Page last updated: 2024-08-23

lithium and Infarction, Middle Cerebral Artery

lithium has been researched along with Infarction, Middle Cerebral Artery in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's3 (60.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Chao, LC; Huang, CC; Huang, SY; Lee, AH; Lee, EJ; Lin, YW; Shan, YS; Tai, SH; Wu, TS1
Ramagiri, S; Taliyan, R1
Bähr, M; Caglayan, AB; Doeppner, TR; Hermann, DM; Kaltwasser, B; Sanchez-Mendoza, EH1
Chuang, DM; Leeds, P; Leng, Y; Munasinghe, J; Tsai, LK; Wang, Z1
Gu, ZL; Han, R; Qin, ZH; Xu, XH; Zhang, HL1

Other Studies

5 other study(ies) available for lithium and Infarction, Middle Cerebral Artery

ArticleYear
Lithium upregulates growth-associated protein-43 (GAP-43) and postsynaptic density-95 (PSD-95) in cultured neurons exposed to oxygen-glucose deprivation and improves electrophysiological outcomes in rats subjected to transient focal cerebral ischemia foll
    Neurological research, 2022, Volume: 44, Issue:10

    Topics: Animals; Brain Ischemia; Brain-Derived Neurotrophic Factor; Disks Large Homolog 4 Protein; Edetic Acid; GAP-43 Protein; Glucose; Glycogen Synthase Kinase 3 beta; Infarction, Middle Cerebral Artery; Lithium; Lithium Chloride; Male; N-Methylaspartate; Neurons; Neuroprotective Agents; Oxygen; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Sodium Dodecyl Sulfate

2022
Delayed neuroprotection against cerebral ischemia reperfusion injury: putative role of BDNF and GSK-3β.
    Journal of receptor and signal transduction research, 2016, Volume: 36, Issue:4

    Topics: Animals; Brain Ischemia; Brain-Derived Neurotrophic Factor; Gene Expression Regulation; Glycogen Synthase Kinase 3 beta; Humans; Infarction, Middle Cerebral Artery; Lithium; Male; Neuroprotective Agents; Oxidative Stress; Rats; Reperfusion Injury; Tumor Necrosis Factor-alpha

2016
Lithium-induced neuroprotection in stroke involves increased miR-124 expression, reduced RE1-silencing transcription factor abundance and decreased protein deubiquitination by GSK3β inhibition-independent pathways.
    Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 2017, Volume: 37, Issue:3

    Topics: Animals; Glycogen Synthase Kinase 3 beta; Infarction, Middle Cerebral Artery; Lithium; Mice; MicroRNAs; Neovascularization, Physiologic; Neuronal Plasticity; Neuroprotection; Repressor Proteins; Stroke; Ubiquitination

2017
Mesenchymal stem cells primed with valproate and lithium robustly migrate to infarcted regions and facilitate recovery in a stroke model.
    Stroke, 2011, Volume: 42, Issue:10

    Topics: Animals; Cell Movement; Infarction, Middle Cerebral Artery; Lithium; Mesenchymal Stem Cell Transplantation; Mesenchymal Stem Cells; Rats; Recovery of Function; Stroke; Valproic Acid

2011
Enhancement of neuroprotection and heat shock protein induction by combined prostaglandin A1 and lithium in rodent models of focal ischemia.
    Brain research, 2006, Aug-02, Volume: 1102, Issue:1

    Topics: Analysis of Variance; Animals; Blotting, Western; Brain Edema; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Therapy, Combination; Heat-Shock Proteins; HSP72 Heat-Shock Proteins; Immunohistochemistry; Infarction, Middle Cerebral Artery; Lithium; Male; Molecular Chaperones; Neurologic Examination; Neuroprotective Agents; Prostaglandins A; Rats; Rats, Sprague-Dawley; Time Factors; Transcription Factor CHOP

2006