ivabradine has been researched along with Diabetic Cardiomyopathies in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, S; Gao, X; Luo, J; Qian, X; Ren, X; Ye, P; Zhang, J; Zuo, G | 1 |
| Chao, YL; Chen, SL; Gao, HB; Gao, XF; Ge, Q; Ge, Z; Gu, Y; Hu, ZY; Luo, J; Ren, XM; Wang, F; Wu, W; Ye, P; Zhang, JJ; Zuo, GF | 1 |
| Chen, SL; Hu, ZY; Li, B; Li, MH; Zuo, GF | 1 |
3 other study(ies) available for ivabradine and Diabetic Cardiomyopathies
| Article | Year |
|---|---|
Inhibition of JNK and p38 MAPK-mediated inflammation and apoptosis by ivabradine improves cardiac function in streptozotocin-induced diabetic cardiomyopathy.
Topics: Animals; Anti-Inflammatory Agents; Apoptosis; Cells, Cultured; Cytokines; Diabetic Cardiomyopathies; Inflammation Mediators; Ivabradine; JNK Mitogen-Activated Protein Kinases; Male; Mice, Inbred C57BL; Myocytes, Cardiac; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Rats; Recovery of Function; Signal Transduction; Streptozocin | 2019 |
Activation of the PP2A catalytic subunit by ivabradine attenuates the development of diabetic cardiomyopathy.
Topics: Animals; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Enzyme Activation; Ivabradine; Male; Mice; Molecular Docking Simulation; Myocytes, Cardiac; NF-kappa B; Protein Phosphatase 2; Rats; Signal Transduction | 2019 |
I(f) current channel inhibitor (ivabradine) deserves cardioprotective effect via down-regulating the expression of matrix metalloproteinase (MMP)-2 and attenuating apoptosis in diabetic mice.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; Benzazepines; Cardiotonic Agents; Caspase 3; Cells, Cultured; Diabetes Mellitus; Diabetic Cardiomyopathies; Disease Models, Animal; Down-Regulation; Ivabradine; Male; Matrix Metalloproteinase 2; Mice; Myocytes, Cardiac; NF-kappa B; Phosphorylation; Recovery of Function; Signal Transduction; Ventricular Function, Left | 2014 |