Page last updated: 2024-09-02

kt 5823 and Disease Models, Animal

kt 5823 has been researched along with Disease Models, Animal in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (28.57)29.6817
2010's4 (57.14)24.3611
2020's1 (14.29)2.80

Authors

AuthorsStudies
Alfayate, A; Bonzón-Kulichenko, E; Campanero, MR; De Backer, J; de la Fuente-Alonso, A; Evangelista, A; Forteza, A; Garcia-Izquierdo, E; González-Valdés, I; López-Maderuelo, D; Martín, CE; Martínez-Martínez, S; Méndez-Olivares, MJ; Mingo, S; Muiño-Mosquera, L; Nistal, JF; Redondo, JM; Ruiz-Rodríguez, MJ; Teixido-Tura, G; Toral, M; Vázquez, J1
Gu, J; Lin, P; Lin, Z; Lu, S; Luo, J; Ma, Y; Tian, X; Wang, W; Wang, X; Xiao, F; Xiong, Y; Xu, D; Yang, Q; Yang, Y; Zhang, Y1
Andersen, A; Bøtker, HE; Jespersen, NR; Johnsen, J; Nielsen-Kudsk, JE; Povlsen, JA1
Asada, Y; Hatakeyama, K; Imamura, T; Kitamura, K; Masuyama, H; Sekita, Y; Tsuruda, T1
Scholz, PM; Tan, T; Weiss, HR1
Bao, L; Gong, LQ; Li, KC; Li, ZY; Lin, LB; Liu, XJ; Lu, YJ; Xiao, HS; Yao, JR; Zhang, FX; Zhang, X; Zhang, XH1
Davidov, T; Grover, GJ; Moalem, J; Scholz, PM; Weiss, HR; Zhang, Q1

Other Studies

7 other study(ies) available for kt 5823 and Disease Models, Animal

ArticleYear
Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO.
    Nature communications, 2021, 05-11, Volume: 12, Issue:1

    Topics: Animals; Aorta; Aortic Aneurysm, Thoracic; Biomarkers; Carbazoles; Cyclic GMP; Cyclic GMP-Dependent Protein Kinase Type I; Disease Models, Animal; Female; Fibrillin-1; Gene Knockdown Techniques; Humans; Male; Marfan Syndrome; Mice; Muscle, Smooth, Vascular; Mutation; Myocytes, Smooth Muscle; Nitric Oxide; Nitric Oxide Donors; Primary Cell Culture; Soluble Guanylyl Cyclase; Ultrasonography

2021
Inhibition of Cgkii Suppresses Seizure Activity and Hippocampal Excitation by Regulating the Postsynaptic Delivery of Glua1.
    Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2018, Volume: 46, Issue:1

    Topics: 4-Aminopyridine; Adolescent; Adult; Animals; Brain; Carbazoles; Child; Cyclic GMP; Cyclic GMP-Dependent Protein Kinase Type II; Disease Models, Animal; Epilepsy; Evoked Potentials; Female; Hippocampus; Humans; Male; Pilocarpine; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Synaptic Transmission; Thionucleotides; Up-Regulation; Young Adult

2018
sGC-cGMP-PKG pathway stimulation protects the healthy but not the failing right ventricle of rats against ischemia and reperfusion injury.
    International journal of cardiology, 2016, Nov-15, Volume: 223

    Topics: Animals; Carbazoles; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Heart Failure; Heart Ventricles; Male; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Organ Culture Techniques; Protein Kinase Inhibitors; Rats; Rats, Wistar; Signal Transduction

2016
Pharmacological stimulation of soluble guanylate cyclase modulates hypoxia-inducible factor-1alpha in rat heart.
    American journal of physiology. Heart and circulatory physiology, 2009, Volume: 297, Issue:4

    Topics: Animals; Animals, Newborn; Carbazoles; Cardiomegaly; Cell Hypoxia; Cells, Cultured; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Down-Regulation; Enzyme Activation; Enzyme Activators; Guanylate Cyclase; Hypertension; Hypoxia-Inducible Factor 1, alpha Subunit; Male; Myocytes, Cardiac; Neovascularization, Physiologic; Protein Kinase Inhibitors; Pyrazoles; Pyridines; Rats; Rats, Wistar; Receptors, Cytoplasmic and Nuclear; Second Messenger Systems; Soluble Guanylyl Cyclase; Time Factors; Vascular Endothelial Growth Factor A; Ventricular Remodeling

2009
Hypoxia inducible factor-1 improves the negative functional effects of natriuretic peptide and nitric oxide signaling in hypertrophic cardiac myocytes.
    Life sciences, 2010, Jul-03, Volume: 87, Issue:1-2

    Topics: Animals; Carbazoles; Cardiomegaly; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Deferoxamine; Disease Models, Animal; Dose-Response Relationship, Drug; Female; Hypoxia-Inducible Factor 1; Hypoxia-Inducible Factor 1, alpha Subunit; Male; Mice; Mice, Inbred C57BL; Myocytes, Cardiac; Natriuretic Peptide, Brain; Natriuretic Peptide, C-Type; Nitric Oxide; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; Up-Regulation

2010
Inhibition of inflammatory pain by activating B-type natriuretic peptide signal pathway in nociceptive sensory neurons.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2010, Aug-11, Volume: 30, Issue:32

    Topics: Analysis of Variance; Animals; Antibodies; Biophysical Phenomena; Calcitonin Gene-Related Peptide; Carbazoles; Disease Models, Animal; Dose-Response Relationship, Drug; Double-Blind Method; Enzyme Inhibitors; Excitatory Postsynaptic Potentials; Freund's Adjuvant; Ganglia, Spinal; Gene Expression Regulation; Glutamic Acid; Hyperalgesia; Inflammation; Lectins; Male; Membrane Potentials; Natriuretic Peptide, Brain; Pain; Pain Measurement; Patch-Clamp Techniques; Peptides; Rats; Rats, Sprague-Dawley; Receptors, Atrial Natriuretic Factor; Sensory Receptor Cells; Signal Transduction; Time Factors

2010
Negative inotropic effects of C-type natriuretic peptide are attenuated in hypertrophied ventricular myocytes associated with reduced cyclic GMP production.
    The Journal of surgical research, 2006, Volume: 135, Issue:1

    Topics: Animals; Carbazoles; Cardiomegaly; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Guanylate Cyclase; Heart Ventricles; Hypertension, Renal; Hypertrophy; Indoles; Myocardial Contraction; Myocytes, Cardiac; Natriuretic Peptide, C-Type; Protein Kinase Inhibitors; Rabbits; Signal Transduction; Surgical Instruments

2006