leucine and Dementias, Transmissible

leucine has been researched along with Dementias, Transmissible in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (40.00)18.2507
2000's2 (40.00)29.6817
2010's1 (20.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Agarwal, S; Gill, AC; Goldmann, W; Graham, JF; Kirby, L1
Mizusawa, H; Shiraishi, A; Yamada, M1
Ghetti, B; Harris, DA; Piccardo, P; Stewart, RS1
Barrow, CJ; Beyreuther, K; Cappai, R; Collins, SJ; Jobling, MF; Masters, CL; Stewart, L; Thyer, JM; White, AR1
Inaba, A; Itoh, Y; Kamata, T; Kayano, T; Kitamoto, T; Matsushita, M; Mizusawa, H; Okeda, R; Otomo, E; Satoh, S; Suematsu, N; Takashima, M; Wada, Y; Yamada, M1

Reviews

1 review(s) available for leucine and Dementias, Transmissible

ArticleYear
An inherited prion disease with a PrP P105L mutation: clinicopathologic and PrP heterogeneity.
    Neurology, 1999, Jul-13, Volume: 53, Issue:1

    Topics: Amino Acid Substitution; Brain; Codon; Female; Humans; Leucine; Male; Methionine; Middle Aged; Mutation, Missense; Nuclear Family; Pedigree; Polymorphism, Genetic; Prion Diseases; Prions; Proline; Valine

1999

Other Studies

4 other study(ies) available for leucine and Dementias, Transmissible

ArticleYear
Inverse correlation of thermal lability and conversion efficiency for five prion protein polymorphic variants.
    Biochemistry, 2010, Feb-23, Volume: 49, Issue:7

    Topics: Amino Acid Substitution; Animals; Cell-Free System; Deer; Genetic Predisposition to Disease; Genetic Variation; Immunity, Innate; Leucine; Mice; Peptide Fragments; Polymorphism, Genetic; Prion Diseases; Prions; Proline; Protein Conformation; Protein Denaturation; Protein Folding; Protein Stability; Sheep; Temperature

2010
Early and persistent sensory-psychiatric symptoms in an inherited prion disease with a PrP P105L mutation.
    Journal of neurology, 2002, Volume: 249, Issue:12

    Topics: Female; Humans; Leucine; Male; Middle Aged; Mutation; Pedigree; Prion Diseases; Prions; Proline

2002
Neurodegenerative illness in transgenic mice expressing a transmembrane form of the prion protein.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2005, Mar-30, Volume: 25, Issue:13

    Topics: Animals; Animals, Newborn; Arginine; Blotting, Western; Brain; Cells, Cultured; Cerebellum; Cricetinae; Cricetulus; Detergents; Disease Models, Animal; Electrophoresis, Polyacrylamide Gel; Fluorescent Antibody Technique; Gene Expression; Glial Fibrillary Acidic Protein; Golgi Apparatus; Golgi Matrix Proteins; Immunoprecipitation; Leucine; Membrane Proteins; Methionine; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mutation; Neurodegenerative Diseases; Neurons; Octoxynol; Prion Diseases; Protein Structure, Tertiary; PrPSc Proteins; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Sulfur Isotopes; Time Factors; Type C Phospholipases; Valine

2005
Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formation.
    Biochemistry, 1999, Mar-16, Volume: 38, Issue:11

    Topics: Amino Acid Substitution; Animals; Circular Dichroism; Drug Resistance; Endopeptidase K; Escherichia coli; Leucine; Mice; Mice, Inbred BALB C; Prion Diseases; Prions; Proline; Protein Structure, Secondary; Recombinant Proteins

1999