ketamine has been researched along with Diabetes Insipidus, Neurogenic in 2 studies
Ketamine: A cyclohexanone derivative used for induction of anesthesia. Its mechanism of action is not well understood, but ketamine can block NMDA receptors (RECEPTORS, N-METHYL-D-ASPARTATE) and may interact with sigma receptors.
ketamine : A member of the class of cyclohexanones in which one of the hydrogens at position 2 is substituted by a 2-chlorophenyl group, while the other is substituted by a methylamino group.
Diabetes Insipidus, Neurogenic: A genetic or acquired polyuric disorder caused by a deficiency of VASOPRESSINS secreted by the NEUROHYPOPHYSIS. Clinical signs include the excretion of large volumes of dilute URINE; HYPERNATREMIA; THIRST; and polydipsia. Etiologies include HEAD TRAUMA; surgeries and diseases involving the HYPOTHALAMUS and the PITUITARY GLAND. This disorder may also be caused by mutations of genes such as ARVP encoding vasopressin and its corresponding neurophysin (NEUROPHYSINS).
| Excerpt | Relevance | Reference |
|---|---|---|
| "We report the first teenage case of ketamine-induced transient central diabetes insipidus." | 7.85 | A case of central diabetes insipidus after ketamine infusion during an external to internal carotid artery bypass. ( Amenta, P; Beakley, BD; Eskander, JP; Gaffar, S; McClure, BP; Pierre, N, 2017) |
| "Limited reports of transient central diabetes insipidus (DI) occurring early after initiation (ie, within 10 hours) of ketamine have been reported." | 5.62 | Delayed Onset of Central Diabetes Insipidus With Ketamine Sedation: A Report of 2 Cases. ( Baker, C; Cahoon, WD; Herity, LB; Kim, C; Lowe, DK, 2021) |
| "We report the first teenage case of ketamine-induced transient central diabetes insipidus." | 3.85 | A case of central diabetes insipidus after ketamine infusion during an external to internal carotid artery bypass. ( Amenta, P; Beakley, BD; Eskander, JP; Gaffar, S; McClure, BP; Pierre, N, 2017) |
| "Limited reports of transient central diabetes insipidus (DI) occurring early after initiation (ie, within 10 hours) of ketamine have been reported." | 1.62 | Delayed Onset of Central Diabetes Insipidus With Ketamine Sedation: A Report of 2 Cases. ( Baker, C; Cahoon, WD; Herity, LB; Kim, C; Lowe, DK, 2021) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Herity, LB | 1 |
| Baker, C | 1 |
| Kim, C | 1 |
| Lowe, DK | 1 |
| Cahoon, WD | 1 |
| Gaffar, S | 1 |
| Eskander, JP | 1 |
| Beakley, BD | 1 |
| McClure, BP | 1 |
| Amenta, P | 1 |
| Pierre, N | 1 |
2 other studies available for ketamine and Diabetes Insipidus, Neurogenic
| Article | Year |
|---|---|
Delayed Onset of Central Diabetes Insipidus With Ketamine Sedation: A Report of 2 Cases.
Topics: Anesthesia; Diabetes Insipidus, Neurogenic; Diabetes Mellitus; Extracorporeal Membrane Oxygenation; | 2021 |
A case of central diabetes insipidus after ketamine infusion during an external to internal carotid artery bypass.
Topics: Adolescent; Anastomosis, Surgical; Anesthetics, Dissociative; Carotid Artery, External; Carotid Arte | 2017 |