lg 100268 has been researched along with Dermatitis, Atopic in 1 studies
LG 100268: a retinoid X receptor (RXR) selective compound; structure given in first source
Dermatitis, Atopic: A chronic inflammatory genetically determined disease of the skin marked by increased ability to form reagin (IgE), with increased susceptibility to allergic rhinitis and asthma, and hereditary disposition to a lowered threshold for pruritus. It is manifested by lichenification, excoriation, and crusting, mainly on the flexural surfaces of the elbow and knee. In infants it is known as infantile eczema.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Mihály, J | 1 |
Gericke, J | 1 |
Lucas, R | 1 |
de Lera, AR | 1 |
Alvarez, S | 1 |
Törőcsik, D | 1 |
Rühl, R | 1 |
1 other study available for lg 100268 and Dermatitis, Atopic
Article | Year |
---|---|
TSLP expression in the skin is mediated via RARγ-RXR pathways.
Topics: Adolescent; Adult; Animals; Calcitriol; Coumaric Acids; Cytokines; Dermatitis, Atopic; Female; Gene | 2016 |