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ketoconazole and Injury, Ischemia-Reperfusion

ketoconazole has been researched along with Injury, Ischemia-Reperfusion in 1 studies

1-acetyl-4-(4-{[2-(2,4-dichlorophenyl)-2-(1H-imidazol-1-ylmethyl)-1,3-dioxolan-4-yl]methoxy}phenyl)piperazine : A dioxolane that is 1,3-dioxolane which is substituted at positions 2, 2, and 4 by imidazol-1-ylmethyl, 2,4-dichlorophenyl, and [para-(4-acetylpiperazin-1-yl)phenoxy]methyl groups, respectively.

Research Excerpts

ExcerptRelevanceReference
"The bioavailability of orally administrated cyclosporine A (CsA) is poor and variable in liver transplantation recipients."5.35Decreased oral absorption of cyclosporine A after liver ischemia-reperfusion injury in rats: the contribution of CYP3A and P-glycoprotein to the first-pass metabolism in intestinal epithelial cells. ( Ikemura, K; Iwamoto, T; Matsuda, H; Mizutani, H; Okuda, M; Urano, K, 2009)
"The bioavailability of orally administrated cyclosporine A (CsA) is poor and variable in liver transplantation recipients."1.35Decreased oral absorption of cyclosporine A after liver ischemia-reperfusion injury in rats: the contribution of CYP3A and P-glycoprotein to the first-pass metabolism in intestinal epithelial cells. ( Ikemura, K; Iwamoto, T; Matsuda, H; Mizutani, H; Okuda, M; Urano, K, 2009)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ikemura, K1
Urano, K1
Matsuda, H1
Mizutani, H1
Iwamoto, T1
Okuda, M1

Other Studies

1 other study available for ketoconazole and Injury, Ischemia-Reperfusion

ArticleYear
Decreased oral absorption of cyclosporine A after liver ischemia-reperfusion injury in rats: the contribution of CYP3A and P-glycoprotein to the first-pass metabolism in intestinal epithelial cells.
    The Journal of pharmacology and experimental therapeutics, 2009, Volume: 328, Issue:1

    Topics: Animals; Area Under Curve; ATP Binding Cassette Transporter, Subfamily B, Member 1; Bile; Biological

2009