leucine has been researched along with Abnormality, Heart in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (25.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cao, J; Chen, WC; Cheng, YR; Gui, YH; Lai, YF; Lin, Y; Liu, L; Liu, YM; Qiao, YN; Wang, F; Xu, W; Yuan, YY; Zhang, RJ; Zhang, X; Zhao, JY | 1 |
| Anzai, M; Aoki, Y; Endo, W; Haginoya, K; Inui, T; Kikuchi, A; Kure, S; Miyabayashi, T; Nagai, K; Niihori, T; Okubo, Y; Sato, R; Suzuki-Muromoto, S; Takezawa, Y; Togashi, N; Yamamura-Suzuki, S | 1 |
| Amark, K; Ekroth, R; Nilsson, K; Söderberg, B; Sunnegårdh, J | 1 |
| Doszpod, J; Fekete, M; Gáti, I; Járai, I; Mestyán, J; Preisz, J; Soltész, G | 1 |
4 other study(ies) available for leucine and Abnormality, Heart
| Article | Year |
|---|---|
Gestational Leucylation Suppresses Embryonic T-Box Transcription Factor 5 Signal and Causes Congenital Heart Disease.
Topics: Animals; Heart Defects, Congenital; Humans; Leucine; Lysine; Mice; Sirtuin 3; T-Box Domain Proteins | 2022 |
Leucine-485 deletion variant of BRAF may exhibit the severe end of the clinical spectrum of CFC syndrome.
Topics: Amino Acid Sequence; Child, Preschool; Ectodermal Dysplasia; Facies; Failure to Thrive; Heart Defects, Congenital; Humans; Leucine; Male; Proto-Oncogene Proteins B-raf; Sequence Deletion; Severity of Illness Index | 2019 |
Myocardial substrates in children with congenital heart disease: relationship to substrate supply, age, growth and desaturation.
Topics: 3-Hydroxybutyric Acid; Adolescent; Age Factors; Analysis of Variance; Blood Chemical Analysis; Blood Glucose; Cardiac Catheterization; Child; Child, Preschool; Coronary Vessels; Fatty Acids; Glutamic Acid; Growth and Development; Heart Defects, Congenital; Humans; Infant; Lactic Acid; Leucine; Myocardial Ischemia; Myocardium; Oxygen Consumption; Regression Analysis; Sweden | 2007 |
The postnatal changes in the circulating free amino acid pool in the newborn infant. 3. The plasma amino acid ratio in infants born after pregnancies complicated by toxaemia, placental infarction, impaired umbilical circulation and chronic maternal diseas
Topics: Amino Acids; Chronic Disease; Female; Glutamine; Glycine; Heart Defects, Congenital; Heart Failure; Humans; Infant, Newborn; Infarction; Isoleucine; Leucine; Maternal-Fetal Exchange; Methionine; Placenta; Pre-Eclampsia; Pregnancy; Pregnancy Complications, Cardiovascular; Pyelonephritis; Serine; Taurine; Torsion Abnormality; Umbilical Cord; Valine | 1971 |