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ketamine and Parkinson Disease, Secondary

ketamine has been researched along with Parkinson Disease, Secondary in 3 studies

Ketamine: A cyclohexanone derivative used for induction of anesthesia. Its mechanism of action is not well understood, but ketamine can block NMDA receptors (RECEPTORS, N-METHYL-D-ASPARTATE) and may interact with sigma receptors.
ketamine : A member of the class of cyclohexanones in which one of the hydrogens at position 2 is substituted by a 2-chlorophenyl group, while the other is substituted by a methylamino group.

Parkinson Disease, Secondary: Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (66.67)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Verma, A1
Kulkarni, SK1
Kucheryanu, VG1
Kryzhanovskii, GN1
Onofrj, M1
Ghilardi, MF1
Basciani, M1
Martinez-Tica, J1
Glover, A1

Other Studies

3 other studies available for ketamine and Parkinson Disease, Secondary

ArticleYear
N-Methyl-D-aspartate receptor participation in Parkinson's disease, a neurodegenerative disorder.
    Annals of the New York Academy of Sciences, 1995, Sep-15, Volume: 765

    Topics: Animals; Antiparkinson Agents; Apomorphine; Benzazepines; Catalepsy; Dizocilpine Maleate; Haloperido

1995
Effect of glutamate and antagonists of N-methyl-D-aspartate receptors on experimental parkinsonian syndrome in rats.
    Bulletin of experimental biology and medicine, 2000, Volume: 130, Issue:7

    Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Animals; Excitatory Amino Acid Antagonists; Glutamic A

2000
Attenuation of the early anterior negativity of median nerve somatosensory evoked potential in the MPTP-treated monkey.
    Neurophysiologie clinique = Clinical neurophysiology, 1990, Volume: 20, Issue:4

    Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Animals; Electric Stimulation; Evoked Potentials, Soma

1990