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ly 171883 and Hyperinsulinism

ly 171883 has been researched along with Hyperinsulinism in 1 studies

LY 171883: structure in first source; leukotriene receptor antagonist
tomelukast : A member of the class of acetophenones that is 1-phenylethanone substituted at position 2 by a hydroxy group, a propyl group at position 3 and a 4-(1H-tetrazol-5-yl)butoxy group at position 4. A leukotriene antagonist, it exhibits anti-asthmatic activity.

Hyperinsulinism: A syndrome with excessively high INSULIN levels in the BLOOD. It may cause HYPOGLYCEMIA. Etiology of hyperinsulinism varies, including hypersecretion of a beta cell tumor (INSULINOMA); autoantibodies against insulin (INSULIN ANTIBODIES); defective insulin receptor (INSULIN RESISTANCE); or overuse of exogenous insulin or HYPOGLYCEMIC AGENTS.

Research Excerpts

ExcerptRelevanceReference
"Moreover, the hyperinsulinemia is mediated in part by prostaglandins just like the well-studied febrile response."1.28Central interleukin 1-elicited hyperinsulinemia is mediated by prostaglandins but not autonomics. ( Cornell, RP, 1989)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19901 (100.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cornell, RP1

Other Studies

1 other study available for ly 171883 and Hyperinsulinism

ArticleYear
Central interleukin 1-elicited hyperinsulinemia is mediated by prostaglandins but not autonomics.
    The American journal of physiology, 1989, Volume: 257, Issue:4 Pt 2

    Topics: Acetophenones; Adrenocorticotropic Hormone; Animals; Atropine; Autonomic Nervous System; Body Temper

1989