isomethyleugenol has been researched along with Precursor T-Cell Lymphoblastic Leukemia-Lymphoma in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Dawidowska, M; Drobna-Śledzińska, M; Jaksik, R; Kosmalska, M; Lejman, M; Maćkowska-Maślak, N; Sędek, Ł; Szarzyńska, B; Szczepański, T; Taghon, T; Van Vlierberghe, P; Witt, M | 1 |
Andrau, JC; Asnafi, V; Dombret, H; Duprez, E; Fenouil, R; Gaspar, HB; Gregoire, C; Gut, IG; Gut, M; Hacein-Bey-Abina, S; Howe, SJ; Ifrah, N; Jaeger, S; Koubi, M; Le Noir, S; Loosveld, M; Macintyre, EA; Malissen, B; Mamessier, E; Maqbool, MA; Morgado, E; Nadel, B; Navarro, JM; Payet-Bornet, D; Pignon, C; Pradel, LC; Thrasher, AJ; Touzart, A | 1 |
Forestier, E; Kristinsson, J; Schmiegelow, K; Söderhäll, S; Vettenranta, K; Weinshilboum, R; Wesenberg, F | 1 |
Drexler, HG; Quentmeier, H; Röhrs, S; Romani, J; Zaborski, M | 1 |
1 trial(s) available for isomethyleugenol and Precursor T-Cell Lymphoblastic Leukemia-Lymphoma
Article | Year |
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Thiopurine methyltransferase activity is related to the risk of relapse of childhood acute lymphoblastic leukemia: results from the NOPHO ALL-92 study.
Topics: Antimetabolites, Antineoplastic; Antineoplastic Combined Chemotherapy Protocols; Biotransformation; Child; Child, Preschool; DNA Damage; Female; Genotype; Humans; Inactivation, Metabolic; Infant; Male; Mercaptopurine; Methylation; Methyltransferases; Neoplasm Proteins; Neoplasms, Second Primary; Polymorphism, Genetic; Precursor B-Cell Lymphoblastic Leukemia-Lymphoma; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma; Recurrence; Risk; Scandinavian and Nordic Countries; Thioguanine | 2009 |
3 other study(ies) available for isomethyleugenol and Precursor T-Cell Lymphoblastic Leukemia-Lymphoma
Article | Year |
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Multiomics to investigate the mechanisms contributing to repression of PTPRC and SOCS2 in pediatric T-ALL: Focus on miR-363-3p and promoter methylation.
Topics: Cell Line, Tumor; Child; Humans; Janus Kinases; Leukocyte Common Antigens; Methylation; MicroRNAs; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma; Proteomics; Signal Transduction; STAT Transcription Factors; Suppressor of Cytokine Signaling Proteins | 2022 |
Site- and allele-specific polycomb dysregulation in T-cell leukaemia.
Topics: Acetylation; Adult; Alleles; Base Sequence; Basic Helix-Loop-Helix Transcription Factors; Chromatin Immunoprecipitation; DNA-Binding Proteins; Epigenesis, Genetic; Gene Expression Regulation, Leukemic; Genetic Loci; Histones; Homeodomain Proteins; Humans; Jurkat Cells; Methylation; Molecular Sequence Data; Mutagenesis, Insertional; Nuclear Proteins; Plasmids; Polycomb-Group Proteins; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma; Proto-Oncogene Proteins; Survival Analysis; T-Cell Acute Lymphocytic Leukemia Protein 1; Treatment Outcome | 2015 |
Hypermethylation of Death-Associated Protein Kinase 1 differentiates natural killer cell lines from cell lines derived from T-acute lymphoblastic leukemia.
Topics: Apoptosis Regulatory Proteins; Biomarkers; Calcium-Calmodulin-Dependent Protein Kinases; Cell Line, Tumor; Death-Associated Protein Kinases; Humans; Killer Cells, Natural; Methylation; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma | 2009 |