khellin has been researched along with Genetic Predisposition in 49 studies
Khellin: A vasodilator that also has bronchodilatory action. It has been employed in the treatment of angina pectoris, in the treatment of asthma, and in conjunction with ultraviolet light A, has been tried in the treatment of vitiligo. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1024)
khellin : A furanochrome in which the basic tricyclic skeleton is substituted at positions 4 and 9 with methoxy groups and at position 7 with a methyl group. A major constituent of the plant Ammi visnaga it is a herbal folk medicine used for various illnesses, its main effect being as a vasodilator.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Transforming growth factor beta1 (TGF-beta1) plays a central role in wound healing and fibrosis and has been implicated in the pathogenesis of keloid disease and hypertrophic scar." | 5.32 | Genetic susceptibility to keloid disease and hypertrophic scarring: transforming growth factor beta1 common polymorphisms and plasma levels. ( Bayat, A; Bock, O; Ferguson, MW; Mrowietz, U; Ollier, WE, 2003) |
| "Transforming growth factor beta (TGF beta) is a family of multifunctional cytokines, which play a central role in wound healing and fibrosis." | 5.31 | Genetic susceptibility to keloid disease and transforming growth factor beta 2 polymorphisms. ( Bayat, A; Bock, O; Ferguson, MW; Mrowietz, U; Ollier, WE, 2002) |
| "Keloids are an exuberant response to cutaneous wound healing." | 2.55 | Current Understanding of the Genetic Causes of Keloid Formation. ( Glass, DA, 2017) |
| "Although there is evidence suggesting genetic susceptibility for keloids, studies investigating the association between Arg72Pro polymorphism in the P53 gene and tendency to form keloids have given variable results." | 2.48 | Meta-analysis demonstrates association between Arg72Pro polymorphism in the P53 gene and susceptibility to keloids in the Chinese population. ( Chen, HD; Chen, JZ; Gao, XH; Li, YH; Luo, YJ; Wang, B; Wei, HC; Wu, Y; Xu, XG, 2012) |
| "There is strong evidence suggesting a genetic susceptibility in individuals affected by KD, including familial heritability, common occurrence in twins and high prevalence in certain ethnic populations." | 2.46 | Genetics of keloid scarring. ( Bayat, A; Shih, B, 2010) |
| "While, fetal wounds heal without scarring, a normal "fine line" scar is the clinical outcome of an undisturbed wound healing in adults." | 2.45 | Keloid scarring: bench and bedside. ( Mrowietz, U; Seifert, O, 2009) |
| "Keloid and hypertrophic scars are multifaceted aberrations of the healing process with as yet incompletely understood aetiologies." | 2.45 | Genetic susceptibility to raised dermal scarring. ( Bayat, A; Brown, JJ, 2009) |
| "BACKGROUND A keloid is a pathological scar hyperplasia that is affected by genetic and environmental factors." | 1.72 | Expression of the GZMB Gene Polymorphism, SNP rs8192917, in 990 Han Chinese Patients with Postoperative Keloids. ( Du, H; Guo, Y; Hao, X; Wang, J; Wen, X, 2022) |
| "The formation of keloid is associated with accumulation of extracellular matrix (ECM) formed mainly of collagen and fibronectin." | 1.51 | Interleukin-6 serum level and gene polymorphism in keloid patients. ( Abdu Allah, AMK; Essam, M; Farag, AGA; Hagag, MM; Mohammed, KI; Tayel, NR, 2019) |
| "Keloids are benign dermal tumors occurring approximately 20 times more often in individuals of African descent as compared to individuals of European descent." | 1.48 | Gene-based evaluation of low-frequency variation and genetically-predicted gene expression impacting risk of keloid formation. ( Edwards, TL; Hellwege, JN; Russell, SB; Velez Edwards, DR; Williams, SM, 2018) |
| "There is strong evidence of genetic susceptibility in individuals with keloid disorder." | 1.42 | Clinical implications of single- versus multiple-site keloid disorder: a retrospective study in an Asian population. ( Chang, CH; Halim, AS; Park, JH; Park, TH; Tirgan, MH, 2015) |
| "Keloids are benign dermal tumors that occur ~20 times more often in African versus Caucasian descent individuals." | 1.40 | Admixture mapping identifies a locus at 15q21.2-22.3 associated with keloid formation in African Americans. ( Edwards, TL; Russell, SB; Tsosie, KS; Velez Edwards, DR; Williams, SM, 2014) |
| "Keloid is benign fibroproliferative dermal tumors with unknown etiology." | 1.39 | Association study confirmed susceptibility loci with keloid in the Chinese Han population. ( Cheng, H; Ding, Y; Gao, J; Ha, W; Hou, J; Li, P; Liu, J; Liu, Y; Qian, F; Sheng, Y; Sun, L; Tang, H; Wang, J; Wang, W; Wang, Y; Wu, B; Yang, S; Yuan, X; Zhai, Y; Zhang, X; Zhou, F; Zhu, F; Zuo, X, 2013) |
| "Keloid is a complex condition with environmental and genetic risk-contributing factors." | 1.38 | Association of TGFβ1 and SMAD4 variants in the etiology of keloid scar in the Malay population. ( Emami, A; Halim, AS; Kannan, TP; Khoo, TL; Salahshourifar, I; Yussof, SJ, 2012) |
| "Keloids are common abnormal raised fibroproliferative lesions that can occur following even minor cutaneous trauma." | 1.37 | Association of HLA haplotype with keloids in Chinese Hans. ( Cai, LQ; Li, Y; Lu, WS; Sun, LD; Wang, ZX; Yang, S; Zhang, XJ; Zheng, HF; Zhu, F; Zuo, XB, 2011) |
| "Keloids are benign fibroproliferative dermal tumors of unknown etiology." | 1.36 | Association of HLA class I alleles with keloids in Chinese Han individuals. ( Cai, LQ; He, SM; Li, Y; Lu, WS; Quan, C; Wang, JF; Wang, ZX; Xiao, FL; Yang, S; Zhang, XJ, 2010) |
| "Keloid is a dermal fibroproliferative growth that results from dysfunction of the wound healing processes." | 1.36 | A genome-wide association study identifies four susceptibility loci for keloid in the Japanese population. ( Chung, S; Hosono, N; Kamatani, N; Kawaguchi, T; Kubo, M; Nakamura, Y; Nakashima, M; Takahashi, A; Tsunoda, T; Zembutsu, H, 2010) |
| "Excessive scar formation in keloids points to altered tissue modeling and repair mechanisms." | 1.35 | Gene expression profiling reveals alteration of caspase 6 and 14 transcripts in normal skin of keloid-prone patients. ( Berman, B; Elgart, G; Gugic, D; Jacob, SE; Nassiri, M; Ramos, S; Romanelli, P; Viciana, A; Vincek, V; Woolery-Lloyd, H, 2009) |
| "Transforming growth factor beta1 (TGF-beta1) plays a central role in wound healing and fibrosis and has been implicated in the pathogenesis of keloid disease and hypertrophic scar." | 1.32 | Genetic susceptibility to keloid disease and hypertrophic scarring: transforming growth factor beta1 common polymorphisms and plasma levels. ( Bayat, A; Bock, O; Ferguson, MW; Mrowietz, U; Ollier, WE, 2003) |
| "Keloids are proliferative fibrous growths that result from an excessive tissue response to skin trauma." | 1.32 | Genome scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11. ( Marneros, AG; Norris, JE; Olsen, BR; Reichenberger, E; Watanabe, S, 2004) |
| "Transforming growth factor beta (TGF beta) is a family of multifunctional cytokines, which play a central role in wound healing and fibrosis." | 1.31 | Genetic susceptibility to keloid disease and transforming growth factor beta 2 polymorphisms. ( Bayat, A; Bock, O; Ferguson, MW; Mrowietz, U; Ollier, WE, 2002) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (2.04) | 18.2507 |
| 2000's | 21 (42.86) | 29.6817 |
| 2010's | 22 (44.90) | 24.3611 |
| 2020's | 5 (10.20) | 2.80 |
| Authors | Studies |
|---|---|
| Li, Y | 4 |
| Li, M | 1 |
| Qu, C | 1 |
| Tang, Z | 1 |
| Zhou, Z | 1 |
| Yu, Z | 1 |
| Wang, X | 1 |
| Xin, L | 1 |
| Shi, T | 1 |
| Wen, X | 1 |
| Du, H | 1 |
| Hao, X | 1 |
| Wang, J | 3 |
| Guo, Y | 1 |
| Liu, S | 1 |
| Yang, H | 1 |
| Song, J | 1 |
| Zhang, Y | 1 |
| Abualhssain, ATH | 1 |
| Yang, B | 1 |
| Liu, J | 2 |
| Cai, L | 1 |
| Zhang, Z | 1 |
| Ma, Y | 1 |
| Wang, Y | 3 |
| Wade, EM | 1 |
| Jenkins, ZA | 1 |
| Morgan, T | 1 |
| Gimenez, G | 1 |
| Gibson, H | 1 |
| Peng, H | 1 |
| Sanchez Russo, R | 1 |
| Skraban, CM | 1 |
| Bedoukian, E | 1 |
| Robertson, SP | 1 |
| Glass, DA | 1 |
| Hellwege, JN | 1 |
| Russell, SB | 2 |
| Williams, SM | 2 |
| Edwards, TL | 2 |
| Velez Edwards, DR | 2 |
| Abdu Allah, AMK | 1 |
| Mohammed, KI | 1 |
| Farag, AGA | 1 |
| Hagag, MM | 1 |
| Essam, M | 1 |
| Tayel, NR | 1 |
| Zhu, F | 2 |
| Wu, B | 1 |
| Li, P | 1 |
| Tang, H | 1 |
| Liu, Y | 2 |
| Zuo, X | 1 |
| Cheng, H | 2 |
| Ding, Y | 1 |
| Wang, W | 2 |
| Zhai, Y | 1 |
| Qian, F | 1 |
| Yuan, X | 1 |
| Ha, W | 1 |
| Hou, J | 1 |
| Zhou, F | 1 |
| Gao, J | 2 |
| Sheng, Y | 1 |
| Sun, L | 1 |
| Yang, S | 4 |
| Zhang, X | 2 |
| Park, TH | 1 |
| Park, JH | 1 |
| Tirgan, MH | 1 |
| Halim, AS | 2 |
| Chang, CH | 1 |
| Tsosie, KS | 1 |
| Tosa, M | 1 |
| Watanabe, A | 1 |
| Ghazizadeh, M | 1 |
| Zhang, M | 1 |
| Xu, Y | 1 |
| Cheng, Y | 1 |
| Zhao, P | 1 |
| Liu, H | 1 |
| Ma, X | 1 |
| A Linjawi, S | 1 |
| E Tork, S | 1 |
| M Shaibah, R | 1 |
| Gong, ZH | 1 |
| Ji, JF | 1 |
| Yang, J | 1 |
| Xiang, T | 1 |
| Zhou, CK | 1 |
| Pan, XL | 1 |
| Yao, J | 1 |
| Lu, WS | 3 |
| Wang, JF | 2 |
| Xiao, FL | 2 |
| Quan, C | 2 |
| Wang, PG | 1 |
| Zhang, AP | 1 |
| Cai, LQ | 3 |
| Zhang, XJ | 3 |
| Huang, ZX | 1 |
| Tian, HY | 1 |
| Hu, ZF | 1 |
| Zhou, YB | 1 |
| Zhao, J | 1 |
| Yao, KT | 1 |
| Nassiri, M | 1 |
| Woolery-Lloyd, H | 1 |
| Ramos, S | 1 |
| Jacob, SE | 1 |
| Gugic, D | 1 |
| Viciana, A | 1 |
| Romanelli, P | 1 |
| Elgart, G | 1 |
| Berman, B | 1 |
| Vincek, V | 1 |
| Brown, JJ | 4 |
| Ollier, WE | 6 |
| Thomson, W | 1 |
| Bayat, A | 11 |
| Seifert, O | 1 |
| Mrowietz, U | 5 |
| Arscott, G | 3 |
| Wang, ZX | 2 |
| He, SM | 1 |
| Shih, B | 2 |
| Nakashima, M | 1 |
| Chung, S | 1 |
| Takahashi, A | 1 |
| Kamatani, N | 1 |
| Kawaguchi, T | 1 |
| Tsunoda, T | 1 |
| Hosono, N | 1 |
| Kubo, M | 1 |
| Nakamura, Y | 1 |
| Zembutsu, H | 1 |
| Zuo, XB | 1 |
| Zheng, HF | 1 |
| Sun, LD | 1 |
| Ashcroft, KJ | 1 |
| Syed, F | 1 |
| Huang, C | 1 |
| Nie, F | 1 |
| Qin, Z | 1 |
| Li, B | 1 |
| Zhao, X | 1 |
| Emami, A | 1 |
| Salahshourifar, I | 1 |
| Yussof, SJ | 1 |
| Khoo, TL | 1 |
| Kannan, TP | 1 |
| Wu, Y | 1 |
| Wang, B | 1 |
| Li, YH | 1 |
| Xu, XG | 1 |
| Luo, YJ | 1 |
| Chen, JZ | 1 |
| Wei, HC | 1 |
| Gao, XH | 1 |
| Chen, HD | 1 |
| Pastuszak-Gabinowska, M | 1 |
| Peregud-Pogorzelski, J | 1 |
| Łuksza, K | 1 |
| Sznelewski, P | 1 |
| Brodkiewicz, A | 1 |
| Bock, O | 4 |
| Ferguson, MW | 4 |
| Marneros, AG | 1 |
| Norris, JE | 1 |
| Watanabe, S | 1 |
| Reichenberger, E | 1 |
| Olsen, BR | 1 |
| Zhuo, Y | 2 |
| Luo, S | 1 |
| Zeng, W | 1 |
| Hu, Z | 1 |
| Lu, F | 2 |
| Zhao, Y | 1 |
| Wang, CM | 1 |
| Hiko, H | 1 |
| Nakazawa, N | 1 |
| Archer, HL | 1 |
| Gupta, S | 1 |
| Enoch, S | 1 |
| Thompson, P | 1 |
| Rowbottom, A | 1 |
| Chua, I | 1 |
| Warren, S | 1 |
| Johnson, D | 1 |
| Ledbetter, DH | 1 |
| Lese-Martin, C | 1 |
| Williams, P | 1 |
| Pilz, DT | 1 |
| Walter, JM | 1 |
| Gao, JH | 4 |
| Luo, SQ | 1 |
| Zeng, WS | 1 |
| Hu, ZQ | 1 |
| Zhao, YZ | 1 |
| Burd, A | 1 |
| Chen, Y | 3 |
| Liu, XJ | 3 |
| Yan, X | 3 |
| Song, M | 3 |
| Odunze, M | 1 |
| Cohn, A | 1 |
| Few, JW | 1 |
| Ollier, W | 1 |
| Ke, X | 1 |
| Lamb, J | 1 |
| Day, P | 1 |
| Deodhar, AK | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Identification of Genetic Variants That Contribute to Keloid Formation in Families and Isolated Cases.[NCT01619553] | 7,000 participants (Anticipated) | Observational | 2009-04-30 | Recruiting | |||
| Pilot Study to Evaluate Clinical Outcomes With the Use of Biovance, a Dehydrated Decellularized Human Amnion Membrane Allograft, Following Keloid Revision Surgery[NCT02521402] | Phase 4 | 10 participants (Anticipated) | Interventional | 2015-08-31 | Not yet recruiting | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
11 reviews available for khellin and Genetic Predisposition
| Article | Year |
|---|---|
Keloid: Genetic susceptibility and contributions of genetics and epigenetics to its pathogenesis.
Topics: DNA Methylation; Epigenesis, Genetic; Female; Genetic Predisposition to Disease; Humans; Keloid; Mal | 2022 |
Current Understanding of the Genetic Causes of Keloid Formation.
Topics: Asian People; Black People; Epigenesis, Genetic; Genetic Loci; Genetic Predisposition to Disease; HL | 2017 |
Keloid scarring: bench and bedside.
Topics: Adult; Aminoquinolines; Cicatrix; Female; Genetic Predisposition to Disease; Humans; Imiquimod; Immu | 2009 |
Genetic susceptibility to raised dermal scarring.
Topics: Cicatrix, Hypertrophic; Genetic Linkage; Genetic Predisposition to Disease; HLA-D Antigens; Humans; | 2009 |
Genetics of keloid scarring.
Topics: Apoptosis; Cicatrix; Clinical Trials as Topic; Genetic Predisposition to Disease; Genome-Wide Associ | 2010 |
Genome-wide association study of skin complex diseases.
Topics: Alopecia; Carcinoma, Basal Cell; Dermatitis, Atopic; Female; Genetic Predisposition to Disease; Geno | 2012 |
A snapshot of gene expression signatures generated using microarray datasets associated with excessive scarring.
Topics: Cicatrix, Hypertrophic; Computational Biology; Gene Expression Profiling; Gene Expression Regulation | 2013 |
Meta-analysis demonstrates association between Arg72Pro polymorphism in the P53 gene and susceptibility to keloids in the Chinese population.
Topics: Amino Acid Substitution; Asian People; Case-Control Studies; Genetic Association Studies; Genetic Pr | 2012 |
[Some aspects of molecular bases of keloid formation].
Topics: Apoptosis; Cytokines; Genetic Predisposition to Disease; Humans; Keloid; Mutation; Polymorphism, Gen | 2011 |
Distinct phenotype associated with a cryptic subtelomeric deletion of 19p13.3-pter.
Topics: Abnormalities, Multiple; Adolescent; Chromosome Deletion; Chromosomes, Human, Pair 19; Cleft Palate; | 2005 |
Aetiopathogenesis of keloids.
Topics: Age Factors; Collagen; Fibroblasts; Genetic Predisposition to Disease; Humans; Hypersensitivity, Del | 1999 |
38 other studies available for khellin and Genetic Predisposition
| Article | Year |
|---|---|
The Polygenic Map of Keloid Fibroblasts Reveals Fibrosis-Associated Gene Alterations in Inflammation and Immune Responses.
Topics: Computational Biology; Disease Susceptibility; Fibroblasts; Fibrosis; Gene Expression Profiling; Gen | 2021 |
Expression of the GZMB Gene Polymorphism, SNP rs8192917, in 990 Han Chinese Patients with Postoperative Keloids.
Topics: Alleles; Case-Control Studies; China; Gene Frequency; Genetic Association Studies; Genetic Predispos | 2022 |
Association of Leptin Receptor Gene Polymorphisms with Keloids in the Chinese Han Population.
Topics: Adult; Asian People; Case-Control Studies; China; Cohort Studies; Ethnicity; Female; Gene Frequency; | 2021 |
Exon skip-inducing variants in FLNA in an attenuated form of frontometaphyseal dysplasia.
Topics: Child; Cicatrix; Exons; Filamins; Forehead; Genes, X-Linked; Genetic Diseases, X-Linked; Genetic Pre | 2021 |
Gene-based evaluation of low-frequency variation and genetically-predicted gene expression impacting risk of keloid formation.
Topics: Adult; Aged; Black or African American; Case-Control Studies; Exome; Female; Gene Flow; Genetic Asso | 2018 |
Interleukin-6 serum level and gene polymorphism in keloid patients.
Topics: Adolescent; Adult; Case-Control Studies; Egypt; Female; Gene Frequency; Genetic Predisposition to Di | 2019 |
Association study confirmed susceptibility loci with keloid in the Chinese Han population.
Topics: Adult; Asian People; Ethnicity; Female; Genetic Loci; Genetic Predisposition to Disease; Haplotypes; | 2013 |
Clinical implications of single- versus multiple-site keloid disorder: a retrospective study in an Asian population.
Topics: Adolescent; Adult; Asian People; Cross-Sectional Studies; Female; Genetic Predisposition to Disease; | 2015 |
Admixture mapping identifies a locus at 15q21.2-22.3 associated with keloid formation in African Americans.
Topics: Black or African American; Case-Control Studies; Chromosome Mapping; Chromosomes, Human, Pair 15; Ex | 2014 |
IL-6 Polymorphism and Susceptibility to Keloid Formation in a Japanese Population.
Topics: Asian People; Case-Control Studies; Confidence Intervals; Female; Genetic Predisposition to Disease; | 2016 |
Chemokine-Like Factor 1 (CKLF-1) is Overexpressed in Keloid Patients: A Potential Indicating Factor for Keloid-Predisposed Individuals.
Topics: Adolescent; Adult; Case-Control Studies; Chemokines; Cicatrix; Female; Genetic Predisposition to Dis | 2016 |
Genetic association of the COL1A1 gene promoter -1997 G/T (rs1107946) andSp1 +1245 G/T (rs1800012) polymorphisms and keloid scars in a Jeddah population.
Topics: Case-Control Studies; Collagen Type I; Collagen Type I, alpha 1 Chain; Female; Gene Frequency; Genet | 2016 |
Association of plasminogen activator inhibitor-1 and vitamin D receptor expression with the risk of keloid disease in a Chinese population.
Topics: Adolescent; Adult; Aged; Aged, 80 and over; Asian People; Biomarkers; Case-Control Studies; Child; F | 2017 |
Association of HLA-DQA1 and DQB1 alleles with keloids in Chinese Hans.
Topics: Adolescent; Adult; Alleles; Asian People; Case-Control Studies; China; Female; Gene Frequency; Genet | 2008 |
GenCLiP: a software program for clustering gene lists by literature profiling and constructing gene co-occurrence networks related to custom keywords.
Topics: Cluster Analysis; Databases, Bibliographic; Gene Expression Profiling; Gene Regulatory Networks; Gen | 2008 |
Gene expression profiling reveals alteration of caspase 6 and 14 transcripts in normal skin of keloid-prone patients.
Topics: Apoptosis; Biopsy; Black People; Case-Control Studies; Caspase 14; Caspase 6; Cicatrix; Cytokines; G | 2009 |
Positive association of HLA-DRB1*15 with keloid disease in Caucasians.
Topics: Gene Frequency; Genetic Predisposition to Disease; HLA-DR Antigens; HLA-DRB1 Chains; Humans; Keloid; | 2008 |
Association of HLA-DRB1* and keloid disease in an Afro-Caribbean population.
Topics: Adolescent; Adult; Aged; Aged, 80 and over; Black People; Case-Control Studies; Child; Child, Presch | 2010 |
Association of HLA class I alleles with keloids in Chinese Han individuals.
Topics: Adolescent; Adult; Aged; Child; Child, Preschool; China; Disease Progression; DNA Mutational Analysi | 2010 |
A genome-wide association study identifies four susceptibility loci for keloid in the Japanese population.
Topics: Asian People; Case-Control Studies; Chromosomes, Human, Pair 1; Chromosomes, Human, Pair 15; Chromos | 2010 |
Association of HLA haplotype with keloids in Chinese Hans.
Topics: Adolescent; Adult; Aged; Asian People; Child; Child, Preschool; China; Female; Gene Frequency; Genet | 2011 |
Assessment of the influence of HLA class I and class II loci on the prevalence of keloid disease in Jamaican Afro-Caribbeans.
Topics: Adult; Alleles; Black People; Ethnicity; Female; Genetic Predisposition to Disease; Histocompatibili | 2011 |
Comparative genomic hybridisation analysis of keloid tissue in Caucasians suggests possible involvement of HLA-DRB5 in disease pathogenesis.
Topics: Adolescent; Adult; Aged; Child; Cicatrix; Cohort Studies; Comparative Genomic Hybridization; DNA Cop | 2012 |
Association of TGFβ1 and SMAD4 variants in the etiology of keloid scar in the Malay population.
Topics: Adolescent; Adult; Case-Control Studies; Child; Female; Gene Frequency; Genetic Association Studies; | 2012 |
Genetic susceptibility to keloid disease and transforming growth factor beta 2 polymorphisms.
Topics: Cicatrix, Hypertrophic; Genetic Predisposition to Disease; Genotype; Humans; Keloid; Polymorphism, G | 2002 |
Genetic susceptibility to keloid disease and hypertrophic scarring: transforming growth factor beta1 common polymorphisms and plasma levels.
Topics: Adolescent; Adult; Cicatrix, Hypertrophic; Codon; Female; Gene Frequency; Genetic Predisposition to | 2003 |
Genetic susceptibility to keloid disease: transforming growth factor beta receptor gene polymorphisms are not associated with keloid disease.
Topics: Case-Control Studies; DNA; DNA Primers; Gene Frequency; Genetic Predisposition to Disease; Genotype; | 2004 |
Genome scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11.
Topics: Black or African American; Chromosomes, Human, Pair 2; Chromosomes, Human, Pair 7; Family Health; Fe | 2004 |
Genome scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11.
Topics: Black or African American; Chromosomes, Human, Pair 2; Chromosomes, Human, Pair 7; Family Health; Fe | 2004 |
Genome scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11.
Topics: Black or African American; Chromosomes, Human, Pair 2; Chromosomes, Human, Pair 7; Family Health; Fe | 2004 |
Genome scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11.
Topics: Black or African American; Chromosomes, Human, Pair 2; Chromosomes, Human, Pair 7; Family Health; Fe | 2004 |
[p53 gene codon 72 polymorphism and susceptibility to keloid in Chinese population].
Topics: Alleles; Arginine; Asian People; China; Codon; Exons; Gene Frequency; Genetic Predisposition to Dise | 2005 |
[Investigation of p53 polymorphism for genetic predisposition of keloid and hypertrophic scar].
Topics: Cicatrix, Hypertrophic; Female; Gene Frequency; Genetic Predisposition to Disease; Genotype; Humans; | 2005 |
Genetic susceptibility to keloid disease: mutation screening of the TGFbeta3 gene.
Topics: Adolescent; Adult; Chromatography, High Pressure Liquid; DNA Mutational Analysis; Exons; Female; Gen | 2005 |
[p53 gene codon 72 polymorphism and susceptibility to keloid].
Topics: Adolescent; Adult; Alleles; Asian People; Base Sequence; Child; China; Codon; Female; Gene Frequency | 2005 |
Keloid epidemiology: population based studies needed.
Topics: Female; Genetic Predisposition to Disease; Humans; Keloid; Patient Acceptance of Health Care | 2006 |
[Linkage analysis of keloid susceptibility loci on chromosome 7p11 in a Chinese pedigree].
Topics: Adolescent; Adult; Asian People; China; Chromosomes, Human, Pair 7; Female; Genetic Linkage; Genetic | 2006 |
[Preliminary linkage analysis and mapping of keloid susceptibility locus in a Chinese pedigree].
Topics: Asian People; Chromosome Mapping; Chromosomes, Human, Pair 15; Chromosomes, Human, Pair 18; Female; | 2007 |
[Location of predisposing gene for one Han Chinese keloid pedigree].
Topics: Asian People; Chromosomes, Human, Pair 10; Fas Ligand Protein; Female; Gene Frequency; Genetic Linka | 2007 |
Restylane and people of color.
Topics: Cicatrix, Hypertrophic; Cosmetic Techniques; Female; Genetic Predisposition to Disease; Humans; Hyal | 2007 |
Genetic susceptibility to keloid scarring: SMAD gene SNP frequencies in Afro-Caribbeans.
Topics: Adolescent; Adult; Aged; Aged, 80 and over; Black People; Case-Control Studies; Child; Child, Presch | 2008 |