Page last updated: 2024-10-30

kojic acid and Hyperplasia

kojic acid has been researched along with Hyperplasia in 4 studies

Hyperplasia: An increase in the number of cells in a tissue or organ without tumor formation. It differs from HYPERTROPHY, which is an increase in bulk without an increase in the number of cells.

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's3 (75.00)18.2507
2000's1 (25.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Tamura, T4
Mitsumori, K4
Totsuka, Y1
Wakabayashi, K1
Kido, R1
Kasai, H1
Nasu, M1
Hirose, M2
Onodera, H3
Takahashi, M3
Funakoshi, T2
Yasuhara, K2
Takegawa, K2
Fujimoto, N1
Maruyama, S1
Ito, A1
Takagi, H1

Other Studies

4 other studies available for kojic acid and Hyperplasia

ArticleYear
Absence of in vivo genotoxic potential and tumor initiation activity of kojic acid in the rat thyroid.
    Toxicology, 2006, May-15, Volume: 222, Issue:3

    Topics: 8-Hydroxy-2'-Deoxyguanosine; Adenoma; Animals; Carcinogens; Carcinoma; Deoxyguanosine; DNA Adducts;

2006
Promoting effects of kojic acid due to serum TSH elevation resulting from reduced serum thyroid hormone levels on development of thyroid proliferative lesions in rats initiated with N-bis(2-hydroxypropyl)nitrosamine.
    Carcinogenesis, 1999, Volume: 20, Issue:1

    Topics: Adenoma; Animals; Biotransformation; Carcinogens; Feedback; Food Additives; Glucuronosyltransferase;

1999
Changes in thyroid function during development of thyroid hyperplasia induced by kojic acid in F344 rats.
    Carcinogenesis, 1999, Volume: 20, Issue:8

    Topics: Animals; Dose-Response Relationship, Drug; Female; Food Additives; Hyperplasia; Iodine Radioisotopes

1999
Time course observation of thyroid proliferative lesions and serum levels of related hormones in rats treated with kojic acid after DHPN initiation.
    The Journal of toxicological sciences, 1999, Volume: 24, Issue:3

    Topics: Adenoma; Animals; Body Weight; Cocarcinogenesis; Glucuronosyltransferase; Hyperplasia; Liver; Male;

1999