isomethyleugenol has been researched along with Hemoglobinopathies in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 2 (66.67) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cunningham, JM; Gumucio, DL; Jane, SM; Ney, PA; Nienhuis, AW | 1 |
| Fischer, KD; Nowock, J | 1 |
| Feingold, EA; Forget, BG | 1 |
3 other study(ies) available for isomethyleugenol and Hemoglobinopathies
| Article | Year |
|---|---|
Methylation-enhanced binding of Sp1 to the stage selector element of the human gamma-globin gene promoter may regulate development specificity of expression.
Topics: Adult; Aging; Animals; Base Sequence; Binding Sites; DNA; Fetal Hemoglobin; Gene Expression Regulation; Globins; Hemoglobinopathies; Humans; Leukemia, Erythroblastic, Acute; Methylation; Mice; Molecular Sequence Data; Oligodeoxyribonucleotides; Promoter Regions, Genetic; Restriction Mapping; Sp1 Transcription Factor; Transfection; Tumor Cells, Cultured | 1993 |
The T----C substitution at -198 of the A gamma-globin gene associated with the British form of HPFH generates overlapping recognition sites for two DNA-binding proteins.
Topics: Base Composition; Base Sequence; Binding Sites; Binding, Competitive; DNA-Binding Proteins; Fetal Hemoglobin; Genes; Globins; Hemoglobinopathies; Humans; Leukemia, Erythroblastic, Acute; Methylation; Molecular Sequence Data; Mutation; Sp1 Transcription Factor; Transcription, Genetic; Tumor Cells, Cultured | 1990 |
The breakpoint of a large deletion causing hereditary persistence of fetal hemoglobin occurs within an erythroid DNA domain remote from the beta-globin gene cluster.
Topics: Amino Acid Sequence; Base Sequence; Blotting, Southern; Chromosome Deletion; Enhancer Elements, Genetic; Erythroid Precursor Cells; Fetal Hemoglobin; Gene Expression Regulation; Globins; Hemoglobinopathies; Humans; Methylation; Molecular Sequence Data; Regulatory Sequences, Nucleic Acid; Restriction Mapping; Thalassemia; Transcription, Genetic | 1989 |