Page last updated: 2024-08-23

lovastatin and Di Guglielmo Disease

lovastatin has been researched along with Di Guglielmo Disease in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's4 (66.67)18.2507
2000's1 (16.67)29.6817
2010's0 (0.00)24.3611
2020's1 (16.67)2.80

Authors

AuthorsStudies
Ben-David, Y; Gajendran, B; Gao, J; Hu, A; Hu, J; Kuang, Y; Liu, W; Sheng, D; Wang, C; Xiao, X; Yu, F; Yu, K; Zacksenhaus, E1
Bhullar, RP1
Bellincampi, L; Finazzi Agrò, A; Maccarrone, M; Melino, G1
Csipai, M; Glasmacher, A; Huhn, D; Lefterova, P; Neubauer, A; Scheffold, C; Schmidt-Wolf, IG; Schöttker, B1
Erdman, RA; Maltese, WA; Repko, EM; Sheridan, KM1
Maltese, WA; Sheridan, KM1

Other Studies

6 other study(ies) available for lovastatin and Di Guglielmo Disease

ArticleYear
Lovastatin inhibits erythroleukemia progression through KLF2-mediated suppression of MAPK/ERK signaling.
    BMC cancer, 2023, Apr-04, Volume: 23, Issue:1

    Topics: Animals; Apoptosis; Cholesterol; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Kruppel-Like Transcription Factors; Leukemia, Erythroblastic, Acute; Lovastatin; Mice; Neoplasms

2023
Expression of a 24 kDa GTP-binding protein (Gn24) is increased in lovastatin treated human erythroleukemia cells.
    Molecular and cellular biochemistry, 1996, Mar-09, Volume: 156, Issue:1

    Topics: ADP Ribose Transferases; Anticholesteremic Agents; Bacterial Proteins; Botulinum Toxins; Cycloheximide; Enzyme Inhibitors; Gene Expression Regulation, Leukemic; GTP-Binding Proteins; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Leukemia, Erythroblastic, Acute; Lovastatin; Molecular Weight; Neoplasm Proteins; Protein Prenylation; Protein Processing, Post-Translational; Protein Synthesis Inhibitors; Tumor Cells, Cultured

1996
Cholesterol, but not its esters, triggers programmed cell death in human erythroleukemia K562 cells.
    European journal of biochemistry, 1998, Apr-01, Volume: 253, Issue:1

    Topics: Anticholesteremic Agents; Apoptosis; Cell Division; Cholesterol; Cholesterol Esters; Humans; Lamin Type B; Lamins; Leukemia, Erythroblastic, Acute; Lovastatin; Membrane Fluidity; Membrane Lipids; Nuclear Proteins; Polyisoprenyl Phosphates; Protein Prenylation; ras Proteins; Sesquiterpenes; Tumor Cells, Cultured

1998
Increased sensitivity of myeloid leukemia cell lines: potential of lovastatin as bone-marrow-purging agent.
    Acta haematologica, 2000, Volume: 104, Issue:2-3

    Topics: Bone Marrow Cells; Bone Marrow Purging; Bone Marrow Transplantation; Cell Survival; Clone Cells; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Growth Inhibitors; Hematopoietic Stem Cells; Humans; K562 Cells; Leukemia, Erythroblastic, Acute; Leukemia, Myeloid, Acute; Lovastatin; Monocytes; Transplantation, Autologous

2000
Post-translational modification of low molecular mass GTP-binding proteins by isoprenoid.
    The Journal of biological chemistry, 1990, Feb-05, Volume: 265, Issue:4

    Topics: Animals; Cell Line; Cell Membrane; Cytosol; Electrophoresis, Gel, Two-Dimensional; Electrophoresis, Polyacrylamide Gel; GTP-Binding Proteins; Guanosine Triphosphate; Immunoblotting; Leukemia, Erythroblastic, Acute; Lovastatin; Methylation; Mevalonic Acid; Mice; Molecular Weight; Protein Binding; Protein Processing, Post-Translational; Rats

1990
Isoprenoid modification of G25K (Gp), a low molecular mass GTP-binding protein distinct from p21ras.
    The Journal of biological chemistry, 1990, Oct-15, Volume: 265, Issue:29

    Topics: Animals; Electrophoresis, Polyacrylamide Gel; GTP-Binding Proteins; Immunoblotting; Kinetics; Leukemia, Erythroblastic, Acute; Leukemia, Experimental; Lovastatin; Mevalonic Acid; Mice; Molecular Weight; Protein Processing, Post-Translational; Proto-Oncogene Proteins p21(ras)

1990