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lg 100268 and Weight Gain

lg 100268 has been researched along with Weight Gain in 2 studies

LG 100268: a retinoid X receptor (RXR) selective compound; structure given in first source

Weight Gain: Increase in BODY WEIGHT over existing weight.

Research Excerpts

ExcerptRelevanceReference
" Long-term (2 weeks) oral treatment with the rexinoids LG100268 and LG100324 reduced food intake and body weight gain, whereas rosiglitazone (BRL49653) tended to increase both food intake and weight gain."7.70The effects of rexinoids and rosiglitazone on body weight and uncoupling protein isoform expression in the Zucker fa/fa rat. ( Cawthorne, MA; Emilsson, V; Heyman, R; Liu, YL; O'Dowd, J; Sennitt, M; Wang, S, 2000)
" Long-term (2 weeks) oral treatment with the rexinoids LG100268 and LG100324 reduced food intake and body weight gain, whereas rosiglitazone (BRL49653) tended to increase both food intake and weight gain."3.70The effects of rexinoids and rosiglitazone on body weight and uncoupling protein isoform expression in the Zucker fa/fa rat. ( Cawthorne, MA; Emilsson, V; Heyman, R; Liu, YL; O'Dowd, J; Sennitt, M; Wang, S, 2000)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Emilsson, V1
O'Dowd, J1
Wang, S1
Liu, YL1
Sennitt, M1
Heyman, R1
Cawthorne, MA1
Ogilvie, KM1
Saladin, R1
Nagy, TR1
Urcan, MS1
Heyman, RA1
Leibowitz, MD1

Other Studies

2 other studies available for lg 100268 and Weight Gain

ArticleYear
The effects of rexinoids and rosiglitazone on body weight and uncoupling protein isoform expression in the Zucker fa/fa rat.
    Metabolism: clinical and experimental, 2000, Volume: 49, Issue:12

    Topics: Adipose Tissue, Brown; Animals; Body Weight; Carrier Proteins; Eating; Ion Channels; Membrane Protei

2000
Activation of the retinoid X receptor suppresses appetite in the rat.
    Endocrinology, 2004, Volume: 145, Issue:2

    Topics: Adipose Tissue; Animals; Anticholesteremic Agents; Apoptosis; Appetite Regulation; Brain; Eating; En

2004