kainic acid has been researched along with Central Nervous System Diseases in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (14.29) | 18.7374 |
1990's | 2 (28.57) | 18.2507 |
2000's | 4 (57.14) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Birnbaum, MJ; Chao, MV; Cho, H; Kim, AH; Margolis, B; Meyer, D; Monks, B; Yano, H | 1 |
Dawson, TM; Dike, S; Fannjiang, Y; Griffin, DE; Hardwick, JM; Huganir, RL; Jonas, EA; Kaczmarek, LK; Kerr, DA; Kim, CH; Larsen, T; Lindsten, T; Mandir, AS; Mito, T; Sappington, AL; Thompson, CB; Traystman, RJ; Zou, S | 1 |
Carrasco, J; Florit, S; Giralt, M; Hidalgo, J; Molinero, A; Penkowa, M; Quintana, A | 1 |
Barbeito, L; Estevez, AG; Stutzmann, JM | 1 |
Baer, JD; Coyle, JT; Hassinger, LC; Leski, ML; Valentine, SL | 1 |
Halsey, MJ; Meldrum, BS; Wardley-Smith, B | 1 |
7 other study(ies) available for kainic acid and Central Nervous System Diseases
Article | Year |
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Akt1 regulates a JNK scaffold during excitotoxic apoptosis.
Topics: Adaptor Proteins, Signal Transducing; Animals; Apoptosis; Arabidopsis Proteins; Carrier Proteins; Cell Survival; Cells, Cultured; Central Nervous System Diseases; Fetus; Gene Deletion; Gene Expression; Glutamic Acid; Hippocampus; Humans; JNK Mitogen-Activated Protein Kinases; Kainic Acid; Mice; Mice, Knockout; Mitogen-Activated Protein Kinases; Neurons; Neurotoxins; Plant Proteins; Potassium Channels; Protein Binding; Rats; Rats, Sprague-Dawley; Receptors, Glutamate; Signal Transduction | 2002 |
BAK alters neuronal excitability and can switch from anti- to pro-death function during postnatal development.
Topics: Age Factors; Animals; Animals, Newborn; Apoptosis; bcl-2 Homologous Antagonist-Killer Protein; Central Nervous System; Central Nervous System Diseases; Central Nervous System Viral Diseases; Disease Models, Animal; Epilepsy; Excitatory Postsynaptic Potentials; Genetic Vectors; Hippocampus; Kainic Acid; Male; Membrane Proteins; Mice; Mice, Knockout; Neurodegenerative Diseases; Neurons; Neurotoxins; Protein Structure, Tertiary; Sindbis Virus; Stroke; Synaptic Transmission | 2003 |
Metallothionein reduces central nervous system inflammation, neurodegeneration, and cell death following kainic acid-induced epileptic seizures.
Topics: Amyloid beta-Peptides; Analysis of Variance; Animals; Astrocytes; Cell Count; Cell Death; Central Nervous System Diseases; Epilepsy; Gene Expression Regulation; Glial Fibrillary Acidic Protein; Growth Substances; Guanine; Hippocampus; Immunohistochemistry; In Situ Nick-End Labeling; Interleukins; Kainic Acid; Matrix Metalloproteinase 3; Matrix Metalloproteinase 9; Metallothionein; Mice; Mice, Inbred C57BL; Mice, Transgenic; Neurodegenerative Diseases; Neurofibrillary Tangles; Staining and Labeling; Tyrosine | 2005 |
Protective effect of riluzole on excitatory amino acid-mediated neurotoxicity in motoneuron-enriched cultures.
Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Cell Survival; Cells, Cultured; Central Nervous System Diseases; Drug Interactions; Excitatory Amino Acid Antagonists; Excitatory Amino Acids; Glutamic Acid; Kainic Acid; Motor Neurons; N-Methylaspartate; Rats; Receptors, Amino Acid; Riluzole; Thiazoles | 1995 |
L-type calcium channels reduce ROS generation in cerebellar granule cells following kainate exposure.
Topics: Animals; Animals, Newborn; Calcium Channel Blockers; Calcium Channels, L-Type; Cell Death; Cell Survival; Cells, Cultured; Central Nervous System; Central Nervous System Diseases; Cerebellum; Chelating Agents; Electron Transport; Excitatory Amino Acid Antagonists; Fluorescent Dyes; Iron; Kainic Acid; Microscopy, Electron; Mitochondria; Neurons; Neurotoxins; Nifedipine; Rats; Reactive Oxygen Species; Receptors, Glutamate | 2002 |
Domoic acid intoxication.
Topics: Animals; Bird Diseases; Birds; California; Central Nervous System Diseases; Food Contamination; Humans; Kainic Acid; Marine Toxins | 1992 |
The effect of two novel dipeptide antagonists of excitatory amino acid neurotransmission on the high pressure neurological syndrome in the rat.
Topics: Animals; Aspartic Acid; Central Nervous System Diseases; Glutamates; Glutamine; High Pressure Neurological Syndrome; Kainic Acid; Male; Motor Activity; Rats; Rats, Inbred Strains; Receptors, N-Methyl-D-Aspartate; Receptors, Neurotransmitter; Seizures | 1987 |