lithium-chloride and Pulmonary-Edema

lithium-chloride has been researched along with Pulmonary-Edema* in 2 studies

Other Studies

2 other study(ies) available for lithium-chloride and Pulmonary-Edema

Article	Year
Lithium dilution cardiac output measurement in oleic acid-induced pulmonary edema. Journal of cardiothoracic and vascular anesthesia, 2002, Volume: 16, Issue:3 To determine whether lung injury influences the accuracy of lithium dilution cardiac output (CO) measurement.. Animal experimental study.. Animal experimental laboratory.. Swine (n = 23) weighing 26.4 +/- 2.47 kg (mean +/- SD).. The animals were anesthetized and tracheotomized, then a pulmonary artery catheter was inserted into the right jugular vein, and a catheter (18G) was placed in the femoral artery. After median sternotomy and pericardiotomy, a left ventricular catheter (18G) was directly inserted. CO was measured by giving a bolus injection of lithium chloride into either the right atrium or the left ventricle in each animal. After control measurements, permeability pulmonary edema was initiated by infusing oleic acid into the central vein (injury). About 2 hours after oleic acid infusion, CO measurements were repeated in the same manner as the control measurement had been taken.. Under each condition, right atrium lithium injection was similar to left ventricle lithium injection. The mean of these differences at injury (-0.06 +/- 0.55 L/min) was the same as that at control (-0.05 +/- 0.36 L/min).. Although the variability of lithium dilution CO measurement after oleic acid-induced pulmonary edema was greater than that of the control, this technique was acceptable even in cases of lung injury. Topics: Animals; Cardiac Catheterization; Cardiac Output; Catheterization, Swan-Ganz; Heart Atria; Heart Ventricles; Indicator Dilution Techniques; Lithium Chloride; Oleic Acid; Pulmonary Edema; Swine	2002
[Effect of lithium preparations on the toxic effects of adrenaline]. Biulleten' eksperimental'noi biologii i meditsiny, 1981, Volume: 91, Issue:6 Experiments on rats have shown that intravenous injection of adrenaline in a dose of 0.3-0.4 mg/kg causes cardiac arrhythmia. In this case the primary arrhythmia developing immediately after adrenaline injection is followed by the recovery of sinusal rhythm which was replaced by the secondary arrhythmia. Apart from arrhythmias, there developed pulmonary edema. The animals died 2--3 minutes after adrenaline injections. Lithium chloride and lithium hydroxybutyrate removed the secondary arrhythmia and pulmonary edema. Lithium hydroxybutyrate has proved to be more effective. Topics: Animals; Anti-Arrhythmia Agents; Arrhythmias, Cardiac; Chlorides; Epinephrine; Hydroxybutyrates; Lithium; Lithium Chloride; Male; Organometallic Compounds; Pulmonary Edema; Rats	1981

Article

Year

Lithium dilution cardiac output measurement in oleic acid-induced pulmonary edema.

Journal of cardiothoracic and vascular anesthesia, 2002, Volume: 16, Issue:3

To determine whether lung injury influences the accuracy of lithium dilution cardiac output (CO) measurement.. Animal experimental study.. Animal experimental laboratory.. Swine (n = 23) weighing 26.4 +/- 2.47 kg (mean +/- SD).. The animals were anesthetized and tracheotomized, then a pulmonary artery catheter was inserted into the right jugular vein, and a catheter (18G) was placed in the femoral artery. After median sternotomy and pericardiotomy, a left ventricular catheter (18G) was directly inserted. CO was measured by giving a bolus injection of lithium chloride into either the right atrium or the left ventricle in each animal. After control measurements, permeability pulmonary edema was initiated by infusing oleic acid into the central vein (injury). About 2 hours after oleic acid infusion, CO measurements were repeated in the same manner as the control measurement had been taken.. Under each condition, right atrium lithium injection was similar to left ventricle lithium injection. The mean of these differences at injury (-0.06 +/- 0.55 L/min) was the same as that at control (-0.05 +/- 0.36 L/min).. Although the variability of lithium dilution CO measurement after oleic acid-induced pulmonary edema was greater than that of the control, this technique was acceptable even in cases of lung injury.

Topics: Animals; Cardiac Catheterization; Cardiac Output; Catheterization, Swan-Ganz; Heart Atria; Heart Ventricles; Indicator Dilution Techniques; Lithium Chloride; Oleic Acid; Pulmonary Edema; Swine

2002

[Effect of lithium preparations on the toxic effects of adrenaline].

Biulleten' eksperimental'noi biologii i meditsiny, 1981, Volume: 91, Issue:6

Experiments on rats have shown that intravenous injection of adrenaline in a dose of 0.3-0.4 mg/kg causes cardiac arrhythmia. In this case the primary arrhythmia developing immediately after adrenaline injection is followed by the recovery of sinusal rhythm which was replaced by the secondary arrhythmia. Apart from arrhythmias, there developed pulmonary edema. The animals died 2--3 minutes after adrenaline injections. Lithium chloride and lithium hydroxybutyrate removed the secondary arrhythmia and pulmonary edema. Lithium hydroxybutyrate has proved to be more effective.

Topics: Animals; Anti-Arrhythmia Agents; Arrhythmias, Cardiac; Chlorides; Epinephrine; Hydroxybutyrates; Lithium; Lithium Chloride; Male; Organometallic Compounds; Pulmonary Edema; Rats

1981