kainic acid has been researched along with Genetic Predisposition in 38 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (5.26) | 18.2507 |
| 2000's | 30 (78.95) | 29.6817 |
| 2010's | 4 (10.53) | 24.3611 |
| 2020's | 2 (5.26) | 2.80 |
| Authors | Studies |
|---|---|
| Berger, TC; Etholm, L; Heuser, K; Hjorthaug, HS; Nome, CG; Selmer, KK; Taubøll, E; Vigeland, MD | 1 |
| Bell, TA; Dalton, KA; Gu, B; Hock, P; Miller, DR; Pan, Y; Pardo-Manuel de Villena, F; Philpot, BD; Shaw, GD; Shorter, JR; Williams, LH | 1 |
| Cook, JK; Lee, KY; Liu, DC; Lizarazo, S; Tsai, NP | 1 |
| Chesebro, B; Pathmajeyan, M; Race, B; Rangel, A; Striebel, JF | 1 |
| Aguilar-Calvo, P; Carulla, P; del Río, JA; Espinosa, JC; Ferrer, I; Gavín, R; Legname, G; Llorens, F; Matamoros-Angles, A; Torres, JM | 1 |
| Berretta, N; Cecconi, F; Cervelli, M; Cervetto, C; D'Amelio, M; Marcoli, M; Mariottini, P; Maura, G; Mercuri, N; Passalacqua, M; Ragazzoni, M; Venturini, A; Vergani, L; Voci, A | 1 |
| Adams, BE; Myers, D; Ng, C; O'Brien, TJ; Phillips, AM; Powell, K; Reid, CA; Williams, DA; Zheng, T | 1 |
| Schauwecker, PE | 2 |
| Cepeda, C; Cohen, RW; Crawford, CA; Levine, MS; Margulies, JE; Watson, JB | 1 |
| Fernandez, SR; Siao, CJ; Tsirka, SE | 1 |
| Clement, AB; Cravatt, BF; Hawkins, EG; Lichtman, AH | 1 |
| Inagaki, C; Omori, K; Suzukawa, J; Yang, L | 1 |
| Fei, J; Guo, LH; Ma, YH; Mei, ZT; Zhao, WJ | 1 |
| Albo, F; Bernardi, G; Ferrari, F; Longone, P; Mercuri, N; Spalloni, A; Zona, C | 1 |
| Gary, D; Mackenzie, A; Mattson, M; Robertson, GS; Thompson, C | 1 |
| Santos, JB; Schauwecker, PE; Williams, RW | 1 |
| Beaulieu, JM; Julien, JP; Kriz, J; Krnjević, K | 1 |
| Akopian, G; Andersen, JK; Boonplueang, R; Kuhlenkamp, JF; Lu, SC; Stevenson, FF; Walsh, JP | 1 |
| Bolwig, TG; Ernfors, P; Husum, H; Kokaia, M; Nanobashvili, A; Sørensen, AT; Sørensen, G; Woldbye, DP | 1 |
| Da Silva, AG; Gilby, KL; McIntyre, DC | 1 |
| McLin, JP; Steward, O; Thompson, LM | 1 |
| Avanzini, G; Buzzi, A; Chikhladze, M; Franceschetti, S; Frassoni, C; Magnaghi, G; Paradiso, B; Sancini, G; Simonato, M; Zucchini, S | 1 |
| Chancer, Z; Lorenzana, A; Schauwecker, PE | 1 |
| B Jaffe, D; Chao, C; Dikkes, P; Guo, WH; Hemond, P; Lopez, MF; Yoon, K; Zurakowski, D | 1 |
| Chaix, Y; Ferraro, TN; Lapouble, E; Martin, B | 1 |
| Chae, JS; Kim, HC; Kim, HJ; Kim, WK; Ko, KH; Shin, EJ; Wie, MB; Yen, TP | 1 |
| Deng, Q; Kong, S; Lorenzana, A; McNeill, TH; Schauwecker, PE | 1 |
| Berrettini, WH; Ferraro, TN; Golden, GT; Smith, GG | 1 |
| Anderson, R; Barnes, JC; Collins, FC; Royle, SJ; Rupniak, HT | 1 |
| Cole, TB; Palmiter, RD; Robbins, CA; Schwartzkroin, PA; Wenzel, HJ | 1 |
| Berrettini, WH; Ferraro, TN; Golden, GT; Jones, NL; Smith, GG; Snyder, RL | 1 |
| Fei, J; Guo, LH; Hu, JH; Ma, Y; Mei, ZT; Yu, Y; Zhao, WJ; Zhou, XG | 1 |
| Bottazzi, B; Garlanda, C; Hirsch, E; Mantovani, A; Moneta, D; Peri, G; Ravizza, T; Richards, GJ; Vezzani, A | 1 |
| Davies, B; Davies, CH; Kearns, IR; Lathe, R; Ure, J | 1 |
| Hohmann, JG; Miller, NS; Palmiter, RD; Pyati, U; Rust, NC; Szot, P; Weinshenker, D; White, SS | 1 |
| Fritschy, JM; Grady, RM; Knuesel, I; Riban, V; Sanes, JR; Schaub, MC; Zuellig, RA | 1 |
| Jiménez-Rivera, CA; Mejías-Aponte, CA; Segarra, AC | 1 |
2 review(s) available for kainic acid and Genetic Predisposition
| Article | Year |
|---|---|
Complications associated with genetic background effects in models of experimental epilepsy.
Topics: Animals; Brain; Disease Models, Animal; Epilepsy; Excitatory Amino Acid Agonists; Genetic Predisposition to Disease; Humans; Kainic Acid; Phenotype; Seizures; Species Specificity | 2002 |
Chemoconvulsant-induced seizure susceptibility: toward a common genetic basis?
Topics: Animals; Carbolines; Chromosome Mapping; Chromosomes; Convulsants; GABA Agonists; Genetic Predisposition to Disease; Humans; Kainic Acid; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Inbred Strains; Pentylenetetrazole; Potassium Channels; Quantitative Trait Loci; Seizures | 2007 |
36 other study(ies) available for kainic acid and Genetic Predisposition
| Article | Year |
|---|---|
Neuronal and glial DNA methylation and gene expression changes in early epileptogenesis.
Topics: Animals; Disease Models, Animal; DNA Methylation; Epigenesis, Genetic; Epilepsy, Temporal Lobe; Galanin; Gene Expression Profiling; Gene Expression Regulation; Gene Regulatory Networks; Genetic Predisposition to Disease; Histone Deacetylases; Kainic Acid; Male; Mice; Neuroglia; Neurons; Osteopontin; Receptors, Dopamine D1; Sequence Analysis, DNA; Sequence Analysis, RNA | 2019 |
Collaborative Cross mice reveal extreme epilepsy phenotypes and genetic loci for seizure susceptibility.
Topics: Animals; Chromosome Mapping; Collaborative Cross Mice; Convulsants; Disease Models, Animal; Epilepsy; Excitatory Amino Acid Agonists; Flurothyl; Gene Expression; Gene Expression Profiling; Genetic Predisposition to Disease; Genotype; Hippocampus; Kainic Acid; Mice; Mice, Inbred Strains; Pentylenetetrazole; Phenotype; Quantitative Trait Loci; Seizures; Sudden Unexpected Death in Epilepsy; Whole Genome Sequencing | 2020 |
ER stress-induced modulation of neural activity and seizure susceptibility is impaired in a fragile X syndrome mouse model.
Topics: Animals; Benzothiazoles; Endoplasmic Reticulum Stress; Fragile X Mental Retardation Protein; Fragile X Syndrome; Genetic Predisposition to Disease; Kainic Acid; Mice; Mice, Inbred C57BL; Mice, Knockout; Nerve Net; Patch-Clamp Techniques; Seizures; Toluene; Tumor Suppressor Protein p53 | 2021 |
Lack of influence of prion protein gene expression on kainate-induced seizures in mice: studies using congenic, coisogenic and transgenic strains.
Topics: Animals; Gene Expression; Genetic Predisposition to Disease; Kainic Acid; Mice; Mice, Transgenic; Neurons; Prions; Seizures; Species Specificity | 2013 |
Involvement of PrP(C) in kainate-induced excitotoxicity in several mouse strains.
Topics: Animals; Biomarkers; Cell Death; Cell Line; Cytokines; Disease Models, Animal; Gene Expression; Genetic Predisposition to Disease; Gliosis; Hippocampus; Inflammation Mediators; Kainic Acid; Mice; Mice, Knockout; PrPC Proteins; Seizures; Transfection | 2015 |
Astrocyte-Dependent Vulnerability to Excitotoxicity in Spermine Oxidase-Overexpressing Mouse.
Topics: Animals; Aspartic Acid; Astrocytes; Benzodiazepines; Biogenic Polyamines; Calcium; Cerebral Cortex; Enzyme Induction; Genetic Predisposition to Disease; Gliosis; Hippocampus; Kainic Acid; Male; Metallothionein; Mice; Mice, Neurologic Mutants; Mice, Transgenic; Nerve Tissue Proteins; Neurons; Neurotoxins; Oxidative Stress; Oxidoreductases Acting on CH-NH Group Donors; Polyamine Oxidase; Receptors, AMPA; Recombinant Fusion Proteins; Seizures; Synaptosomes; Up-Regulation | 2016 |
Excitotoxic-mediated transcriptional decreases in HCN2 channel function increase network excitability in CA1.
Topics: Action Potentials; Animals; Biological Clocks; Cortical Synchronization; Epilepsy; Excitatory Amino Acid Antagonists; Genetic Predisposition to Disease; Hippocampus; Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels; Ion Channels; Kainic Acid; Lamotrigine; Nerve Net; Neurotoxins; Organ Culture Techniques; Potassium Channels; Rats; Rats, Sprague-Dawley; RNA, Messenger; Synaptic Transmission; Time Factors; Transcriptional Activation; Triazines | 2009 |
Enhanced epileptogenic susceptibility in a genetic model of reactive synaptogenesis: the spastic Han-Wistar rat.
Topics: Animals; Behavior, Animal; Disease Models, Animal; Electroencephalography; Excitatory Amino Acid Agonists; Genetic Predisposition to Disease; Hippocampus; Immunohistochemistry; In Situ Hybridization; Kainic Acid; Proto-Oncogene Proteins c-fos; Rats; Rats, Mutant Strains; Receptors, AMPA; RNA, Messenger; Seizures; Synapses | 2002 |
Differences in ionotropic glutamate receptor subunit expression are not responsible for strain-dependent susceptibility to excitotoxin-induced injury.
Topics: Animals; Cell Death; Disease Models, Animal; Drug Resistance; Epilepsy; Genetic Predisposition to Disease; Glutamic Acid; Immunohistochemistry; Kainic Acid; Male; Mice; Mice, Inbred C57BL; Nerve Degeneration; Neurotoxins; Protein Subunits; Receptors, AMPA; Receptors, Glutamate; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Species Specificity; Synaptic Transmission | 2003 |
Cell type-specific roles for tissue plasminogen activator released by neurons or microglia after excitotoxic injury.
Topics: Animals; Cell Count; Cells, Cultured; Disease Progression; Gene Expression; Genes, fms; Genetic Predisposition to Disease; Kainic Acid; Mice; Mice, Knockout; Mice, Transgenic; Microglia; Neurodegenerative Diseases; Neurofilament Proteins; Neurons; Neurotoxins; Organ Specificity; Promoter Regions, Genetic; Tissue Plasminogen Activator | 2003 |
Increased seizure susceptibility and proconvulsant activity of anandamide in mice lacking fatty acid amide hydrolase.
Topics: Amidohydrolases; Animals; Arachidonic Acids; Bicuculline; Cannabinoid Receptor Modulators; Convulsants; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Synergism; Endocannabinoids; GABA Antagonists; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Limbic System; Mice; Mice, Knockout; Neurons; Neurotoxins; Polyunsaturated Alkamides; Seizures | 2003 |
Continuous administration of antisense oligonucleotides to c-fos reduced the development of seizure susceptibility after ethacrynic acid-induced seizure in mice.
Topics: Animals; Disease Models, Animal; Drug Administration Schedule; Drug Interactions; Epilepsy; Ethacrynic Acid; Gene Expression Regulation; Genetic Predisposition to Disease; Injections, Intraventricular; Kainic Acid; Male; Mice; Oligonucleotides, Antisense; Proto-Oncogene Proteins c-fos; Up-Regulation | 2003 |
Increase in drug-induced seizure susceptibility of transgenic mice overexpressing GABA transporter-1.
Topics: Animals; Animals, Genetically Modified; Anticonvulsants; Carrier Proteins; Disease Models, Animal; Female; GABA Plasma Membrane Transport Proteins; Genetic Predisposition to Disease; Kainic Acid; Male; Membrane Proteins; Membrane Transport Proteins; Mice; Mice, Inbred C57BL; Nipecotic Acids; Pentylenetetrazole; Picrotoxin; Seizures | 2003 |
Cu/Zn-superoxide dismutase (GLY93-->ALA) mutation alters AMPA receptor subunit expression and function and potentiates kainate-mediated toxicity in motor neurons in culture.
Topics: Alanine; Amyotrophic Lateral Sclerosis; Animals; Cells, Cultured; Drug Resistance; Fetus; Genetic Predisposition to Disease; Glycine; Kainic Acid; Membrane Potentials; Mice; Mice, Transgenic; Motor Neurons; Mutation; Neurotoxins; Protein Subunits; Receptors, AMPA; RNA, Messenger; Superoxide Dismutase; Synaptic Transmission | 2004 |
Kainic acid-induced naip expression in the hippocampus is blocked in mice lacking TNF receptors.
Topics: Animals; Apoptosis; Brain Ischemia; Disease Models, Animal; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Mice; Mice, Knockout; Nerve Degeneration; Nerve Tissue Proteins; Neuronal Apoptosis-Inhibitory Protein; Neuroprotective Agents; Receptors, Tumor Necrosis Factor; RNA, Messenger; Tumor Necrosis Factor-alpha | 2004 |
Genetic control of sensitivity to hippocampal cell death induced by kainic acid: a quantitative trait loci analysis.
Topics: Animals; Behavior, Animal; Cell Count; Cell Death; Chromosome Mapping; Crosses, Genetic; Excitatory Amino Acid Agonists; Female; Genetic Linkage; Genetic Predisposition to Disease; Genotype; Hippocampus; Histocytochemistry; Kainic Acid; Male; Mice; Mice, Inbred Strains; Nerve Degeneration; Neurons; Phenotype; Quantitative Trait Loci; Seizures | 2004 |
Up-regulation of peripherin is associated with alterations in synaptic plasticity in CA1 and CA3 regions of hippocampus.
Topics: Animals; Animals, Genetically Modified; Behavior, Animal; Disease Models, Animal; Down-Regulation; Epilepsy; Fluorescent Antibody Technique; Gene Expression; Genetic Predisposition to Disease; Hippocampus; Intermediate Filament Proteins; Kainic Acid; Long-Term Potentiation; Membrane Glycoproteins; Mice; Microtubule-Associated Proteins; Nerve Tissue Proteins; Neuronal Plasticity; Organ Culture Techniques; Peripherins; Promoter Regions, Genetic; Synaptic Transmission; Synaptophysin; Thalamus; Up-Regulation | 2005 |
Increased susceptibility of glutathione peroxidase-1 transgenic mice to kainic acid-related seizure activity and hippocampal neuronal cell death.
Topics: Animals; Cell Death; Cells, Cultured; Disease Models, Animal; Epilepsy; Genetic Predisposition to Disease; Glutathione Disulfide; Glutathione Peroxidase; Hippocampus; Kainic Acid; Mice; Mice, Transgenic; N-Methylaspartate; Nerve Degeneration; Neurons; Neurotoxins; Organ Culture Techniques; Oxidative Stress; Receptors, N-Methyl-D-Aspartate; Up-Regulation | 2005 |
Differential suppression of seizures via Y2 and Y5 neuropeptide Y receptors.
Topics: Animals; Cells, Cultured; Convulsants; Cyclohexanes; Disease Models, Animal; Epilepsy; Epilepsy, Temporal Lobe; Excitatory Amino Acid Agonists; Female; Gene Expression Regulation; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Male; Mice; Mice, Inbred BALB C; Mice, Knockout; Neurons; Neuropeptide Y; Organ Culture Techniques; Receptors, Neuropeptide Y; Synaptic Transmission; Xanthenes | 2005 |
Differential GABA(A) subunit expression following status epilepticus in seizure-prone and seizure-resistant rats: a putative mechanism for refractory drug response.
Topics: Amygdala; Animals; Anticonvulsants; Disease Models, Animal; Disease Susceptibility; Excitatory Amino Acid Agonists; Gene Expression; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Kindling, Neurologic; Male; Peptide Fragments; Polymerase Chain Reaction; Rats; Rats, Long-Evans; Rats, Wistar; Receptors, GABA-A; RNA, Messenger; Seizures; Species Specificity; Status Epilepticus; Transcription, Genetic | 2005 |
Differential susceptibility to striatal neurodegeneration induced by quinolinic acid and kainate in inbred, outbred and hybrid mouse strains.
Topics: Animals; Chimera; Corpus Striatum; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Resistance; Genetic Predisposition to Disease; Huntington Disease; Kainic Acid; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Inbred CBA; Mice, Inbred DBA; Mice, Inbred ICR; Nerve Degeneration; Neurons; Neurotoxins; Phenotype; Quinolinic Acid; Species Specificity | 2006 |
A pathogenetic hypothesis of Unverricht-Lundborg disease onset and progression.
Topics: Animals; Cystatin B; Cystatins; Disease Models, Animal; Disease Progression; Electrophysiology; Epilepsies, Myoclonic; Excitatory Amino Acid Agonists; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Mice; Mice, Inbred C57BL; Mice, Knockout; Microglia; Nerve Degeneration; Neurons; Organ Culture Techniques; Unverricht-Lundborg Syndrome | 2007 |
A quantitative trait locus on chromosome 18 is a critical determinant of excitotoxic cell death susceptibility.
Topics: Animals; Brain; Cell Death; Chromosomes, Mammalian; Disease Models, Animal; Epilepsy, Temporal Lobe; Excitatory Amino Acid Agonists; Female; Genetic Predisposition to Disease; Humans; Kainic Acid; Male; Mice; Mice, Congenic; Neurons; Phenotype; Quantitative Trait Loci; Seizures | 2007 |
IGF2 knockout mice are resistant to kainic acid-induced seizures and neurodegeneration.
Topics: Animals; Apoptosis; Brain; Disease Models, Animal; Epilepsy; Genetic Predisposition to Disease; Hippocampus; Insulin-Like Growth Factor II; Kainic Acid; Limbic System; Mice; Mice, Knockout; Nerve Degeneration; Neural Pathways; Neurotoxins; Organ Culture Techniques | 2007 |
Role of glutathione peroxidase in the ontogeny of hippocampal oxidative stress and kainate seizure sensitivity in the genetically epilepsy-prone rats.
Topics: Aging; Animals; Antioxidants; Drug Resistance; Enzyme Activation; Epilepsy; Excitatory Amino Acid Agonists; Genetic Predisposition to Disease; Glutathione; Glutathione Peroxidase; Hippocampus; Kainic Acid; Lipid Peroxidation; Male; Nerve Degeneration; Neuroglia; Neurotoxins; Oxidative Stress; Rats; Rats, Mutant Strains; Rats, Sprague-Dawley; Superoxide Dismutase; Superoxide Dismutase-1 | 2008 |
Variation in Galr1 expression determines susceptibility to exocitotoxin-induced cell death in mice.
Topics: Animals; Base Sequence; Cell Death; Epilepsy; Excitatory Amino Acid Agonists; Genetic Predisposition to Disease; Genetic Variation; Genomics; Haplotypes; Hippocampus; Kainic Acid; Male; Mice; Mice, Congenic; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Inbred DBA; Myelin Basic Protein; Nerve Tissue Proteins; Neurotoxins; Phenotype; Polymorphism, Single Nucleotide; Receptor, Galanin, Type 1; Species Specificity; Transcription Factors | 2008 |
Differential susceptibility to seizures induced by systemic kainic acid treatment in mature DBA/2J and C57BL/6J mice.
Topics: Animals; Blood-Brain Barrier; Brain; Disease Models, Animal; Dose-Response Relationship, Drug; Genetic Predisposition to Disease; Kainic Acid; Male; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Seizures; Species Specificity | 1995 |
Behavioural analysis and susceptibility to CNS injury of four inbred strains of mice.
Topics: Analysis of Variance; Animals; Central Nervous System; Genetic Predisposition to Disease; Genotype; Glial Fibrillary Acidic Protein; Image Processing, Computer-Assisted; Immunohistochemistry; Kainic Acid; Lipid Peroxidation; Male; Maze Learning; Mice; Mice, Inbred Strains; Motor Activity; Species Specificity; Synaptophysin | 1999 |
Seizures and neuronal damage in mice lacking vesicular zinc.
Topics: Animals; Bicuculline; Carrier Proteins; Cation Transport Proteins; Convulsants; Dose-Response Relationship, Drug; Electroencephalography; Flurothyl; Genetic Predisposition to Disease; Kainic Acid; Membrane Proteins; Membrane Transport Proteins; Metallothionein 3; Mice; Mice, Knockout; Nerve Tissue Proteins; Neurons; Pentylenetetrazole; Seizures; Synapses; Synaptic Vesicles; Zinc | 2000 |
Acute cocaine-induced seizures: differential sensitivity of six inbred mouse strains.
Topics: Animals; Brain; Cerebral Cortex; Cocaine; Disease Models, Animal; Dose-Response Relationship, Drug; Electroencephalography; Female; Genetic Predisposition to Disease; Hippocampus; Kainic Acid; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Inbred Strains; Seizures | 2001 |
Overexpression of gamma-aminobutyric acid transporter subtype I leads to susceptibility to kainic acid-induced seizure in transgenic mice.
Topics: Action Potentials; Amino Acid Transport System X-AG; Animals; ATP-Binding Cassette Transporters; Brain; Carrier Proteins; Electroencephalography; Excitatory Amino Acid Agonists; GABA Plasma Membrane Transport Proteins; gamma-Aminobutyric Acid; Gene Expression Regulation; Genetic Predisposition to Disease; Glutamic Acid; Kainic Acid; Membrane Proteins; Membrane Transport Proteins; Mice; Mice, Transgenic; Neural Inhibition; Neurons; Organic Anion Transporters; Receptors, GABA-A; Seizures; Synaptosomes; Up-Regulation | 2001 |
Dynamic induction of the long pentraxin PTX3 in the CNS after limbic seizures: evidence for a protective role in seizure-induced neurodegeneration.
Topics: 2-Amino-5-phosphonovalerate; Animals; C-Reactive Protein; Epilepsy; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Fluorescent Dyes; Genetic Predisposition to Disease; Immunohistochemistry; Kainic Acid; Limbic System; Male; Mice; Mice, Knockout; Nerve Degeneration; Neurons; Neuroprotective Agents; Prosencephalon; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; RNA, Messenger; Serum Amyloid P-Component | 2001 |
Loss of hippocampal serine protease BSP1/neuropsin predisposes to global seizure activity.
Topics: Action Potentials; Animals; Behavior, Animal; Cell Line; Electric Stimulation; Excitatory Postsynaptic Potentials; Gene Targeting; Genetic Predisposition to Disease; Hippocampus; In Situ Hybridization; In Vitro Techniques; Kainic Acid; Kallikreins; Long-Term Potentiation; Maze Learning; Mice; Mice, Inbred C57BL; Mice, Knockout; Proto-Oncogene Proteins c-fos; RNA, Messenger; Seizures; Serine Endopeptidases; Stem Cells | 2001 |
Genetic comparison of seizure control by norepinephrine and neuropeptide Y.
Topics: Animals; Dopamine beta-Hydroxylase; Exercise Test; Flurothyl; Genetic Predisposition to Disease; Handling, Psychological; In Situ Hybridization; Kainic Acid; Male; Mice; Mice, Knockout; Neuropeptide Y; Norepinephrine; Pentylenetetrazole; Phenotype; Seizures | 2001 |
Increased vulnerability to kainate-induced seizures in utrophin-knockout mice.
Topics: Animals; Cell Count; Cell Size; Cell Survival; Cytoskeletal Proteins; Dentate Gyrus; Dystrophin; Epilepsy; Epilepsy, Temporal Lobe; Excitatory Amino Acid Agonists; Female; Genetic Predisposition to Disease; Genotype; Hypertrophy; Immunohistochemistry; Kainic Acid; Male; Membrane Proteins; Mice; Mice, Knockout; Nerve Degeneration; Neurons; RNA, Messenger; Up-Regulation; Utrophin | 2002 |
Sex differences in models of temporal lobe epilepsy: role of testosterone.
Topics: Animals; Behavior, Animal; Brain; Corticosterone; Disease Models, Animal; Epilepsy, Temporal Lobe; Excitatory Amino Acid Agonists; Female; Genetic Predisposition to Disease; Hypothalamo-Hypophyseal System; Kainic Acid; Male; Muscarinic Agonists; Neurons; Pilocarpine; Rats; Rats, Sprague-Dawley; Reaction Time; Sex Characteristics; Testosterone | 2002 |