lovastatin has been researched along with Cancer of Cervix in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Duan, S; Gao, J; Hu, N; Lin, J; Lin, S | 1 |
| Chang, CC; Huang, SM; Lin, CK; Liu, ST | 1 |
| Brañes, J; Carvajal, J; Chen-Lin, K; Cuello, M; Gejman, R; Kato, S; Oliva, B; Owen, GI; Sadarangani, A; Smalley, S | 1 |
| Chen, E; Chin, S; Dimitroulakos, J; Irish, J; Kamel-Reid, S; Knox, JJ; LaFramboise, S; Moore, MJ; Oza, AM; Siu, LL | 1 |
1 trial(s) available for lovastatin and Cancer of Cervix
| Article | Year |
|---|---|
A Phase I trial of prolonged administration of lovastatin in patients with recurrent or metastatic squamous cell carcinoma of the head and neck or of the cervix.
Topics: Adult; Aged; Carcinoma, Squamous Cell; Female; Head and Neck Neoplasms; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Lovastatin; Male; Middle Aged; Neoplasm Recurrence, Local; Treatment Outcome; Uterine Cervical Neoplasms | 2005 |
3 other study(ies) available for lovastatin and Cancer of Cervix
| Article | Year |
|---|---|
Lovastatin inhibits the proliferation of human cervical cancer hela cells through the regulation of tp53 pathway by mir-92a-1-5p.
Topics: Apoptosis; Cell Line, Tumor; Cell Movement; Cell Proliferation; Female; Gene Expression Regulation, Neoplastic; HeLa Cells; Humans; Lovastatin; MicroRNAs; Tumor Suppressor Protein p53; Uterine Cervical Neoplasms | 2022 |
Regulatory mechanisms of fluvastatin and lovastatin for the p21 induction in human cervical cancer HeLa cells.
Topics: Activating Transcription Factor 3; Apoptosis; Autophagy; Cell Cycle; Cyclin D1; Cyclin-Dependent Kinase Inhibitor p21; DNA Damage; Female; Fluvastatin; Gene Expression Regulation, Neoplastic; HeLa Cells; Histone Deacetylase Inhibitors; Humans; Lovastatin; Tumor Suppressor Protein p53; Uterine Cervical Neoplasms | 2019 |
Lipophilic but not hydrophilic statins selectively induce cell death in gynaecological cancers expressing high levels of HMGCoA reductase.
Topics: Cell Death; Cell Line, Tumor; Cell Survival; Drug Resistance, Neoplasm; Drug Synergism; Epithelium; Female; Gene Expression Regulation, Enzymologic; Gene Expression Regulation, Neoplastic; Genital Neoplasms, Female; Humans; Hydroxymethylglutaryl CoA Reductases; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Lipids; Lovastatin; Mevalonic Acid; Ovarian Neoplasms; Polyisoprenyl Phosphates; Pravastatin; Sesquiterpenes; Signal Transduction; Simvastatin; Uterine Cervical Neoplasms; Water | 2010 |