kt 5823 has been researched along with Disease Models, Animal in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (28.57) | 29.6817 |
2010's | 4 (57.14) | 24.3611 |
2020's | 1 (14.29) | 2.80 |
Authors | Studies |
---|---|
Alfayate, A; Bonzón-Kulichenko, E; Campanero, MR; De Backer, J; de la Fuente-Alonso, A; Evangelista, A; Forteza, A; Garcia-Izquierdo, E; González-Valdés, I; López-Maderuelo, D; Martín, CE; Martínez-Martínez, S; Méndez-Olivares, MJ; Mingo, S; Muiño-Mosquera, L; Nistal, JF; Redondo, JM; Ruiz-Rodríguez, MJ; Teixido-Tura, G; Toral, M; Vázquez, J | 1 |
Gu, J; Lin, P; Lin, Z; Lu, S; Luo, J; Ma, Y; Tian, X; Wang, W; Wang, X; Xiao, F; Xiong, Y; Xu, D; Yang, Q; Yang, Y; Zhang, Y | 1 |
Andersen, A; Bøtker, HE; Jespersen, NR; Johnsen, J; Nielsen-Kudsk, JE; Povlsen, JA | 1 |
Asada, Y; Hatakeyama, K; Imamura, T; Kitamura, K; Masuyama, H; Sekita, Y; Tsuruda, T | 1 |
Scholz, PM; Tan, T; Weiss, HR | 1 |
Bao, L; Gong, LQ; Li, KC; Li, ZY; Lin, LB; Liu, XJ; Lu, YJ; Xiao, HS; Yao, JR; Zhang, FX; Zhang, X; Zhang, XH | 1 |
Davidov, T; Grover, GJ; Moalem, J; Scholz, PM; Weiss, HR; Zhang, Q | 1 |
7 other study(ies) available for kt 5823 and Disease Models, Animal
Article | Year |
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Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO.
Topics: Animals; Aorta; Aortic Aneurysm, Thoracic; Biomarkers; Carbazoles; Cyclic GMP; Cyclic GMP-Dependent Protein Kinase Type I; Disease Models, Animal; Female; Fibrillin-1; Gene Knockdown Techniques; Humans; Male; Marfan Syndrome; Mice; Muscle, Smooth, Vascular; Mutation; Myocytes, Smooth Muscle; Nitric Oxide; Nitric Oxide Donors; Primary Cell Culture; Soluble Guanylyl Cyclase; Ultrasonography | 2021 |
Inhibition of Cgkii Suppresses Seizure Activity and Hippocampal Excitation by Regulating the Postsynaptic Delivery of Glua1.
Topics: 4-Aminopyridine; Adolescent; Adult; Animals; Brain; Carbazoles; Child; Cyclic GMP; Cyclic GMP-Dependent Protein Kinase Type II; Disease Models, Animal; Epilepsy; Evoked Potentials; Female; Hippocampus; Humans; Male; Pilocarpine; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Synaptic Transmission; Thionucleotides; Up-Regulation; Young Adult | 2018 |
sGC-cGMP-PKG pathway stimulation protects the healthy but not the failing right ventricle of rats against ischemia and reperfusion injury.
Topics: Animals; Carbazoles; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Heart Failure; Heart Ventricles; Male; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Organ Culture Techniques; Protein Kinase Inhibitors; Rats; Rats, Wistar; Signal Transduction | 2016 |
Pharmacological stimulation of soluble guanylate cyclase modulates hypoxia-inducible factor-1alpha in rat heart.
Topics: Animals; Animals, Newborn; Carbazoles; Cardiomegaly; Cell Hypoxia; Cells, Cultured; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Down-Regulation; Enzyme Activation; Enzyme Activators; Guanylate Cyclase; Hypertension; Hypoxia-Inducible Factor 1, alpha Subunit; Male; Myocytes, Cardiac; Neovascularization, Physiologic; Protein Kinase Inhibitors; Pyrazoles; Pyridines; Rats; Rats, Wistar; Receptors, Cytoplasmic and Nuclear; Second Messenger Systems; Soluble Guanylyl Cyclase; Time Factors; Vascular Endothelial Growth Factor A; Ventricular Remodeling | 2009 |
Hypoxia inducible factor-1 improves the negative functional effects of natriuretic peptide and nitric oxide signaling in hypertrophic cardiac myocytes.
Topics: Animals; Carbazoles; Cardiomegaly; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Deferoxamine; Disease Models, Animal; Dose-Response Relationship, Drug; Female; Hypoxia-Inducible Factor 1; Hypoxia-Inducible Factor 1, alpha Subunit; Male; Mice; Mice, Inbred C57BL; Myocytes, Cardiac; Natriuretic Peptide, Brain; Natriuretic Peptide, C-Type; Nitric Oxide; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; Up-Regulation | 2010 |
Inhibition of inflammatory pain by activating B-type natriuretic peptide signal pathway in nociceptive sensory neurons.
Topics: Analysis of Variance; Animals; Antibodies; Biophysical Phenomena; Calcitonin Gene-Related Peptide; Carbazoles; Disease Models, Animal; Dose-Response Relationship, Drug; Double-Blind Method; Enzyme Inhibitors; Excitatory Postsynaptic Potentials; Freund's Adjuvant; Ganglia, Spinal; Gene Expression Regulation; Glutamic Acid; Hyperalgesia; Inflammation; Lectins; Male; Membrane Potentials; Natriuretic Peptide, Brain; Pain; Pain Measurement; Patch-Clamp Techniques; Peptides; Rats; Rats, Sprague-Dawley; Receptors, Atrial Natriuretic Factor; Sensory Receptor Cells; Signal Transduction; Time Factors | 2010 |
Negative inotropic effects of C-type natriuretic peptide are attenuated in hypertrophied ventricular myocytes associated with reduced cyclic GMP production.
Topics: Animals; Carbazoles; Cardiomegaly; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Disease Models, Animal; Guanylate Cyclase; Heart Ventricles; Hypertension, Renal; Hypertrophy; Indoles; Myocardial Contraction; Myocytes, Cardiac; Natriuretic Peptide, C-Type; Protein Kinase Inhibitors; Rabbits; Signal Transduction; Surgical Instruments | 2006 |