Page last updated: 2024-09-02

kt 5823 and Marfan Syndrome, Type I

kt 5823 has been researched along with Marfan Syndrome, Type I in 1 studies

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's1 (100.00)2.80

Authors

AuthorsStudies
Alfayate, A; Bonzón-Kulichenko, E; Campanero, MR; De Backer, J; de la Fuente-Alonso, A; Evangelista, A; Forteza, A; Garcia-Izquierdo, E; González-Valdés, I; López-Maderuelo, D; Martín, CE; Martínez-Martínez, S; Méndez-Olivares, MJ; Mingo, S; Muiño-Mosquera, L; Nistal, JF; Redondo, JM; Ruiz-Rodríguez, MJ; Teixido-Tura, G; Toral, M; Vázquez, J1

Other Studies

1 other study(ies) available for kt 5823 and Marfan Syndrome, Type I

ArticleYear
Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO.
    Nature communications, 2021, 05-11, Volume: 12, Issue:1

    Topics: Animals; Aorta; Aortic Aneurysm, Thoracic; Biomarkers; Carbazoles; Cyclic GMP; Cyclic GMP-Dependent Protein Kinase Type I; Disease Models, Animal; Female; Fibrillin-1; Gene Knockdown Techniques; Humans; Male; Marfan Syndrome; Mice; Muscle, Smooth, Vascular; Mutation; Myocytes, Smooth Muscle; Nitric Oxide; Nitric Oxide Donors; Primary Cell Culture; Soluble Guanylyl Cyclase; Ultrasonography

2021