Page last updated: 2024-10-30

meclofenamic acid and Glioma

meclofenamic acid has been researched along with Glioma in 2 studies

Meclofenamic Acid: A non-steroidal anti-inflammatory agent with antipyretic and antigranulation activities. It also inhibits prostaglandin biosynthesis.
meclofenamic acid : An aminobenzoic acid that is anthranilic acid in which one of the hydrogens attached to the nitrogen is replaced by a 2,6-dichloro-3-methylphenyl group. A non-steroidal anti-inflammatory drug, it is used as the sodium salt for the treatment of dysmenorrhoea (painful periods), osteoarthritis and rheumatoid arthritis.

Glioma: Benign and malignant central nervous system neoplasms derived from glial cells (i.e., astrocytes, oligodendrocytes, and ependymocytes). Astrocytes may give rise to astrocytomas (ASTROCYTOMA) or glioblastoma multiforme (see GLIOBLASTOMA). Oligodendrocytes give rise to oligodendrogliomas (OLIGODENDROGLIOMA) and ependymocytes may undergo transformation to become EPENDYMOMA; CHOROID PLEXUS NEOPLASMS; or colloid cysts of the third ventricle. (From Escourolle et al., Manual of Basic Neuropathology, 2nd ed, p21)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's0 (0.00)24.3611
2020's1 (50.00)2.80

Authors

AuthorsStudies
Xiao, L1
Li, X1
Mu, Z1
Zhou, J1
Zhou, P1
Xie, C1
Jiang, S1
Li, Z1
Yan, Y1
Powers, EA1
Ying, X1
Janjua, K1
Garyantes, T1
Baron, B1

Other Studies

2 other studies available for meclofenamic acid and Glioma

ArticleYear
FTO Inhibition Enhances the Antitumor Effect of Temozolomide by Targeting MYC-miR-155/23a Cluster-MXI1 Feedback Circuit in Glioma.
    Cancer research, 2020, 09-15, Volume: 80, Issue:18

    Topics: Alpha-Ketoglutarate-Dependent Dioxygenase FTO; Animals; Antineoplastic Agents, Alkylating; Basic Hel

2020
Identification of gap junction blockers using automated fluorescence microscopy imaging.
    Journal of biomolecular screening, 2003, Volume: 8, Issue:5

    Topics: Animals; Automation; Cell Communication; Cell Division; Central Nervous System Neoplasms; Dose-Respo

2003