Page last updated: 2024-10-30

meclizine and Parkinson Disease

meclizine has been researched along with Parkinson Disease in 1 studies

Meclizine: A histamine H1 antagonist used in the treatment of motion sickness, vertigo, and nausea during pregnancy and radiation sickness.

Parkinson Disease: A progressive, degenerative neurologic disease characterized by a TREMOR that is maximal at rest, retropulsion (i.e. a tendency to fall backwards), rigidity, stooped posture, slowness of voluntary movements, and a masklike facial expression. Pathologic features include loss of melanin containing neurons in the substantia nigra and other pigmented nuclei of the brainstem. LEWY BODIES are present in the substantia nigra and locus coeruleus but may also be found in a related condition (LEWY BODY DISEASE, DIFFUSE) characterized by dementia in combination with varying degrees of parkinsonism. (Adams et al., Principles of Neurology, 6th ed, p1059, pp1067-75)

Research Excerpts

ExcerptRelevanceReference
"Meclizine is a well-tolerated drug routinely used as an anti-histamine agent in the management of disequilibrium."5.43Meclizine-induced enhanced glycolysis is neuroprotective in Parkinson disease cell models. ( Chau, KY; Hong, CT; Schapira, AH, 2016)
"Meclizine is a well-tolerated drug routinely used as an anti-histamine agent in the management of disequilibrium."1.43Meclizine-induced enhanced glycolysis is neuroprotective in Parkinson disease cell models. ( Chau, KY; Hong, CT; Schapira, AH, 2016)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Hong, CT1
Chau, KY1
Schapira, AH1

Other Studies

1 other study available for meclizine and Parkinson Disease

ArticleYear
Meclizine-induced enhanced glycolysis is neuroprotective in Parkinson disease cell models.
    Scientific reports, 2016, 05-05, Volume: 6

    Topics: Animals; Apoptosis; Cell Line; Fructosediphosphates; Gene Expression Regulation; Glycolysis; Humans;

2016