Page last updated: 2024-08-17

levodopa and Electron Transport Chain Deficiencies, Mitochondrial

levodopa has been researched along with Electron Transport Chain Deficiencies, Mitochondrial in 8 studies

Research

Studies (8)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's5 (62.50)29.6817
2010's3 (37.50)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Caetano, A; Meira, B; Pinto, M; Roque, R1
Cai, H; Matsubara, S; Mukai, M; Nakano, I; Sasaki, H; Sugaya, K; Yabe, I1
Harper, JW; Hoekstra, JG; Huang, CH; Kennedy, SR; Ordureau, A; Pickrell, AM; Sideris, DP; Youle, RJ1
Artuch, R; Briones, P; Carrilho, I; Duarte, S; Garcia-Cazorla, A; Garesse, R; Montoya, J; Nascimento, A; Ormazabal, A; Pineda, M; Sala-Castellvi, P; Serrano, M1
Ekstrand, MI; Galter, D1
Albanese, A; Bellacchio, E; Dallapiccola, B; Elia, AE; Romito, LM; Valente, EM1
Asanuma, M; Diaz-Corrales, FJ; Miyazaki, I; Miyoshi, K; Ogawa, N1
Finsterer, J1

Reviews

3 review(s) available for levodopa and Electron Transport Chain Deficiencies, Mitochondrial

ArticleYear
[Familial progressive external opthalmoplegia, parkinsonism and polyneuropathy associated with POLG1 mutation].
    Rinsho shinkeigaku = Clinical neurology, 2014, Volume: 54, Issue:5

    Topics: Aged; DNA Polymerase gamma; DNA-Directed DNA Polymerase; DNA, Mitochondrial; Female; Gene Deletion; Genes, Dominant; Humans; Levodopa; Male; Menopause, Premature; Mitochondrial Diseases; Muscular Diseases; Mutation; Ophthalmoplegia, Chronic Progressive External; Parkinsonian Disorders; Pedigree; Polyneuropathies; Syndrome

2014
The MitoPark Mouse - an animal model of Parkinson's disease with impaired respiratory chain function in dopamine neurons.
    Parkinsonism & related disorders, 2009, Volume: 15 Suppl 3

    Topics: Animals; Antiparkinson Agents; Disease Models, Animal; DNA-Binding Proteins; Dopamine; Electron Transport; Humans; Levodopa; Mice; Mice, Transgenic; Mitochondrial Diseases; Mitochondrial Proteins; Neurons; Parkinson Disease; Transcription Factors

2009
L-DOPA treatment from the viewpoint of neuroprotection. Possible mechanism of specific and progressive dopaminergic neuronal death in Parkinson's disease.
    Journal of neurology, 2005, Volume: 252 Suppl 4

    Topics: Animals; Cell Death; Dopamine; Humans; Levodopa; Mitochondrial Diseases; Neurons; Neuroprotective Agents; Oxidative Stress; Parkinsonian Disorders

2005

Other Studies

5 other study(ies) available for levodopa and Electron Transport Chain Deficiencies, Mitochondrial

ArticleYear
Late-onset presentation of POLG1-associated mitochondrial disease.
    BMJ case reports, 2019, Mar-31, Volume: 12, Issue:3

    Topics: Age of Onset; Aged; Antiparkinson Agents; Blepharoptosis; Disease Progression; DNA Polymerase gamma; Humans; Levodopa; Male; Mitochondrial Diseases; Ophthalmoplegia, Chronic Progressive External; Parkinsonian Disorders; Point Mutation; Treatment Outcome

2019
Endogenous Parkin Preserves Dopaminergic Substantia Nigral Neurons following Mitochondrial DNA Mutagenic Stress.
    Neuron, 2015, Jul-15, Volume: 87, Issue:2

    Topics: Analysis of Variance; Animals; DNA, Mitochondrial; Dopamine Plasma Membrane Transport Proteins; Dopaminergic Neurons; Gene Expression Regulation; Green Fluorescent Proteins; In Vitro Techniques; Levodopa; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mitochondrial Diseases; Multienzyme Complexes; Mutation; Proteomics; Substantia Nigra; Tyrosine 3-Monooxygenase; Ubiquitin; Ubiquitin-Protein Ligases

2015
Mitochondrial diseases mimicking neurotransmitter defects.
    Mitochondrion, 2008, Volume: 8, Issue:3

    Topics: Alanine; Brain; Child, Preschool; Consanguinity; Diagnosis, Differential; Dopamine Agents; Dystonia; Electron Transport Complex I; Electron Transport Complex II; Electron Transport Complex III; Female; Homovanillic Acid; Humans; Hydroxyindoleacetic Acid; Infant; Infant, Newborn; Lactic Acid; Levodopa; Male; Mitochondrial Diseases; Neurotransmitter Agents; Pterins; Radiography; Treatment Outcome

2008
The PINK1 phenotype can be indistinguishable from idiopathic Parkinson disease.
    Neurology, 2005, Jun-14, Volume: 64, Issue:11

    Topics: Age of Onset; Brain; Diagnosis, Differential; Dopamine Agents; Energy Metabolism; Genetic Predisposition to Disease; Humans; Italy; Levodopa; Male; Middle Aged; Mitochondria; Mitochondrial Diseases; Mutation; Parkinson Disease; Parkinsonian Disorders; Phenotype; Protein Kinases; Shy-Drager Syndrome; Sympathetic Nervous System; Tomography, Emission-Computed, Single-Photon; Treatment Outcome

2005
Parkinson syndrome as a manifestation of mitochondriopathy.
    Acta neurologica Scandinavica, 2002, Volume: 105, Issue:5

    Topics: Aged; Aged, 80 and over; Amantadine; Antiparkinson Agents; Biopsy; DNA, Mitochondrial; Dopamine; Electromyography; Female; Humans; Levodopa; Male; Middle Aged; Mitochondrial Diseases; Muscle, Skeletal; Oxidative Phosphorylation; Parkinsonian Disorders; Point Mutation; Severity of Illness Index

2002