73 | Effect of melatonin on cerebral vasospasm following experimental subarachnoid hemorrhage. ( Altinors, N; Atalay, B; Aydin, MV; Caner, H; Cekinmez, M; Ozen, O; Sen, O, 2005) |
"In our study, we established a subarachnoid hemorrhage model in male SD rats." | 5.91 | Melatonin alleviates early brain injury by inhibiting the NRF2-mediated ferroptosis pathway after subarachnoid hemorrhage. ( Gao, C; Jiang, GY; Li, C; Li, JY; Liu, HL; Liu, N; Ma, SJ; Yan, C; Yan, HC; Yang, HR, 2023) |
"Melatonin is a sleep-promoting hormone known to have cerebral anti-inflammatory properties." | 5.62 | Melatonin and risk of mortality in subjects with aneurysmal subarachnoid hemorrhage. ( Allan, LD; Bayman, E; Galet, C; Hasan, D; Lin, SH; Rogers, WK; Zanaty, M, 2021) |
"Melatonin or vehicle was injected intraperitoneally 2 hr after SAH." | 5.43 | Melatonin attenuated early brain injury induced by subarachnoid hemorrhage via regulating NLRP3 inflammasome and apoptosis signaling. ( Deng, C; Di, S; Dong, Y; Fan, C; Hu, W; Jiang, S; Liang, G; Ma, Z; Reiter, RJ; Wu, G; Xin, Z; Yan, X; Yang, Y, 2016) |
"Melatonin is a strong antioxidant that has beneficial effects against SAH in rats, including reduced mortality and reduced neurological deficits." | 5.42 | Melatonin attenuates neurogenic pulmonary edema via the regulation of inflammation and apoptosis after subarachnoid hemorrhage in rats. ( Cao, S; Chen, G; Chen, J; Duan, H; Gu, C; Li, J; Qian, C; Wang, L; Yan, F; Yu, X, 2015) |
"Melatonin is a strong antioxidant that has low toxicity and easily passes through the blood-brain barrier." | 5.35 | Melatonin decreases mortality following severe subarachnoid hemorrhage. ( Ayer, RE; Chen, W; Sugawara, T; Tong, W; Zhang, JH, 2008) |
"Melatonin is a strong antioxidant that has low toxicity and easily passes through the BBB." | 5.35 | Effects of melatonin in early brain injury following subarachnoid hemorrhage. ( Ayer, RE; Sugawara, T; Zhang, JH, 2008) |
"The melatonin was administered intraperitoneally at a dose of 5 mg/kg/12 h simultaneously with SAH from day 0 to day 5." | 5.35 | Influence of melatonin on cerebrovascular proinflammatory mediators expression and oxidative stress following subarachnoid hemorrhage in rabbits. ( Chen, G; Dong, W; Fang, Q; Wang, Z; Zhu, W, 2009) |
"To determine whether melatonin (MLT) mitigates white matter (WM) injury by attenuating NOD-like receptor family pyrin domain-containing 3 (NLRP3)-associated oligodendrocyte apoptosis after subarachnoid hemorrhage (SAH)." | 3.96 | Melatonin Attenuates White Matter Injury via Reducing Oligodendrocyte Apoptosis After Subarachnoid Hemorrhage in Mice. ( Dong, Y; Feng, H; Hao, G; Li, G; Liang, G; Liu, D; Pan, P; Zou, Z, 2020) |
"The objective of this study was to identify the protective effect of melatonin (MT) against early brain injury (EBI) following subarachnoid hemorrhage (SAH) and explore the underlying molecular mechanism." | 3.88 | Long non-coding RNA and microRNA-675/let-7a mediates the protective effect of melatonin against early brain injury after subarachnoid hemorrhage via targeting TP53 and neural growth factor. ( He, Y; Li, S; Sun, B; Tang, W; Wen, L; Yang, S, 2018) |
"BACKGROUND A mouse model of subarachnoid hemorrhage (SAH) investigated the effects of melatonin treatment on the generation of reactive oxygen species (ROS) and the activation of the SIRT3 gene in early brain injury (EBI)." | 3.88 | Melatonin Treatment Regulates SIRT3 Expression in Early Brain Injury (EBI) Due to Reactive Oxygen Species (ROS) in a Mouse Model of Subarachnoid Hemorrhage (SAH). ( Chen, X; Hang, C; Li, S; Sun, B; Yang, S, 2018) |
"BACKGROUND The aim of this study was to investigate whether melatonin is involved in brain injury following subarachnoid hemorrhage (SAH)." | 3.88 | Melatonin Upregulates Nuclear Factor Erythroid-2 Related Factor 2 (Nrf2) and Mediates Mitophagy to Protect Against Early Brain Injury After Subarachnoid Hemorrhage. ( Hang, C; Li, S; Sun, B; Yang, S, 2018) |
"Melatonin (Mel) has been reported to alleviate early brain injury (EBI) following subarachnoid hemorrhage (SAH)." | 3.85 | Melatonin Attenuates Early Brain Injury via the Melatonin Receptor/Sirt1/NF-κB Signaling Pathway Following Subarachnoid Hemorrhage in Mice. ( Li, X; Lin, Y; Liu, H; Qu, Y; Wang, B; Yue, L; Zhang, J; Zhao, L, 2017) |
" The objective of this prospective study was to determine, in a cohort of patients with treated ruptured intracranial aneurysm (IA), the proportion of asthenia at 2months, in a cohort of patients treated with melatonin and in a control cohort." | 3.83 | Effects of melatonin in the treatment of asthenia in aneurysmal subarachnoid hemorrhage. ( Curey, S; Derrey, S; Ferracci, FX; Gilard, V; Langlois, O; Proust, F, 2016) |
"Melatonin is a strong antioxidant that has beneficial effects against early brain injury (EBI) following a subarachnoid hemorrhage (SAH) in rats; protection includes reduced mortality and brain water content." | 3.80 | Melatonin-enhanced autophagy protects against neural apoptosis via a mitochondrial pathway in early brain injury following a subarachnoid hemorrhage. ( Chen, G; Chen, J; Gu, C; Hu, Q; Li, J; Wang, L; Wu, C; Yan, F; Yan, W, 2014) |
"Melatonin is a strong anti-oxidant that has beneficial effects against early brain injury (EBI) following a subarachnoid hemorrhage (SAH) in rats; protection includes the reduction of both mortality and neurological deficits." | 3.80 | Melatonin attenuates inflammatory response-induced brain edema in early brain injury following a subarachnoid hemorrhage: a possible role for the regulation of pro-inflammatory cytokines. ( Chen, G; Chen, J; Gu, C; Li, J; Mo, H; Qian, C; Wang, L; Yan, F; Yan, W, 2014) |
"Previous studies proved that melatonin protected against secondary brain damage by modulating oxidative stress after experimental subarachnoid hemorrhage (SAH), but it has not been evaluated yet about its effects on inflammatory pathway and secondary cognitive dysfunction in SAH model." | 3.79 | Melatonin alleviates secondary brain damage and neurobehavioral dysfunction after experimental subarachnoid hemorrhage: possible involvement of TLR4-mediated inflammatory pathway. ( Chen, G; Ji, C; Wang, Z; Wu, L; You, W, 2013) |
"Melatonin has beneficial effects against early brain injury (EBI) by modulating cerebral oxidative stress after experimental subarachnoid hemorrhage (SAH); however, few investigations relate to the precise underlying molecular mechanisms." | 3.78 | Melatonin activates the Nrf2-ARE pathway when it protects against early brain injury in a subarachnoid hemorrhage model. ( Chen, G; He, WC; Huo, L; Liu, HX; Ma, C; Meng, CJ; Shen, XM; Shu, Z; Sun, XB; Wang, Z; Zhang, J; Zhu, GQ, 2012) |
"The current study was undertaken to determine whether melatonin therapy reverses vasospasm and prevents apoptosis by inhibiting lipid peroxidation in an experimental subarachnoid hemorrhage (SAH) model." | 3.73 | Effect of melatonin on cerebral vasospasm following experimental subarachnoid hemorrhage. ( Altinors, N; Atalay, B; Aydin, MV; Caner, H; Cekinmez, M; Ozen, O; Sen, O, 2005) |
"In our study, we established a subarachnoid hemorrhage model in male SD rats." | 1.91 | Melatonin alleviates early brain injury by inhibiting the NRF2-mediated ferroptosis pathway after subarachnoid hemorrhage. ( Gao, C; Jiang, GY; Li, C; Li, JY; Liu, HL; Liu, N; Ma, SJ; Yan, C; Yan, HC; Yang, HR, 2023) |
"Melatonin is a sleep-promoting hormone known to have cerebral anti-inflammatory properties." | 1.62 | Melatonin and risk of mortality in subjects with aneurysmal subarachnoid hemorrhage. ( Allan, LD; Bayman, E; Galet, C; Hasan, D; Lin, SH; Rogers, WK; Zanaty, M, 2021) |
"Melatonin or vehicle was injected intraperitoneally 2 hr after SAH." | 1.43 | Melatonin attenuated early brain injury induced by subarachnoid hemorrhage via regulating NLRP3 inflammasome and apoptosis signaling. ( Deng, C; Di, S; Dong, Y; Fan, C; Hu, W; Jiang, S; Liang, G; Ma, Z; Reiter, RJ; Wu, G; Xin, Z; Yan, X; Yang, Y, 2016) |
"Melatonin is a strong antioxidant that has beneficial effects against SAH in rats, including reduced mortality and reduced neurological deficits." | 1.42 | Melatonin attenuates neurogenic pulmonary edema via the regulation of inflammation and apoptosis after subarachnoid hemorrhage in rats. ( Cao, S; Chen, G; Chen, J; Duan, H; Gu, C; Li, J; Qian, C; Wang, L; Yan, F; Yu, X, 2015) |
"The melatonin was administered intraperitoneally at a dose of 5 mg/kg/12 h simultaneously with SAH from day 0 to day 5." | 1.35 | Influence of melatonin on cerebrovascular proinflammatory mediators expression and oxidative stress following subarachnoid hemorrhage in rabbits. ( Chen, G; Dong, W; Fang, Q; Wang, Z; Zhu, W, 2009) |
"Melatonin is a strong antioxidant that has low toxicity and easily passes through the BBB." | 1.35 | Effects of melatonin in early brain injury following subarachnoid hemorrhage. ( Ayer, RE; Sugawara, T; Zhang, JH, 2008) |
"Melatonin is a strong antioxidant that has low toxicity and easily passes through the blood-brain barrier." | 1.35 | Melatonin decreases mortality following severe subarachnoid hemorrhage. ( Ayer, RE; Chen, W; Sugawara, T; Tong, W; Zhang, JH, 2008) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 7 (29.17) | 29.6817 |
2010's | 12 (50.00) | 24.3611 |
2020's | 5 (20.83) | 2.80 |
Authors | Studies |
---|---|
Lin, SH | 1 |
Galet, C | 1 |
Zanaty, M | 1 |
Bayman, E | 1 |
Rogers, WK | 1 |
Hasan, D | 1 |
Allan, LD | 1 |
Xu, Z | 1 |
Zhang, F | 1 |
Xu, H | 1 |
Yang, F | 1 |
Zhou, G | 1 |
Tong, M | 1 |
Li, Y | 1 |
Yang, S | 4 |
Ma, SJ | 1 |
Li, C | 1 |
Yan, C | 1 |
Liu, N | 1 |
Jiang, GY | 1 |
Yang, HR | 1 |
Yan, HC | 1 |
Li, JY | 1 |
Liu, HL | 1 |
Gao, C | 1 |
Liu, D | 1 |
Dong, Y | 2 |
Li, G | 1 |
Zou, Z | 1 |
Hao, G | 1 |
Feng, H | 1 |
Pan, P | 1 |
Liang, G | 2 |
Zhan, CP | 1 |
Zhuge, CJ | 1 |
Yan, XJ | 1 |
Dai, WM | 1 |
Yu, GF | 1 |
Cao, S | 2 |
Shrestha, S | 1 |
Li, J | 4 |
Yu, X | 2 |
Chen, J | 4 |
Yan, F | 4 |
Ying, G | 1 |
Gu, C | 4 |
Wang, L | 4 |
Chen, G | 7 |
Tang, W | 1 |
He, Y | 1 |
Wen, L | 1 |
Sun, B | 3 |
Li, S | 3 |
Chen, X | 1 |
Hang, C | 2 |
Wang, Z | 3 |
Wu, L | 1 |
You, W | 1 |
Ji, C | 1 |
Wu, C | 1 |
Hu, Q | 1 |
Yan, W | 2 |
Qian, C | 2 |
Mo, H | 1 |
Duan, H | 1 |
Fan, C | 1 |
Hu, W | 1 |
Jiang, S | 1 |
Ma, Z | 1 |
Yan, X | 1 |
Deng, C | 1 |
Di, S | 1 |
Xin, Z | 1 |
Wu, G | 1 |
Yang, Y | 1 |
Reiter, RJ | 1 |
Zhao, L | 1 |
Liu, H | 1 |
Yue, L | 1 |
Zhang, J | 2 |
Li, X | 1 |
Wang, B | 1 |
Lin, Y | 1 |
Qu, Y | 1 |
Gilard, V | 1 |
Ferracci, FX | 1 |
Langlois, O | 1 |
Derrey, S | 1 |
Proust, F | 1 |
Curey, S | 1 |
Ersahin, M | 1 |
Toklu, HZ | 1 |
Cetinel, S | 1 |
Yüksel, M | 1 |
Yeğen, BC | 1 |
Sener, G | 1 |
Ayer, RE | 2 |
Sugawara, T | 2 |
Zhang, JH | 2 |
Aladag, MA | 1 |
Turkoz, Y | 1 |
Parlakpinar, H | 1 |
Ozen, H | 1 |
Egri, M | 1 |
Unal, SC | 1 |
Fang, Q | 1 |
Zhu, W | 1 |
Dong, W | 1 |
Ma, C | 1 |
Meng, CJ | 1 |
Zhu, GQ | 1 |
Sun, XB | 1 |
Huo, L | 1 |
Liu, HX | 1 |
He, WC | 1 |
Shen, XM | 1 |
Shu, Z | 1 |
Martinez-Cruz, F | 1 |
Espinar, A | 1 |
Pozo, D | 1 |
Osuna, C | 1 |
Guerrero, JM | 1 |
Aydin, MV | 1 |
Caner, H | 1 |
Sen, O | 1 |
Ozen, O | 1 |
Atalay, B | 1 |
Cekinmez, M | 1 |
Altinors, N | 1 |
Chen, W | 1 |
Tong, W | 1 |
24 other studies available for melatonin and Hemorrhage, Subarachnoid
Article | Year |
---|---|
Melatonin and risk of mortality in subjects with aneurysmal subarachnoid hemorrhage.
Topics: Adult; Aged; Antioxidants; Case-Control Studies; Cohort Studies; Female; Humans; Male; Melatonin; Mi | 2021 |
Melatonin affects hypoxia-inducible factor 1α and ameliorates delayed brain injury following subarachnoid hemorrhage via H19/miR-675/HIF1A/TLR4.
Topics: Animals; Apoptosis; Brain; Hypoxia-Inducible Factor 1, alpha Subunit; Male; Melatonin; Mice; Mice, I | 2022 |
Melatonin alleviates early brain injury by inhibiting the NRF2-mediated ferroptosis pathway after subarachnoid hemorrhage.
Topics: Animals; Brain Injuries; Ferroptosis; Iron; Male; Melatonin; NF-E2-Related Factor 2; Rats; Rats, Spr | 2023 |
Melatonin Attenuates White Matter Injury via Reducing Oligodendrocyte Apoptosis After Subarachnoid Hemorrhage in Mice.
Topics: Animals; Apoptosis; Male; Melatonin; Mice; Mice, Inbred C57BL; Neuroprotective Agents; Oligodendrogl | 2020 |
Measuring serum melatonin concentrations to predict clinical outcome after aneurysmal subarachnoid hemorrhage.
Topics: Humans; Melatonin; Prognosis; Prospective Studies; Subarachnoid Hemorrhage | 2021 |
Melatonin-mediated mitophagy protects against early brain injury after subarachnoid hemorrhage through inhibition of NLRP3 inflammasome activation.
Topics: Animals; Biomarkers; Brain; Cytokines; Inflammasomes; Inflammation Mediators; Male; Melatonin; Mitop | 2017 |
Long non-coding RNA and microRNA-675/let-7a mediates the protective effect of melatonin against early brain injury after subarachnoid hemorrhage via targeting TP53 and neural growth factor.
Topics: Animals; Apoptosis; Brain; Brain Injuries; Cell Line, Tumor; Humans; Male; Melatonin; Mice, Inbred C | 2018 |
Melatonin Treatment Regulates SIRT3 Expression in Early Brain Injury (EBI) Due to Reactive Oxygen Species (ROS) in a Mouse Model of Subarachnoid Hemorrhage (SAH).
Topics: Animals; Apoptosis; Brain Edema; Brain Injuries; Cell Line, Tumor; Disease Models, Animal; Humans; M | 2018 |
Melatonin Upregulates Nuclear Factor Erythroid-2 Related Factor 2 (Nrf2) and Mediates Mitophagy to Protect Against Early Brain Injury After Subarachnoid Hemorrhage.
Topics: Animals; Apoptosis; Autophagy; Brain Edema; Brain Injuries; Male; Melatonin; Mice; Mice, Inbred C57B | 2018 |
Melatonin alleviates secondary brain damage and neurobehavioral dysfunction after experimental subarachnoid hemorrhage: possible involvement of TLR4-mediated inflammatory pathway.
Topics: Animals; Blotting, Western; Brain Injuries; Electrophoretic Mobility Shift Assay; Enzyme-Linked Immu | 2013 |
Melatonin-enhanced autophagy protects against neural apoptosis via a mitochondrial pathway in early brain injury following a subarachnoid hemorrhage.
Topics: Analysis of Variance; Animals; Apoptosis; Autophagy; bcl-2-Associated X Protein; Brain Injuries; Cyt | 2014 |
Melatonin attenuates inflammatory response-induced brain edema in early brain injury following a subarachnoid hemorrhage: a possible role for the regulation of pro-inflammatory cytokines.
Topics: Animals; Brain Edema; Brain Injuries; Cytokines; Inflammation Mediators; Male; Melatonin; Rats; Rats | 2014 |
Melatonin attenuates neurogenic pulmonary edema via the regulation of inflammation and apoptosis after subarachnoid hemorrhage in rats.
Topics: Animals; Antioxidants; Apoptosis; Inflammation; Interleukin-1beta; Matrix Metalloproteinase 9; Melat | 2015 |
Melatonin attenuated early brain injury induced by subarachnoid hemorrhage via regulating NLRP3 inflammasome and apoptosis signaling.
Topics: Animals; Apoptosis; Blood-Brain Barrier; Brain Hemorrhage, Traumatic; Caspase 1; Gene Expression Reg | 2016 |
Melatonin Attenuates Early Brain Injury via the Melatonin Receptor/Sirt1/NF-κB Signaling Pathway Following Subarachnoid Hemorrhage in Mice.
Topics: Animals; Antioxidants; Brain Injuries; Male; Melatonin; Mice; Mice, Inbred C57BL; NF-kappa B; Random | 2017 |
Effects of melatonin in the treatment of asthenia in aneurysmal subarachnoid hemorrhage.
Topics: Adult; Aged; Aneurysm, Ruptured; Asthenia; Female; Humans; Intracranial Aneurysm; Male; Melatonin; M | 2016 |
Melatonin reduces experimental subarachnoid hemorrhage-induced oxidative brain damage and neurological symptoms.
Topics: Analysis of Variance; Animals; Antioxidants; Basilar Artery; Brain; Disease Models, Animal; Lipid Pe | 2009 |
Effects of melatonin in early brain injury following subarachnoid hemorrhage.
Topics: Animals; Brain Edema; Brain Injuries; Disease Models, Animal; Dose-Response Relationship, Drug; Male | 2008 |
Melatonin ameliorates cerebral vasospasm after experimental subarachnoidal haemorrhage correcting imbalance of nitric oxide levels in rats.
Topics: Animals; Antioxidants; Arginase; Basilar Artery; Brain; Constriction, Pathologic; Free Radicals; Imm | 2009 |
Influence of melatonin on cerebrovascular proinflammatory mediators expression and oxidative stress following subarachnoid hemorrhage in rabbits.
Topics: Animals; Antioxidants; Disease Models, Animal; Electrophoretic Mobility Shift Assay; Enzyme-Linked I | 2009 |
Melatonin activates the Nrf2-ARE pathway when it protects against early brain injury in a subarachnoid hemorrhage model.
Topics: Animals; Blotting, Western; Brain Injuries; Disease Models, Animal; Immunohistochemistry; In Situ Ni | 2012 |
Melatonin prevents focal rat cerebellum injury as assessed by induction of heat shock protein (HO-1) following subarachnoid injections of lysed blood.
Topics: Animals; Antioxidants; Brain Chemistry; Cerebellum; Disease Models, Animal; Female; Heme Oxygenase ( | 2002 |
Effect of melatonin on cerebral vasospasm following experimental subarachnoid hemorrhage.
Topics: Animals; Apoptosis; Basilar Artery; Cell Count; Disease Models, Animal; Endothelial Cells; Female; M | 2005 |
Melatonin decreases mortality following severe subarachnoid hemorrhage.
Topics: Animals; Blood-Brain Barrier; Disease Models, Animal; Male; Melatonin; Neuroprotective Agents; Oxida | 2008 |