isomethyleugenol has been researched along with Left Ventricular Hypertrophy in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 2 (66.67) | 2.80 |
| Authors | Studies |
|---|---|
| Cai, S; Ding, Y; Lan, R; Li, J; Li, Z; Liu, P; Lu, J; Wang, J; Wang, P; Xie, T; Ye, J | 1 |
| Chen, XZ; Dong, YH; Gao, XQ; Ju, J; Li, PF; Li, XM; Liu, CY; Liu, F; Ponnusamy, M; Qian, LL; Shan, C; Shan, PP; Wang, K; Wang, M; Wang, T; Wang, Y; Wang, YH; Yu, T; Zhai, M; Zhang, J; Zhang, YH; Zhao, XM; Zhou, LY | 1 |
| Chen, HZ; Hill, JA; Liu, DP; Liu, ZP; Wang, L; Zhang, QJ | 1 |
3 other study(ies) available for isomethyleugenol and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
Histone H4R3 symmetric di-methylation by Prmt5 protects against cardiac hypertrophy via regulation of Filip1L/β-catenin.
Topics: Animals; Arginine; beta Catenin; Cells, Cultured; Disease Models, Animal; Epigenesis, Genetic; Histones; Hypertrophy, Left Ventricular; Male; Methylation; Mice, Inbred C57BL; Myocytes, Cardiac; Protein-Arginine N-Methyltransferases; Rats, Sprague-Dawley; Signal Transduction; Ventricular Function, Left; Ventricular Remodeling | 2020 |
The piRNA CHAPIR regulates cardiac hypertrophy by controlling METTL3-dependent N
Topics: Adenosine; Animals; Cells, Cultured; Disease Models, Animal; Gene Expression Regulation, Enzymologic; Glycogen Synthase Kinase 3 beta; Heart Ventricles; Hypertrophy, Left Ventricular; Male; Methylation; Methyltransferases; Mice, Inbred C57BL; Mice, Knockout; Myocytes, Cardiac; NFATC Transcription Factors; Poly(ADP-ribose) Polymerases; Proto-Oncogene Proteins; RNA Stability; RNA, Messenger; RNA, Small Interfering; Signal Transduction; Ventricular Function, Left; Ventricular Remodeling | 2020 |
The histone trimethyllysine demethylase JMJD2A promotes cardiac hypertrophy in response to hypertrophic stimuli in mice.
Topics: Animals; Aortic Diseases; Base Sequence; Cells, Cultured; Constriction, Pathologic; Female; Gene Expression Regulation; Heart Failure; Histone Demethylases; Histones; Humans; Hypertrophy, Left Ventricular; Intracellular Signaling Peptides and Proteins; Jumonji Domain-Containing Histone Demethylases; LIM Domain Proteins; Male; Methylation; Mice; Mice, Inbred C57BL; Mice, Transgenic; Molecular Sequence Data; Muscle Proteins; Myocytes, Cardiac; Nuclear Proteins; Promoter Regions, Genetic; Protein Processing, Post-Translational; Rats; RNA, Small Interfering; Serum Response Element; Trans-Activators; Ventricular Remodeling | 2011 |