Page last updated: 2024-08-25

isouramil and Glucosephosphate Dehydrogenase Deficiency

isouramil has been researched along with Glucosephosphate Dehydrogenase Deficiency in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19903 (100.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chevion, M; Golenser, J; Miller, J; Navok, T; Spira, DT1
Arese, P; Bosia, A; D'Aquino, M; Gaetani, GF; Gaetani, S; Naitana, A1
Golenser, J; Jensen, JB; Kosower, NS; Miller, J; Spira, DT; Vande Waa, JA1

Other Studies

3 other study(ies) available for isouramil and Glucosephosphate Dehydrogenase Deficiency

ArticleYear
Inhibitory effect of a fava bean component on the in vitro development of Plasmodium falciparum in normal and glucose-6-phosphate dehydrogenase deficient erythrocytes.
    Blood, 1983, Volume: 61, Issue:3

    Topics: Barbiturates; Cells, Cultured; Erythrocytes; Female; Glucosephosphate Dehydrogenase Deficiency; Humans; Malaria; Male; Plasmodium falciparum

1983
Effect of divicine and isouramil on red cell metabolism in normal and G6PD-deficient (Mediterranean variant) subjects. Possible role in the genesis of favism.
    Progress in clinical and biological research, 1981, Volume: 55

    Topics: Ascorbic Acid; Barbiturates; Erythrocytes; Favism; Glucosephosphate Dehydrogenase Deficiency; Glucosides; Glutathione; Humans; Kinetics; Pyrimidinones; Reference Values; Species Specificity; Toxins, Biological; Uracil; Uridine

1981
Inhibition of the intraerythrocytic development of Plasmodium falciparum in glucose-6-phosphate dehydrogenase deficient erythrocytes is enhanced by oxidants and by crisis form factor.
    Tropical medicine and parasitology : official organ of Deutsche Tropenmedizinische Gesellschaft and of Deutsche Gesellschaft fur Technische Zusammenarbeit (GTZ), 1988, Volume: 39, Issue:4

    Topics: Animals; Barbiturates; Biological Factors; Cells, Cultured; Diamide; Erythrocytes; Glucosephosphate Dehydrogenase Deficiency; Humans; Male; Oxidation-Reduction; Plasmodium falciparum

1988